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Human Monoclonal SLC5A3 Primary Antibody für ELISA, WB - ABIN520244
Hernández-Ochoa, Robison, Contreras, Shen, Zhao, Schneider: Elevated extracellular glucose and uncontrolled type 1 diabetes enhance NFAT5 signaling and disrupt the transverse tubular network in mouse skeletal muscle. in Experimental biology and medicine (Maywood, N.J.) 2012
Human Polyclonal SLC5A3 Primary Antibody für IHC (p) - ABIN2476515
Persson, Carlsson, Hambleton, Guidry: Immunoglobulins, lysozyme and lactoferrin in the teat and udder of the dry cow during endotoxin-induced inflammation. in Zentralblatt für Veterinärmedizin. Reihe B. Journal of veterinary medicine. Series B 1992
Show all 3 Pubmed References
KCNQ1 (zeige KCNQ1 Antikörper), KCNE2 (zeige KCNE2 Antikörper), and SMIT1 form reciprocally regulating complexes that affect neuronal excitability.
activation of NFAT5 (zeige NFAT5 Antikörper) and its downstream SMIT induction is important in protecting neurons from ischemia-induced oxidative stress
At low concentration, where SMIT1/2 activity gains importance, myo (zeige SYNPO2 Antikörper)-inositol uptake is reduced by ammonia-induced intracellular acidification
These findings add Mus (zeige TRPV6 Antikörper) spicilegus endogenous retrovirus HEMV as a second member to the murine leukemia virus subgroup that uses mSMIT1 to gain entry into cells.
brain SMIT1 levels were higher than SMIT2, however, regional differences were observed
Sodium/MI cotransporter 1 (SMIT1, the major cotransporter of MI) knockout (SMIT1(-/-)) mice with markedly reduced tissue myoinositol (MI) levels were used to characterize the essential roles of MI and SMIT1 in osteogenesis.
SLC5a3 is responsible for the Ins (zeige INS Antikörper) concentration gradient in the murine fetal-placental unit
used by Mus (zeige TRPV6 Antikörper) cervicolor Murine Leukemia Virus for cell entry
When Sertoli cells were cultured in a hypertonic medium, there was an increase in the expression of Isyna1 (zeige ISYNA1 Antikörper) and Slc5a3
SMIT-1 has a role in the development and function of the peripheral nerves
SMIT1 Modifies KCNQ (zeige KCNQ1 Antikörper) Channel Function and Pharmacology by Physical Interaction with the Pore
overexpression of the Na(+)/myo-inositol cotransporter (SMIT1) and myo (zeige SYNPO2 Antikörper)-inositol supplementation enlarged intracellular PI(4,5)P2 pools, modulated several PI(4,5)P2-dependent ion channels including KCNQ2 (zeige KCNQ2 Antikörper)/3 channels, and attenuated the action potential firing of superior cervical ganglion neurons
Hypotonic stress causes a significant upregulation of SLC5A3 gene expression as detected by semiquantitative RT-PCR and Western blot analysis.
A kinetic model has been generated for the transport mechanism of SMIT2 allowing insight into the transport of members of the LeuT structural family at the millisecond timescale.
JAK2 (zeige JAK2 Antikörper) contributes to the regulation of the myoinositol transporter SMIT.
Tumor cells silence SLC5A8 (zeige SLC5A8 Antikörper) and convert pyruvate into lactate as complementary mechanisms to avoid pyruvate-induced cell death.
Expression and/or function of SMIT2 may be reduced in diabetes mellitus, insulin (zeige INS Antikörper) resistance and polycystic ovary syndrome causing abnormal inositol meteabolism
Functions as a retroviral receptor for M813 murine leukemia virus (MuLV) entry. Prevents intracellular accumulation of high concentrations of myo-inositol (an osmolyte) that result in impairment of cellular function.
, sodium myo-inositol transporter 1
, sodium/myo-inositol cotransporter
, sodium/myo-inositol transporter 1
, solute carrier family 5 member 3
, solute carrier family 5, member 3
, solute carrier family 5, member alpha 3
, BCW2 protein
, human solute carrier family 5, member 3, Sodium/myo-inositol cotransporter
, sodium/myo-inositol cotransporter 1
, solute carrier family 5 (inositol transporter), member 3
, solute carrier family 5 (inositol transporters), member 3
, osmoregulatory Na+/myo-inositol cotransporter