Distal-Less Homeobox 5 (DLX5) ELISA Kits

DLX5 encodes a member of a homeobox transcription factor gene family similiar to the Drosophila distal-less gene. Zusätzlich bieten wir Ihnen Distal-Less Homeobox 5 Antikörper (106) und Distal-Less Homeobox 5 Proteine (8) und viele weitere Produktgruppen zu diesem Protein an.

list all ELISA KIts Gen GeneID UniProt
DLX5 1749 P56178
DLX5 13395 P70396
DLX5 25431 P50575
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Top Distal-Less Homeobox 5 ELISA Kits auf antikoerper-online.de

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Katalog Nr. Reaktivität Sensitivität Bereich Bilder Menge Anbieter Lieferzeit Preis Details
Human 1.0 pg/mL 250-5000 pg/mL   96 Tests Anmelden zum Anzeigen 15 bis 18 Tage
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Weitere ELISA Kits für Distal-Less Homeobox 5 Interaktionspartner

Human Distal-Less Homeobox 5 (DLX5) Interaktionspartner

  1. Our data suggest that loss of DLX5, TLX1 and HOXA10 expression in late gestation is required for proper placental differentiation and function.

  2. Our results in mice suggest that long-range DLX5 enhancer elements located in the human SLC25A13 gene may underlie the sensorineural hearing loss that is sometimes associated with SHFM1.

  3. These results indicate activation of DLX5 and RUNX2 via its distal promoter represents a unique feature of GFs, and is important for ECM regulation. Down-regulation of these transcription factors in PAFs could be associated with their property to degrade collagen, which may impact on the process of periodontitis.

  4. these findings indicate that, in MSCs, DLX5 is a master regulator of osteogenesis. Furthermore, tanshinone IIA may be valuable for stem cell-based therapies of certain bone diseases.

  5. As a result of disturbed imprinting, the upregulated DLX5 affects trophoblast proliferation in preeclampsia.

  6. These data indicate that certain missense mutations diminish the ability of the Dlx5 homeodomain to recognize and bind target DNAs, and they likely destabilize the formation of functional complexes.

  7. In cells grown on titanium support, DLX5 and RUNX1 were respectively upregulated (+3.12-fold) and downregulated (-2.14-fold)

  8. Absent expression of the osteoblast-specific maternally imprinted genes, DLX5 and DLX6, causes split hand/split foot malformation type I.

  9. Heterozygous DLX5 nonsense mutation c.G115T(p.E39X) associated with isolated split-hand/foot malformation with reduced penetrance and variable expressivity in two unrelated Polish families.

  10. A novel heterozygous mutation in exon 3 of DLX5 found in the family members with SHFM1 phenotype.

  11. Genome sequencing of the deletion breakpoints showed that the DLX5 and DLX6 genes are disomic but the putative DYNC1I1 exon 15 and 17 enhancers are deleted.

  12. The duplicated region harbors only DLX5 and DLX6, which are known for their role in SHFM1.

  13. The strongest evidence for altered methylation patterns in shiftworkers was observed for DLX5 gene.

  14. Cyclic tensile stress may induce differentiation of periodontal ligament stem cells towards mineralized tissue cells by promoting Dlx5 mRNA expression and decreasing Msx2 expression.

  15. Two patients that have in common a p63-Dlx5/Dlx6 pathway dysregulation.

  16. the first intragenic DLX5 mutation in split hand and foot malformation is found; a potential dual role for DLX5 in limb development is suggested

  17. DLX5 is significantly increased in heart tissue from calcific aortic valve patients compared to controls.

  18. MDA-MB-231 breast neoplasms did not express DLX5 but the resulting bone/lung metastases did.

  19. Data suggest that DLX5 plays a significant role in the pathogenesis of some ovarian cancers by enhancing IRS-2-AKT signaling.

  20. p63 binds to an enhancer element in the SHFM1 locus and this element controls expression of DLX6 and DLX5 which are important for limb development.

Mouse (Murine) Distal-Less Homeobox 5 (DLX5) Interaktionspartner

  1. T-614 promotes osteoblastic differentiation by increasing the expression of Osterix and Dlx5.

  2. Results indicate that Dlx5 and Runx2 are critical factors for the upregulated Osterix expression in shPP2A cells, which is considered to be important for the accelerated osteoblast differentiation in these cells.

  3. FGF10 pathway is downregulated in Dlx5(-/-) mice, and activation of FGF10 signaling rescues cranial neural crest cell proliferation and myogenic differentiation.

  4. High DLX5 expression is associated with T-cell lymphomagenesis.

  5. Both transient and stable expression of Necdin induced osteoblast-specific markers in an osteogenic cell line through formation of a complex with distal-less Homeobox 5 (Dlx5) and Runx2 promoter activation.

  6. DLX5 and DLX6 reciprocally inhibit BMP/H2-mediated H1 enhancer regulation in mandible embryonic development.

  7. We found that in Dlx5;6 DKO limbs, the AER expresses lower levels of Wnt5a, shows scattered beta-catenin responsive cells and altered basolateral and planar cell polarity (PCP).

  8. Dlx5 and Dlx6 expression determines uterine architecture and adenogenesis and is needed for implantation

  9. The results presented here indicate that loss of Dlx5 causes a down-modulation of miR-9 and of miR-200-class, which results in the over-expression of the Foxg1 protein.

  10. Lck-Dlx5 mice develop T-ALLs that consistently acquire overexpression of Myc and activation of Akt.

  11. Study demonstrated a novel role of miR-124 and Dlx5 in regulating the differentiation of mesenchymal stem cells toward the myogenic lineage, that is, miR-124 inhibits myogenic differentiation partially through targeting Dlx5 expression.

  12. Mash1 is required for the expression of GAD67 and Dlx5 in taste bud cells.

  13. Dlx5 and Mef2 directly bound to the runx2 enhancer, and the binding sites were required for the osteoblast-specific expression

  14. the prethalamic factors Dlxs upregulated by Ascl1/Helt deficiency play unique roles in regulating thalamic progenitor specification.

  15. Results show that DLX5, p63, Pin1 and FGF8 participate to the same time- and location-restricted regulatory loop essential for apical ectodermal ridge stratification, hence for normal patterning and skeletal morphogenesis of the limb buds.

  16. The Edn1/Ednra signaling regulates neural crest differentiation to ensure the proper patterning of pharyngeal arch arteries, which is independent of the mechanism mediated by Dlx5/Dlx6.

  17. Data show that Dlx5 and Msx2 play a critical role in controlling cranial neural tube morphogenesis by regulating cell adhesion via the ephrinA5 and EphA7 pathway.

  18. in AER cells and, at later stages, in the limb mesoderm the regulation of by Dlx5 and Dlx6 occurs also cell autonomously

  19. allelic reduction of Dlx5 and Dlx6 in the mouse results in a POI-like phenotype, characterized by reduced fertility and early follicular exhaustion.

  20. Dlx5, Msx2, and Runx2 enhancers exhibit a dual requirement for TCF/LEF1 and Smad binding element (SBE) sites that mediate synexpression of the three genes during osteoblast differentiation.

Xenopus laevis Distal-Less Homeobox 5 (DLX5) Interaktionspartner

  1. Retinoic acid treatment leads to a progressive loss of Dlx5 and Dlx6 expression in the first pharyngeal arch.

Pig (Porcine) Distal-Less Homeobox 5 (DLX5) Interaktionspartner

  1. Imprinting analysis shows that DLX5 is maternally expressed in some organs and tissues but not imprinted in others.

Zebrafish Distal-Less Homeobox 5 (DLX5) Interaktionspartner

  1. Data indicate that retinoic acid (RA) induces the expression patterns of genes normally expressed in the posterior tooth-forming region, such as pitx2 and dlx2b.

  2. Zebrafish dlx2b can drive reporter expression in oral teeth of the related characiform Astyanax mexicanus.

Distal-Less Homeobox 5 (DLX5) Antigen-Profil

Beschreibung des Gens

This gene encodes a member of a homeobox transcription factor gene family similiar to the Drosophila distal-less gene. The encoded protein may play a role in bone development and fracture healing. Mutation in this gene, which is located in a tail-to-tail configuration with another member of the family on the long arm of chromosome 7, may be associated with split-hand/split-foot malformation.

Genbezeichner und Symbole assoziert mit DLX5

  • distal-less homeobox 5 (DLX5) Antikörper
  • distal-less homeobox 5 (Dlx5) Antikörper
  • distal-less homeobox 5 L homeolog (dlx5.L) Antikörper
  • distal-less homeobox 5 (dlx5) Antikörper
  • distal-less homeobox 2b (dlx2b) Antikörper
  • AI385752 Antikörper
  • dll3 Antikörper
  • dlx5 Antikörper
  • MGC69418 Antikörper
  • RDLX Antikörper
  • SHFM1D Antikörper
  • X-dll3 Antikörper

Bezeichner auf Proteinebene für DLX5

distal-less homeo box 5 , homeobox protein DLX-5 , split hand/foot malformation type 1 with sensorineural hearing loss , DLX-3 , homeobox protein DLX-3 , cDlx , XDLL-3 , homeobox protein DLL-3 , putative transcription factor DLL3 , distal-less homeobox 5 , distal-less homeobox protein 5 , DLX-5 , distal-less homeobox gene 5 , distal-less homeobox protein 2b , homeobox protein Dlx2b

1749 Homo sapiens
13395 Mus musculus
25431 Rattus norvegicus
373969 Gallus gallus
399265 Xenopus laevis
447997 Xenopus (Silurana) tropicalis
463554 Pan troglodytes
482313 Canis lupus familiaris
507415 Bos taurus
697853 Macaca mulatta
100008630 Oryctolagus cuniculus
100286863 Sus scrofa
30557 Danio rerio
101119298 Ovis aries
100736137 Cavia porcellus
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