Uncoupling Protein 1 (Mitochondrial, Proton Carrier) (UCP1) Antikörper

Antigen: Uncoupling Protein 1 (Mitochondrial, Proton Carrier) (UCP1)

Synonyme: UCP1, ARABIDOPSIS THALIANA PLANT UNCOUPLING MITOCHONDRIAL PROTEIN 1, ARABIDOPSIS THALIANA UNCOUPLING PROTEIN 1, ATPUMP1, ATUCP1, PLANT UNCOUPLING MITOCHONDRIAL PROTEIN 1, PUMP1, UCP, UNCOUPLING PROTEIN 1, SLC25A7, Ucp, Slc25a7, AI385626, Ucpa, Uncp, MGC108736, ucp, slc25a7

Klonalität: Polyklonal

Wirt: Ziege

Reaktivität: Human

Applikation: ELISA, Western Blot (WB)

Produktnummer: ABIN185512

Produktbeschreibung

Weitere Bezeichnung: UCP1

Gen-ID: 7350

Immunogen: C-EQLKRELSKSRQ

Format: Affinity-purified

Beschreibung: All names and symbols: UCP1, uncoupling protein 1 (mitochondrial, proton carrier) , HGNC:12517, SLC25A7, UCP , mitochondrial brown fat uncoupling protein, uncoupling protein 1. Official Symbol: UCP1. RefSeq number(s): NP_068605.1

Spezifität: Human

Synonyme: UCP1, ARABIDOPSIS THALIANA PLANT UNCOUPLING MITOCHONDRIAL PROTEIN 1, ARABIDOPSIS THALIANA UNCOUPLING PROTEIN 1, ATPUMP1, ATUCP1, PLANT UNCOUPLING MITOCHONDRIAL PROTEIN 1, PUMP1, UCP, UNCOUPLING PROTEIN 1, SLC25A7, Ucp, Slc25a7, AI385626, Ucpa, Uncp, MGC108736, ucp, slc25a7

Anwendungen

Applikationshinweise: Peptide ELISA: antibody detection limit dilution 1:128,000. Western Blot: Approx 35kDa band observed in Human Adipose lysates (calculated MW of 33.0kDa according to NP_068605.1). Recommended concentration: 0.1-1 µg/ml.

Reinigung: Purified from goat serum by ammonium sulphate precipitation followed by antigen affinity chromatography using the immunizing peptide.

Buffer: 0.5 mg/ml in Tris saline, 0.02% sodium azide, pH7.3 with 0.5% bovine serum albumin.

Lagerung: Aliquot and store at -20°C. Minimize freezing and thawing.

Forschungsgebiet: Krebs, Metabolismus

Beschränkungen: Nur für Forschungszwecke einsetzbar

Bilder

ABIN185512 (1 ug/ml) staining of Human Adipose lysate (35 ug protein in RIPA buffer). Primary incubation was 1 hour. Detected by chemiluminescence.

Publikationen

Publikationen: Ishigaki, Katagiri, Yamada et al.: "Dissipating excess energy stored in the liver is a potential treatment strategy for diabetes associated with obesity." in: Diabetes, Vol. 54, Issue 2, pp. 322-32, 2005 (PubMed).