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BCL2L2 encodes a member of the BCL-2 protein family.
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Our comprehensive analysis indicates B-cell lymphomas commonly select for BCLW overexpression in combination with or instead of other antiapoptotic BCL2 (zeige BCL2 ELISA Kits) family members.
BCL-W contributes to the threshold of anti-apoptotic activity during mitosis
we demonstrated that miR (zeige MLXIP ELISA Kits)-126-5p plays an inhibitory role in human cervical cancer progression, regulating the apoptosis of cancer cells via directly targeting Bcl2l2.
High expression of Bcl-w was associated with mesenchymal changes and invading populations in the glioblastoma multiforme; Bcl-w functions as a positive regulator of invasion by enhancing mesenchymal traits of glioblastoma multiforme, consequently contributing to malignancy.
BCL2L2 was the virtual target of miR (zeige MLXIP ELISA Kits)-133b, and we found a negative regulatory relationship between miR (zeige MLXIP ELISA Kits)-133b and BCL2L2. MiR (zeige MLXIP ELISA Kits)-133b and BCL2L2 interfered with the viability and apoptosis of cells.
overexpression of miR (zeige MLXIP ELISA Kits)-15a in the FaDu cells was associated with significantly decreased BCL2L2 and BCL2 (zeige BCL2 ELISA Kits) expression and a significant increase in the apoptosis rate. The opposite results were observed in HPV-positive HSCC, where downregulation of miR (zeige MLXIP ELISA Kits)-15a suppressed apoptosis
we conclude that BER treatment reduces cisplatin resistance of gastric cancer cells by modulating the miR (zeige MLXIP ELISA Kits)-203/Bcl-w apoptotic axis. BER may be a novel agent to enhance chemotherapeutic responses in cisplatin-resistant gastric cancer patients
Genetic and pharmacological inhibition of BCL-W and BCL-XL (zeige BCL2L1 ELISA Kits) causes directed elimination of senescent cells.
Data show that BCL2-like 2 protein (BCL2L2) is a direct target of micrRNA miR (zeige MLXIP ELISA Kits)-29b.
these results indicate that miR (zeige MLXIP ELISA Kits)-335 acts as a novel tumor suppressor to regulate ccRCC cell proliferation and invasion through downregulation of BCL-W expression.
MYC (zeige MYC ELISA Kits) regulates BCL-W expression through its transcriptional regulation of specific miR (zeige MLXIP ELISA Kits).
NF-kappaB (zeige NFKB1 ELISA Kits) p65 (zeige NFkBP65 ELISA Kits)/p52 signaling mediated the effects of GDNF on Bcl-2 (zeige BCL2 ELISA Kits) and Bcl-w expressions
Target-derived neurotrophins coordinate transcription of the antiapoptotic gene bclw with transport of bclw mRNA to the axon, and thereby prevent axonal degeneration in rat and mouse sensory neurons.
By using human cancer cells and mouse embryonic fibroblasts, the study shows that Bcl-w functions in the mitochondria to increase the levels of reactive oxygen species (ROS (zeige ROS1 ELISA Kits)), which subsequently stimulates the invasion-promoting signaling pathway.
Bcl-2 (zeige BCL2 ELISA Kits), Bcl-x(L (zeige BCL2L1 ELISA Kits)) and Bcl-w have only minor roles in thymic lymphoma development elicited by defects in p53 (zeige TP53 ELISA Kits), and this may indicate that Mcl-1 (zeige MCL1 ELISA Kits) and/or A1 may feature more prominently in this process.
Induction of autocrine Bcl-w signaling through pericytes promoting endothelial cell survival is associated with melanoma development.
Bcl-w(-/-) sensory neurons exhibit mitochondrial abnormalities, including alterations in axonal mitochondrial size, axonal mitochondrial membrane potential, and cellular ATP levels.
Bcl-x(L) and Bcl-w target protein phosphatase 1alpha to Bad
Bcl-2 (zeige BCL2 ELISA Kits), Bcl-w, and Bax (zeige BAX ELISA Kits) act in a redundant manner in regulating granulocyte survival and death
Chromosome mapping of BCL2L2 gene in cows.
This gene encodes a member of the BCL-2 protein family. The proteins of this family form hetero- or homodimers and act as anti- and pro-apoptotic regulators. Expression of this gene in cells has been shown to contribute to reduced cell apoptosis under cytotoxic conditions. Studies of the related gene in mice indicated a role in the survival of NGF- and BDNF-dependent neurons. Mutation and knockout studies of the mouse gene demonstrated an essential role in adult spermatogenesis. Alternative splicing results in multiple transcript variants. Read-through transcription also exists between this gene and the neighboring downstream PABPN1 (poly(A) binding protein, nuclear 1) gene.
, BCL2-like 2
, apoptosis regulator BCL-W
, bcl-2-like protein 2
, protein phosphatase 1, regulatory subunit 51
, apoptosis regulator Bcl-W