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This study demonstrates that cyclic stretch disrupts tight junctions and adherens junctions by a JNK2 (zeige MAPK9 Proteine), c-Src (zeige SRC Proteine), and MLCK (zeige MYLK Proteine)-dependent mechanism.
Biochemical and physiological regulation of cardiac myocyte contraction by cardiac-specific myosin light chain kinase.
These findings suggest that hepatitis B virus X protein disrupts stress fiber formation and triggers apoptosis via an MLCK and a PTEN-dependent pathway.
Integrated cDNA expression analysis of failing human myocardia uncovered a novel protein kinase (zeige CDK7 Proteine), cardiac-specific myosin light chain kinase (cardiac-MLCK), which acts on MLC2v (zeige MYL2 Proteine).
data indicate that the IL-1beta (zeige IL1B Proteine)-induced increase in MLCK (zeige MYLK Proteine) protein expression and Caco-2 TJ permeability was mediated by an increase in MLCK (zeige MYLK Proteine) expression and activity and by an NF-kappaB (zeige NFKB1 Proteine)-dependent increase in MLCK (zeige MYLK Proteine) gene transcription
Acanthopanax senticosus polysaccharide pretreatment may be associated with inhibition of the NF-kappaB (zeige NFKB1 Proteine)/MLCK (zeige MYLK Proteine) pathway and concomitant amelioration of LPS (zeige TLR4 Proteine)-induced tight junction dysfunction of intestinal epithelium in endotoxemia
that MLCK (zeige MYLK Proteine) is competing with centrally active ROCK for a limiting pool of monomer with which to drive lamellar filament assembly
This study demonstrated that catchlike property during dynamic performance in mouse hindlimb muscles with (wild-type) and without MLCK(-/-) ) the primary mechanism for PTP (myosin phosphorylation).
Ablation of cardiac Mylk3 suggests that Mylk3 plays a pivotal role in the transition from compensated to decompensated hypertrophy via cardiac myocyte atrophy and sarcomeric disorganization.
Data suggest that galectin-3 (zeige LGALS3 Proteine) in hepatocellular carcinoma cells promotes cell migration by inducing RhoA GTPase activity, activating Mlck (zeige MYLK Proteine), up-regulating phosphorylation of MLC2 (myosin light chain 2 (zeige MYL12B Proteine)), and inducing actin rearrangement.
epithelial MLCK (zeige MYLK Proteine)-activated brush border fanning by IFN-gamma (zeige IFNG Proteine) promotes adherence and internalization of normally noninvasive enteric bacteria
MLCK may regulate the structure and the motility of stereocilia through F-actin polymerization.
Silencing TNFR2 (zeige TNFRSF1B Proteine), but not TNFR1 (zeige TNFRSF1A Proteine), resulted in restoration of epithelial tight junction (TJ) associated with decreased MLCK (zeige MYLK Proteine) expression.
Calcium regulation of MLCK (zeige MYLK Proteine) facilitates GLUT4 (zeige SLC2A4 Proteine)-mediated glucose uptake in 3T3-L1 adipocytes.
nmMLCK has a critical role in thrombin (zeige F2 Proteine)-induced endothelial cell inflammation and lung PMN (zeige TBCE Proteine) infiltration
Phosphorylation of cardiac myosin heavy chains (see MYH7B, MIM 609928) and light chains (see MYL2, MIM 160781) by a kinase, such as MYLK3, potentiates the force and rate of cross-bridge recruitment in cardiac myocytes (Chan et al., 2008
, cardiac-MyBP-C associated Ca/CaM kinase
, cardiac-MyBP-C-associated Ca/CaM kinase
, putative myosin light chain kinase 3
, cardiac MLCK
, cardiac-specific myosin light chain kinase
, myosin light chain kinase 3
, Putative myosin light chain kinase 3