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The protein encoded by AMOTL1 is a peripheral membrane protein that is a component of tight junctions or TJs.
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HECW2, a novel EC ubiquitin E3 ligase, plays a critical role in stabilizing endothelial cell-to-cell junctions by regulating AMOT (zeige AMOT ELISA Kits)-like 1 (AMOTL1) stability.
AMOTL1 Promotes Breast Cancer Progression and Is Antagonized by Merlin (zeige NF2 ELISA Kits).
phosphorylation-deficient S793Ala mutant of AMOTL1 showed a shorter half-life and conferred resistance to energy-stress-induced YAP (zeige YAP1 ELISA Kits) inhibition.
study defined eight additional recurrently mutated genes in SMZL; these genes are CREBBP, CBFA2T3, AMOTL1, FAT4, FBXO11, PLA2G4D, TRRAP and USH2A.
miR (zeige MLXIP ELISA Kits)-124 binds AmotL1 3'UTR (zeige UTS2R ELISA Kits) and down-regulates its expression repressing vasculogenic mimicry and cell motility in cervical cancer cells.
Scaffold proteins angiomotin (Amot (zeige AMOT ELISA Kits)) and angiomotin (zeige AMOT ELISA Kits)-related AmotL1 and AmotL2 were recently identified as negative regulators of YAP (zeige YAP1 ELISA Kits) and TAZ (zeige TAZ ELISA Kits) by preventing their nuclear translocation.
These results suggested that IFN-gamma exhibits anti-angiogenesis effects by regulating the expression of TNF-alpha-induced AmotL1 via NFkappaB in emphysema lungs.
AmotL1 and ZO-2 (zeige TJP2 ELISA Kits) are two candidates that could be harnessed to control the oncogenic function of YAP (zeige YAP1 ELISA Kits).
By yeast two-hybrid screening, angiomotin-like 1 (AmotL1) was identified as a host factor that interacts with the M protein (zeige MYOM2 ELISA Kits) of parainfluenza virus 5 (PIV5).
The angiomotin-like 1 is involved in actin-cytoskeleton-based processes, in part, via its interaction with a p80-angiomotin-containing complex and the actin cytoskeleton
Amot (zeige AMOT ELISA Kits) and AmotL1 have similar effects on endothelial migration and tight junction formation in vitro. In vivo Amot (zeige AMOT ELISA Kits) appears to control the cell polarity and AmotL1 affects the stability of cell-cell junctions.
The authors propose that AmotL1 is an essential effector of the N-cadherin (zeige CDH2 ELISA Kits) mediated endothelial/pericyte junctional complex.
Amot (zeige AMOT ELISA Kits), Amotl1, and Amotl2 are differentially expressed in uterine cells during the peri (zeige POSTN ELISA Kits)-implantation period.
Data show that MUPP1 interacts with angiomotin (Amot (zeige AMOT ELISA Kits)), JEAP/Amot (zeige AMOT ELISA Kits)-like 1 and MASCOT/Amot (zeige AMOT ELISA Kits)-like 2, and that all the Amot (zeige AMOT ELISA Kits)/JEAP family proteins also interacted with Patj, a close relative of MUPP1.
The protein encoded by this gene is a peripheral membrane protein that is a component of tight junctions or TJs. TJs form an apical junctional structure and act to control paracellular permeability and maintain cell polarity. This protein is related to angiomotin, an angiostatin binding protein that regulates endothelial cell migration and capillary formation.
angiomotin like 1
, angiomotin-like protein 1-like
, angiomotin-like protein 1
, junction-enriched and associated protein
, junction-enriched and -associated protein