BCL2-Like 2 (BCL2L2) ELISA Kits

BCL2L2 encodes a member of the BCL-2 protein family. Zusätzlich bieten wir Ihnen BCL2-Like 2 Antikörper (142) und BCL2-Like 2 Proteine (17) und viele weitere Produktgruppen zu diesem Protein an.

list all ELISA KIts Gen GeneID UniProt
BCL2L2 599 Q92843
BCL2L2 12050 P70345
BCL2L2 60434  
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Top BCL2-Like 2 ELISA Kits auf antikoerper-online.de

Showing 7 out of 9 products:

Katalog Nr. Reaktivität Sensitivität Bereich Bilder Menge Lieferzeit Preis Details
Ratte 0.067 ng/mL 0.15 ng/mL - 10 ng/mL 96 Tests 13 bis 16 Tage
96 Tests 2 bis 3 Tage
Human 0.041 ng/mL n/a   96 Tests 11 bis 16 Tage
Ratte 0.04 ng/mL n/a   96 Tests 11 bis 16 Tage
Human 6 pg/mL n/a   96 Tests 11 bis 16 Tage
  96 Tests 31 bis 41 Tage
Human 0.188 ng/mL n/a   96 Tests 16 bis 21 Tage

Weitere ELISA Kits für BCL2-Like 2 Interaktionspartner

Human BCL2-Like 2 (BCL2L2) Interaktionspartner

  1. Upregulation of miR-335-5p expression enhanced the cisplatin sensitivity of ovarian cancer cells through suppressing BCL2L2, suggesting the potential of miR-335-5p/BCL2L2 axis as a therapeutic target for the cisplatin resistance of patients with ovarian cancer.

  2. miR-16 functions as a tumor suppressor miRNA to inhibit cell proliferation and induce apoptosis in oral squamous cell carcinoma through decreasing the oncogenes AKT3 and BCL2L2.

  3. HOTAIR up-regulates Bcl-w to enhance cell proliferation through sequestering miR-206 in breast cancer

  4. Data found that Bcl-w promotes epithelial-mesenchymal transition by increasing respiratory complex-I activity and reactive oxygen species (ROS) levels.

  5. the present study demonstrates that miR-422a may serve as a tumor suppressor in osteosarcoma via inhibiting BCL2L2 and KRAS translation both in vitro and in vivo Therefore, miR-422a could be developed as a novel therapeutic target in osteosarcoma.

  6. BCL2L2 knockdown was attenuated the effects of SNHG1 overexpression on cell viability, cell apoptosis and protein levels of cleaved caspase-3, cleaved caspase-9 and Bax in H2O2-treated human cardiomyocytes.

  7. Our comprehensive analysis indicates B-cell lymphomas commonly select for BCLW overexpression in combination with or instead of other antiapoptotic BCL2 family members.

  8. BCL-W contributes to the threshold of anti-apoptotic activity during mitosis

  9. we demonstrated that miR-126-5p plays an inhibitory role in human cervical cancer progression, regulating the apoptosis of cancer cells via directly targeting Bcl2l2.

  10. High expression of Bcl-w was associated with mesenchymal changes and invading populations in the glioblastoma multiforme; Bcl-w functions as a positive regulator of invasion by enhancing mesenchymal traits of glioblastoma multiforme, consequently contributing to malignancy.

  11. BCL2L2 was the virtual target of miR-133b, and we found a negative regulatory relationship between miR-133b and BCL2L2. MiR-133b and BCL2L2 interfered with the viability and apoptosis of cells.

  12. overexpression of miR-15a in the FaDu cells was associated with significantly decreased BCL2L2 and BCL2 expression and a significant increase in the apoptosis rate. The opposite results were observed in HPV-positive HSCC, where downregulation of miR-15a suppressed apoptosis

  13. we conclude that BER treatment reduces cisplatin resistance of gastric cancer cells by modulating the miR-203/Bcl-w apoptotic axis. BER may be a novel agent to enhance chemotherapeutic responses in cisplatin-resistant gastric cancer patients

  14. Genetic and pharmacological inhibition of BCL-W and BCL-XL causes directed elimination of senescent cells.

  15. Data show that BCL2-like 2 protein (BCL2L2) is a direct target of micrRNA miR-29b.

  16. these results indicate that miR-335 acts as a novel tumor suppressor to regulate ccRCC cell proliferation and invasion through downregulation of BCL-W expression.

  17. miR-15a acts as a tumor suppressor in NSCLC by directly targeting BCL2L2 and may serve as a potential diagnostic biomarker and therapeutic target for NSCLC.

  18. The crystal structures of BCL-W and BCL-XL, along with cellular, studies reveal critical features of the BH3 domains of pro-survival proteins that distinguish them functionally from their pro-apoptotic counterparts.

  19. over-expression of miR-195 sensitized resistant cells to DOX and enhanced cell apoptosis activity, all of which can be partly rescued by BCL2L2 siRNA and cDNA expression

  20. A structural basis for a conserved binding mechanism between p53DBD and the anti-apoptotic Bcl-2 family proteins.

Mouse (Murine) BCL2-Like 2 (BCL2L2) Interaktionspartner

  1. MYC regulates BCL-W expression through its transcriptional regulation of specific miR.

  2. Genetic and pharmacological inhibition of BCL-W and BCL-XL causes directed elimination of senescent cells.

  3. NF-kappaB p65/p52 signaling mediated the effects of GDNF on Bcl-2 and Bcl-w expressions

  4. Target-derived neurotrophins coordinate transcription of the antiapoptotic gene bclw with transport of bclw mRNA to the axon, and thereby prevent axonal degeneration in rat and mouse sensory neurons.

  5. By using human cancer cells and mouse embryonic fibroblasts, the study shows that Bcl-w functions in the mitochondria to increase the levels of reactive oxygen species (ROS), which subsequently stimulates the invasion-promoting signaling pathway.

  6. Bcl-2, Bcl-x(L) and Bcl-w have only minor roles in thymic lymphoma development elicited by defects in p53, and this may indicate that Mcl-1 and/or A1 may feature more prominently in this process.

  7. Induction of autocrine Bcl-w signaling through pericytes promoting endothelial cell survival is associated with melanoma development.

  8. Bcl-w(-/-) sensory neurons exhibit mitochondrial abnormalities, including alterations in axonal mitochondrial size, axonal mitochondrial membrane potential, and cellular ATP levels.

  9. Bcl-x(L) and Bcl-w target protein phosphatase 1alpha to Bad

  10. Bcl-2, Bcl-w, and Bax act in a redundant manner in regulating granulocyte survival and death

  11. amino acid and base sequences of c98 gene (bcl-2 homolog) expressed in newborn epidermal keratinocytes treated with IGF-1

  12. The crypt expression of Bax and Bcl-w is influenced by epidermal growth factor receptor signaling and is key for the regulation of apoptosis.

  13. These data identify Bcl-w as an endogenous neuroprotectant that may have seizure-suppressive functions.

  14. Bcl-w and Grid2 act in distinct pathways to regulate mitochondrial morphogenesis and control Purkinje cell dendrite development and synapse formation.

Cow (Bovine) BCL2-Like 2 (BCL2L2) Interaktionspartner

  1. Chromosome mapping of BCL2L2 gene in cows.

BCL2-Like 2 (BCL2L2) Antigen-Profil

Beschreibung des Gens

This gene encodes a member of the BCL-2 protein family. The proteins of this family form hetero- or homodimers and act as anti- and pro-apoptotic regulators. Expression of this gene in cells has been shown to contribute to reduced cell apoptosis under cytotoxic conditions. Studies of the related gene in mice indicated a role in the survival of NGF- and BDNF-dependent neurons. Mutation and knockout studies of the mouse gene demonstrated an essential role in adult spermatogenesis. Alternative splicing results in multiple transcript variants. Read-through transcription also exists between this gene and the neighboring downstream PABPN1 (poly(A) binding protein, nuclear 1) gene.

Genbezeichner und Symbole assoziert mit BCL2-Like 2 (BCL2L2) ELISA Kits

  • BCL2-like 2 (bcl2l2) Antikörper
  • BCL2 like 2 (BCL2L2) Antikörper
  • BCL2-like 2 (Bcl2l2) Antikörper
  • Bcl2-like 2 (Bcl2l2) Antikörper
  • AW048834 Antikörper
  • Bcl-w Antikörper
  • BCL-WEL Antikörper
  • BCL-WS Antikörper
  • BCL2-L-2 Antikörper
  • BCL2L2 Antikörper
  • Bclw Antikörper
  • c98 Antikörper
  • Gtrgal2 Antikörper
  • Gtrosa41 Antikörper
  • PPP1R51 Antikörper

Bezeichner auf Proteinebene für BCL2-Like 2 (BCL2L2) ELISA Kits

Bcl2-like 2 , BCL2-like 2 , apoptosis regulator BCL-W , bcl-2-like protein 2 , protein phosphatase 1, regulatory subunit 51 , apoptosis regulator Bcl-W , bcl2-L-2

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