Coagulation Factor V (F5) ELISA Kits

F5 encodes an essential cofactor of the blood coagulation cascade.

list all ELISA KIts Gen GeneID UniProt
Anti-Maus F5 F5 14067 O88783
Anti-Human F5 F5 2153 P12259
Anti-Ratte F5 F5 304929  
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Top Coagulation Factor V ELISA Kits auf antikoerper-online.de

Showing 10 out of 67 products:

Katalog Nr. Reaktivität Sensitivität Bereich Bilder Menge Anbieter Lieferzeit Preis Details
Maus 45 pg/mL 125 pg/mL - 8000 pg/mL 96 Tests Anmelden zum Anzeigen 13 bis 16 Tage
$682.11
Details
Human 0.78 ng/mL 3.12-200 ng/mL Typical standard curve 96 Tests Anmelden zum Anzeigen 13 bis 16 Tage
$910.56
Details
Schwein 0.027 0.027 Typical standard curve 96 Tests Anmelden zum Anzeigen 15 bis 18 Tage
$1,026.67
Details
Ratte 0.058 ng/mL 0.15 ng/mL - 10 ng/mL 96 Tests Anmelden zum Anzeigen 13 bis 16 Tage
$720.00
Details
Meerschweinchen 0.1 ng/mL 0.5-10 ng/mL   96 Tests Anmelden zum Anzeigen 15 bis 18 Tage
$707.14
Details
Kaninchen 0.1 ng/mL 0.5-10 ng/mL   96 Tests Anmelden zum Anzeigen 15 bis 18 Tage
$707.14
Details
Affe 0.1 ng/mL 2.5-50 ng/mL   96 Tests Anmelden zum Anzeigen 15 bis 18 Tage
$707.14
Details
Hund 0.1 ng/mL 0.5-10 ng/mL   96 Tests Anmelden zum Anzeigen 15 bis 18 Tage
$707.14
Details
Huhn
  96 Tests Anmelden zum Anzeigen 15 bis 18 Tage
$707.14
Details
Schaf
  96 Tests Anmelden zum Anzeigen 15 bis 18 Tage
$707.14
Details

Am meisten referenzierte Coagulation Factor V ELISA Kits

  1. Mouse (Murine) Coagulation Factor V ELISA Kit für Sandwich ELISA - ABIN415764 : Wang, Braun, Zhang, Norström, Thorlacius: Thrombin generation in abdominal sepsis is Rho-kinase-dependent. in Biochemical and biophysical research communications 2015 (PubMed)
    Show all 2 Pubmed References

  2. Mouse (Murine) Coagulation Factor V ELISA Kit für Sandwich ELISA - ABIN832489 : Schlegel, Lesurtel, Melloul, Limani, Tschuor, Graf, Humar, Clavien: ALPPS: from human to mice highlighting accelerated and novel mechanisms of liver regeneration. in Annals of surgery 2014 (PubMed)

Weitere ELISA Kits für Coagulation Factor V Interaktionspartner

Mouse (Murine) Coagulation Factor V (F5) Interaktionspartner

  1. Mice with the FVL mutation do not have increased spermatogenesis as compared to wildtype mice.

  2. Platelet-derived FV contributes to the control of angiogenesis and is likely associated with thrombin (zeige F2 ELISA Kits) generation in hind limb ischemia model.

  3. These findings reveal a novel biological function and mechanism of the protein C (zeige PROC ELISA Kits) pathway in which protein S and the aPC (zeige APC ELISA Kits)-cleaved form of fV are cofactors for anti-inflammatory cell signaling by aPC (zeige APC ELISA Kits) in the context of endotoxemia and infection

  4. Mice deficient in LMAN1 exhibit FV and FVIII deficiencies and liver accumulation of alpha1-antitrypsin.

  5. The FVL mutation does not influence coagulation activation, lung inflammation or survival in lethal influenza A.

  6. It suggested that there could be a combination of GLA (zeige GLA ELISA Kits) deficiency and FVL or other thrombosis-related gene defect in patients with genetic severe early-onset thrombosis.

  7. Data suggest that tissue factor and factor V induction by LPS may in part accelerate mesangioproliferative glomerulonephritis through activation of factor X and downstream proinflammatory and procoagulant mechanisms.

  8. The source of the FVL leading to accelerated thrombosis appears to be circulating, non-platelet-derived plasma FVL.

  9. FVL has the ability to improve the hemophilia A or B phenotype, but this effect is principally evident at the microcirculation level following a particular vascular injury.

  10. observations demonstrate a synergistic interaction between alpha-galactosidase A (zeige GLA ELISA Kits) deficiency and Factor V Leiden toward tissue fibrin deposition; concomitant mutations in these genes may increase the penetrance of vascular thrombotic events in humans

Human Coagulation Factor V (F5) Interaktionspartner

  1. Human FVL carriers have a higher total sperm count than non-carriers, with an adjusted mean difference of 31 x 106 (95%CI 0.2-61.7; P = 0.048).

  2. contribution of FVLeiden causing resistance to activated protein C (zeige PROC ELISA Kits) in Indian population is not as strong as previously reported in Western countries

  3. The frequencies of GA and AA genotypes and A allele of coagulation factor V (FV) 1691G>A polymorphism significantly increased in the lower extremity deep venous thrombosis (LDVT) group. Patients with LDVT carrying A allele (GA + AA) had both higher patency and recurrence rates than those carrying GG genotype. Coagulation factor V (FV) 1691G>A polymorphism may be associated with both the risk and prognosis of LDVT.

  4. Factor V Leiden-mutations were found in 16.8% of patients with cerebral sinus venous thrombosis and in 17.8% of patients with arterial ischemic stroke, which was significantly more frequent than in controls at a rate of 4.95% (ORs: 3.89 and 4.16).

  5. Double heterozygotes had a clinical presentation intermediate between FVL and prothrombin (zeige F2 ELISA Kits) mutation single carriers.

  6. increased frequency of factor V Leiden G1691A and prothrombin (zeige F2 ELISA Kits) G20210A mutation in venous thromboembolism patients indicates a significant role of these mutations in the development of VTE in the Kashmiri population

  7. genetic study of Factor V Leiden (G1691A) mutation in young ischemic strokes with large vessel disease in a South Indian population

  8. the routine screening of patients with NAIS for F5 G1691A, F2 G20210A and MTHFR (zeige MTHFR ELISA Kits) C677T gene mutations might not be justified, and additional prothrombotic mechanisms should be considered.

  9. There were no significant differences in factor V and factor II genotypes between infertile men and normal controls.

  10. FVBonn induces hypercoagulability via a combination of increased activation/procoagulant activity, decreased susceptibility to Activated protein C (zeige PROC ELISA Kits)-mediated inactivation, and slightly reduced APC (zeige APC ELISA Kits) cofactor activity

Cow (Bovine) Coagulation Factor V (F5) Interaktionspartner

  1. Data suggest factor Xa (FXa (zeige F10 ELISA Kits)) and factor Va (FVa) compete to bind FXa (zeige F10 ELISA Kits) on both PS model membranes and microparticles from activated platelets; this competition between dimerization/prothrombinase (zeige FGL2 ELISA Kits) complex formation appears to regulate blood coagulation.

Coagulation Factor V (F5) Antigen-Profil

Beschreibung des Gens

This gene encodes an essential cofactor of the blood coagulation cascade. This factor circulates in plasma, and is converted to the active form by the release of the activation peptide by thrombin during coagulation. This generates a heavy chain and a light chain which are held together by calcium ions. The activated protein is a cofactor that participates with activated coagulation factor X to activate prothrombin to thrombin. Defects in this gene result in either an autosomal recessive hemorrhagic diathesis or an autosomal dominant form of thrombophilia, which is known as activated protein C resistance.

Genbezeichner und Symbole assoziert mit Coagulation Factor V (F5) ELISA Kits

  • coagulation factor V (f5) Antikörper
  • coagulation factor V (F5) Antikörper
  • Ac2-120 Antikörper
  • AI173222 Antikörper
  • Cf-5 Antikörper
  • Cf5 Antikörper
  • FVL Antikörper
  • PCCF Antikörper
  • RPRGL1 Antikörper
  • THPH2 Antikörper

Bezeichner auf Proteinebene für Coagulation Factor V (F5) ELISA Kits

coagulation factor V , activated protein C cofactor , activated protein c cofactor , coagulation factor V jinjiang A2 domain , factor V Leiden , proaccelerin, labile factor

GENE ID SPEZIES
100534566 Oreochromis niloticus
14067 Mus musculus
2153 Homo sapiens
304929 Rattus norvegicus
280687 Bos taurus
100715882 Cavia porcellus
397217 Sus scrofa
100685858 Canis lupus familiaris
101123649 Ovis aries
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