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The protein encoded by LST1 is a membrane protein that can inhibit the proliferation of lymphocytes.
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Increased expression of LST1 (zeige NFKBIL1 Antikörper) in GC-responders before therapy warrants further investigation into their role as potential predictors for the response to GC, and in the inflammatory process of rheumatoid arthritis.
Data indicate that the MHC (zeige HLAE Antikörper) class III gene LST1 (zeige NFKBIL1 Antikörper) 5' untranslated regions (5'-UTR (zeige UTS2R Antikörper)) contains several upstream open reading frames (uORFs).
Psoriasis was associated with promoter variant, rs9267502 of LST1 (zeige NFKBIL1 Antikörper).Promoter activity was examined and it was larger than that of the other genetic variants, most probably due to changes in binding affinity to transcription factors.
This is the first report demonstrating regulated LST1 (zeige NFKBIL1 Antikörper) expression in human intestinal epithelial and microvascular endothelial cells and in inflamed colonic tissue from IBD patients.
LST1 is expressed specifically in leukocytes of the myeloid lineage, where it localizes to the tetraspanin-enriched microdomains andit binds SHP-1 and SHP-2 phosphatases.
Study found association between extended LTA (zeige LTA Antikörper), TNF (zeige TNF Antikörper), LST1 (zeige NFKBIL1 Antikörper) and HLA gene haplotypes and rubella vaccine-induced immunity.
Human dendritic cells activate resting natural killer (NK) cells and are recognized via the NKp30 (zeige NCR3 Antikörper) receptor by activated NK cells.
Expression of this receptor, inhibited by TGF-beta1 (zeige TGFB1 Antikörper), affects NK-mediated killing of dendritic cells
The expression of LST1 (zeige NFKBIL1 Antikörper), specifically splice variants encoding soluble isoforms and NCR3 (zeige NCR3 Antikörper), was increased in rheumatoid-arthritis-affected blood and synovium and was associated with more severe inflammation in the synovium.
LST1 (zeige NFKBIL1 Antikörper) protein expression in human cell lines was found to be tightly regulated, allowing the expression of transmembrane isoforms but suppressing soluble isoforms
The absence of Lst1 leads to a slightly more susceptible phenotype. However, deletion of Lst1 in DBA (zeige RPS19 Antikörper)/2 J mice alone does not explain the high susceptibility of this strain to PR8M influenza infections.
The protein encoded by this gene is a membrane protein that can inhibit the proliferation of lymphocytes. Expression of this gene is enhanced by lipopolysaccharide, interferon-gamma, and bacteria. Several transcript variants encoding different isoforms have been found for this gene.
leukocyte-specific transcript 1 protein
, lymphocyte antigen 117
, leucocyte specific transcript 1