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The protein encoded by CTNNBIP1 binds CTNNB1 and prevents interaction between CTNNB1 and TCF family members. Zusätzlich bieten wir Ihnen CTNNBIP1 Antikörper (49) und CTNNBIP1 Proteine (15) und viele weitere Produktgruppen zu diesem Protein an.
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miR (zeige MLXIP ELISA Kits)-215-5p is a negative regulator of adipocyte differentiation through post-transcriptional regulation of FNDC3B (zeige FNDC3B ELISA Kits) and CTNNBIP1 during early adipogenesis
miR (zeige MLXIP ELISA Kits)-29b plays a pivotal role in fetal mouse neurogenesis by regulating ICAT-mediated Wnt (zeige WNT2 ELISA Kits)/beta-catenin (zeige CTNNB1 ELISA Kits) signaling.
Mechanistic studies revealed that CTNNBIP1 suppresses Wnt (zeige WNT2 ELISA Kits)/beta-catenin (zeige CTNNB1 ELISA Kits) signaling and that miR (zeige MLXIP ELISA Kits)-215 promotes beta-catenin (zeige CTNNB1 ELISA Kits) activation and upregulates alpha-SMA (zeige SMN1 ELISA Kits) and fibronectin (zeige FN1 ELISA Kits) expression in TGF-beta1 (zeige TGFB1 ELISA Kits)-treated MMCs by targeting CTNNBIP1
Overexpression of Icat induces G(2) arrest and cell death in tumor cell mutants for adenomatous polyposis coli (zeige APC ELISA Kits), beta-catenin (zeige CTNNB1 ELISA Kits), or Axin (zeige AXIN1 ELISA Kits).
ICAT plays an important role in the anteriorization of neural cells by inhibiting the posteriorizing activity of Wnt (zeige WNT2 ELISA Kits) signaling
These results suggest that the loss of ICAT gene function causes the arrest of UB branching and the apoptotic death of MM cells, resulting in renal agenesis.
Inhibition of beta-catenin (zeige CTNNB1 ELISA Kits) signaling in articular chondrocytes causes increased cell apoptosis and articular cartilage destruction in Col2a1 (zeige COL2A1 ELISA Kits)-ICAT- transgenic mice.
Data demonstrated that ICAT was highly expressed in cervical cancer tissues and had a role in control of epithelial-mesenchymal transition (EMT) in cervical cancer cells. Its overexpression promoted cell proliferation, migration, invasion and resulted in EMT by disrupting the stability of E-cadherin/betacatenin complex.
Low PLD1 expression and high ICAT expression were significantly associated with increased survival in colorectal cancer patients.
The present work aims to investigate the relationship between the expression of AEG-1 (zeige MTDH ELISA Kits)(astrocyte elevated gene-1), b-FGF(basic-fibroblast growth factor (zeige FGF2 ELISA Kits)), beta-catenin (zeige CTNNB1 ELISA Kits), Ki-67 (zeige MKI67 ELISA Kits), TNF-alpha (tumor necrosis factor (zeige TNF ELISA Kits)-alfa) other prognostic parameters in DC (Ductal Carcinomas) and ductal intraepithelial neoplasm. We found a relationship between these factors.
Overexpression of ICAT promoted Caski cells' proliferation, arrested the cell cycle in the S phase and enhanced cell migration. Conclusion Overexpression of ICAT can promote the proliferation and migration of Caski cervical cancer cells.
Somatic mutation of beta-catenin (CTNNB1 (zeige CTNNB1 ELISA Kits)) is known to be crucial for Wilms tumor development in up to 15% of cases.
CTNNBIP1 expression correlated with longer overall survival in LAC (zeige LCT ELISA Kits) patients. This study reveals that miR (zeige MLXIP ELISA Kits)-214 plays a critical role in CSLC self-renewal and stemness by targeting CTNNBIP1.
Data show that microRNA miR (zeige MLXIP ELISA Kits)-215 activates beta-catenin (zeige CTNNB1 ELISA Kits) pathways by decreasing catenin beta interacting protein 1 (CTNNBIP1)expression in gliomas.
Simultaneous silencing of beta-catenin (zeige CTNNB1 ELISA Kits) and STAT3 (zeige STAT3 ELISA Kits) synergistically induces apoptosis and inhibits cell proliferation in HepG2 liver cancer cells.
Finally, pro-incubation with idebenone inhibited mitochondrial dysfunction induced by oxLDL through the mitochondrial-dependent apoptotic pathway and GSK3beta/beta-catenin (zeige CTNNB1 ELISA Kits) signalling pathways.
A potent beta-catenin (zeige CTNNB1 ELISA Kits) inhibitor, ICAT/CTNNBIP1 was a direct target of miR (zeige MLXIP ELISA Kits)-424-5p.
The protein encoded by this gene binds CTNNB1 and prevents interaction between CTNNB1 and TCF family members. The encoded protein is a negative regulator of the Wnt signaling pathway. Two transcript variants encoding the same protein have been found for this gene.
, beta-catenin-interacting protein 1
, catenin, beta interacting protein 1 a
, catenin beta interacting protein 1 b
, catenin, beta interacting protein 1
, inhibitor of beta-catenin and Tcf-4
, beta-catenin-interacting protein ICAT