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6-phosphogluconate dehydrogenase is the second dehydrogenase in the pentose phosphate shunt. Zusätzlich bieten wir Ihnen PGD Antikörper (81) und PGD Proteine (19) und viele weitere Produktgruppen zu diesem Protein an.
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identified that 6-phosphogluconate dehydrogenase (6PGD) is critically involved in the development of anaplastic thyroid cancer (ATC (zeige SRPK1 ELISA Kits)) resistance to doxorubicin. 6PGD inhibition disrupts metabolic reprogramming in doxorubicin-resistant ATC (zeige SRPK1 ELISA Kits) cells.
Studies indicate link between the 6-phosphogluconate dehydrogenase, oxidative pentose phosphate pathway (PPP) and lipogenesis through Ru-5-P-dependent inhibition of serine/threonine protein kinase LKB1 (zeige STK11 ELISA Kits)-AMPK (zeige PRKAA1 ELISA Kits) signalling.
copy number gain of the PGD gene was consistently identified in cervical cancers but not in the normal tissues
Results show that 6PGD activity is upregulated with increased lysine acetylation in primary leukemia cells from human patients, providing mechanistic insights into 6PGD upregulation in cancer cells.
Expression of 6PGD positively correlates with advancing stage of lung carcinoma. Knockdown of 6PGD by shRNA potently inhibits c-Met tyrosine phosphorylation.
Glycolytic cancer cells lacking 6-phosphogluconate dehydrogenase metabolize glucose to induce senescence
Data suggest that the affinity of Nox2 (zeige CYBB ELISA Kits) for NADPH (zeige NQO1 ELISA Kits) is increased in the presence of 6PGDH on cell stimulation.
Hsa (zeige CD24 ELISA Kits)-miR (zeige MLXIP ELISA Kits)-132 and -212 were negatively correlated with PGD in the prefrontal cortex of individuals affected with schizophrenia and bipolar disorders.
Taken together, these data suggest that ADD1/SREBP1c (zeige SREBF1 ELISA Kits) is a key transcription factor for 6PGDH gene expression and would coordinate glucose metabolism and lipogenesis for energy homeostasis.
6-phosphogluconate dehydrogenase is the second dehydrogenase in the pentose phosphate shunt. Deficiency of this enzyme is generally asymptomatic, and the inheritance of this disorder is autosomal dominant. Hemolysis results from combined deficiency of 6-phosphogluconate dehydrogenase and 6-phosphogluconolactonase suggesting a synergism of the two enzymopathies.
6-phosphogluconate dehydrogenase, decarboxylating
, 6-phosphogluconate dehydrogenase
, NADP-6 phosphogluconate dehydrogenase
, phosphogluconate dehydrogenase
, 6-phosphogluconate dehydrogenase (decarboxylating)
, 6-phosphogluconate dehydrogenase, decarboxylating-like