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anti-Mouse (Murine) RBM24 Antikörper:
anti-Rat (Rattus) RBM24 Antikörper:
anti-Human RBM24 Antikörper:
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Seb4 is a target of MyoD (zeige MYOD1 Antikörper) during myogenesis and is required for myogenic gene expression.
Both Rbm24a and Rbm24b are also required for normal somite and craniofacial development.
Rbm24 involving in the regulation of cardiac gene expression, sarcomeric assembly, and cardiac contractility.
These results revealed that Stk38 (zeige STK38 Antikörper) kinase catalyzes the phosphorylation of Rbm24 during sarcomerogensis and this orchestrates accurate sarcomere alignment.
Study suggests that p63 (zeige CKAP4 Antikörper) is regulated by RBM24 via mRNA stability, which gives an insight into understanding how posttranscriptional regulatory mechanisms contribute to p63 (zeige CKAP4 Antikörper) expression.
RBM24 is sufficient to mediate muscle-specific (zeige EIF3K Antikörper) exon inclusion.
Rbm24 plays a crucial role in myogenic differentiation at least in part through a myogenin (zeige MYOG Antikörper)-dependent post-transcriptional regulatory pathway.
Data suggests that RBM24 binds to coding region of CHRM2 (zeige CHRM2 Antikörper) to regulate mRNA stability in cardiomyocytes; RBM24appears to drive changes in alternative splicing and in production of alternative transcript isoforms. (RBM24 = RNA binding motif protein-24; CHRM2 (zeige CHRM2 Antikörper) = cholinergic receptor muscarinic 2 (zeige CHRM2 Antikörper))
RBM24 interacted with the 5' TR of HBV pregenomic RNA to block 80S ribosome assembly on HBV pgRNA and thus inhibited core protein translation, whereas the interaction between RBM24 and the 3' TR enhanced the stability of HBV RNA.
embryonic stem cell switching into the differentiation state can be initiated by a tissue-specific splicing regulator (zeige PTBP2 Antikörper), Rbm24.
Our results show that tissue-specific expression of RBM24 can explain the neuron-specific aberrant splicing of IKBKAP (zeige IKBKAP Antikörper) exon 20 in familial dysautonomia, and that ectopic expression of RBM24 in neuronal tissue could be a novel therapeutic target of the disease.
RBM24 acts at least in part through upregulating the expression of miR-25, which in turn targets MALAT1 for degradation.
Authors have identified a new function of microRNA-222 leading to alteration of myogenic differentiation at the level of alternative splicing, and provided evidence that this effect is mediated by Rbm24 protein.
Study suggests that p63 (zeige RPE65 Antikörper) is regulated by RBM24 via mRNA stability, which gives an insight into understanding how posttranscriptional regulatory mechanisms contribute to p63 (zeige RPE65 Antikörper) expression.
Rbm24 is a novel player in the p53 (zeige TP53 Antikörper) pathway, which may be explored to restore proper cell cycle control in p53 (zeige TP53 Antikörper)-deficient tumors via p21 (zeige CDKN1A Antikörper).
it was found that this protein Rbm24 regulates myogenic differentiation via the p21 (zeige CDKN1A Antikörper) signal pathway.
Plays a role in myogenic differentiation by regulating MYOG levels. Binds to the 3'-UTR of MYOG mRNA and regulates its stability (By similarity).
RNA-binding motif protein 24-A
, RNA-binding protein 24-A
, RRM domain-containing protein SEB-4
, RRM-containing protein SEB-4
, RNA-binding protein 24
, RNA-binding motif protein 24
, RNA-binding region (RNP1, RRM) containing 1
, RNA binding motif protein 24
, RNA binding protein
, RNA-binding protein SEB-4
, RNA-binding region (RNP1, RRM) containing 6
, RNA-binding region-containing protein 6