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The protein encoded by SMAD9 is a member of the SMAD family, which transduces signals from TGF-beta family members. Zusätzlich bieten wir Ihnen SMAD9 Antikörper (81) und SMAD9 Proteine (7) und viele weitere Produktgruppen zu diesem Protein an.
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Mutations in SMAD9 gene encoding for transforming growth factor (TGF)-[beta] superfamily have been identified in Pulmonary Arterial Hypertension.
Phosphorylated Smad1/5/8/9 specifically bound to the BREs of Smad8/9 gene. The present study reveals that Smad8/9 is a unique R-Smad regulated through the BMP pathway at the mRNA level.
Case Report: hamartomatous olyposis and gastrointestinal ganglioneuromas with SMAD9 germline mutation that reduces PTEN expression.
a detailed computational model for TGF-beta (zeige TGFB1 ELISA Kits) signalling that incorporates elements of previous models together with crosstalking between Smad1 (zeige GARS ELISA Kits)/5/8 and Smad2 (zeige SMAD2 ELISA Kits)/3 channels through a negative feedback loop dependent on Smad7 (zeige SMAD7 ELISA Kits).
ATP production by NaF (zeige IL8 ELISA Kits) promotes hypertrophy-like changes via activation of phospho-Smad8
TNF (zeige TNF ELISA Kits) activated NF-kappaB (zeige NFKB1 ELISA Kits) pathway and inhibited the phosphorylation of Smad 1 (zeige GARS ELISA Kits)/5/8 and BMP-2 (zeige BMP2 ELISA Kits)-induced osteoblastic differentiation in BMMSCs
CREBZF, a novel Smad8-binding protein.
Data suggest that Smads 1, 5 and 8 as potential prognostic markers and therapeutic targets for mTOR (zeige FRAP1 ELISA Kits) inhibition therapy of prostate cancer.
Endoglin promotes transforming growth factor beta-mediated Smad 1/5/8 signaling and inhibits endothelial cell migration through its association with GIPC
Study described the first mutation in SMAD8 in a patient with IPAH, and suggested the involvement of SMAD8 in the pathogenesis of PAH.
Sphingosine 1 phosphate also up-regulated runt-related transcription factor 2 (Runx2 (zeige RUNX2 ELISA Kits)) expression through S1PR2 (zeige S1PR2 ELISA Kits)/RhoA (zeige RHOA ELISA Kits)/ROCK/Smad1 (zeige SMAD1 ELISA Kits)/5/8 signaling.
BetA can enhance in vivo osteogenic potentials of BMP2 (zeige BMP2 ELISA Kits), possibly via stimulating Smad 1 (zeige SMAD1 ELISA Kits)/5/8 and p38 (zeige CRK ELISA Kits) pathways, and combination of both agents can be considered as a therapeutic strategy for bone diseases.
findings suggest that Smad9 is a new type of transcriptional regulator in BMP signaling.
The findings demonstrate for the first time that KMUP-1 can promote osteoblast maturation and differentiation in vitro via BMP-2 (zeige BMP2 ELISA Kits)/Smad1 (zeige SMAD1 ELISA Kits)/5/8 and Wnt (zeige WNT2 ELISA Kits)/beta-catenin (zeige CTNNB1 ELISA Kits) pathways.
Data indicate that PDGF (zeige PDGFA ELISA Kits)-AA promotes mesenchymal stem cel migration via bone morphogenetic protein 2 (BMP2 (zeige BMP2 ELISA Kits))-smad (zeige SMAD1 ELISA Kits) proteins smad1 (zeige SMAD1 ELISA Kits)/5/8 activation requires lysosome-mediated degradation of PDGF (zeige PDGFA ELISA Kits) alpha Receptor (PDGFRalpha).
our data suggest a pivotal role for canonical BMP signaling demonstrating distinguished nonoverlapping function of pSmad8 with pSmad1 and pSmad5 in hfSCs regulation and hair morphogenesis but a redundant role in adult hair progenitors differentiation.
Data show that HtrA1 (zeige HTRA1 ELISA Kits) is produced during osteoclastogenesis, and negatively regulates osteoblast differentiation, osteoblast differentiation and BMP2 (zeige BMP2 ELISA Kits)-induced Smad1 (zeige SMAD1 ELISA Kits)/5/8, ERK1/2 (zeige MAPK1/3 ELISA Kits) and p38 (zeige CRK ELISA Kits) phosphorylation in pre-osteoblasts.
FGF2 (zeige FGF2 ELISA Kits) inhibited BMP9 (zeige GDF2 ELISA Kits)-induced osteogenic differentiation by blocking BMP9 (zeige GDF2 ELISA Kits)-induced Smads signaling and subsequently reducing Smads dependent up-regulation of ALK1 (zeige ACVRL1 ELISA Kits) and ALK2 (zeige ACRV1 ELISA Kits) in mesenchymal stem cells.
a detailed computational model for TGF-beta (zeige TGFB1 ELISA Kits) signalling that incorporates elements of previous models together with crosstalking between Smad1 (zeige SMAD1 ELISA Kits)/5/8 and Smad2 (zeige SMAD2 ELISA Kits)/3 channels through a negative feedback loop dependent on Smad7 (zeige SMAD7 ELISA Kits).
The protein encoded by this gene is a member of the SMAD family, which transduces signals from TGF-beta family members. The encoded protein is activated by bone morphogenetic proteins and interacts with SMAD4. Two transcript variants encoding different isoforms have been found for this gene.
MAD, mothers against decapentaplegic homolog 9
, Mothers against decapentaplegic-like protein 9
, mothers against decapentaplegic homolog 9
, SMAD family member 9
, mothers against decapentaplegic homolog 9-like
, MAD homolog 9
, Mothers against decapentaplegic, drosophila, homolog of, 9
, SMAD, mothers against DPP homolog 9
, mothers against DPP homolog 9
, SMAD 9
, SMAD, mothers against DPP 8
, TGF-beta signal transducer Smad8