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In yeast, over 200 protein and RNA cofactors are required for ribosome assembly, and these are generally conserved in eukaryotes. Zusätzlich bieten wir Ihnen ADP-Ribosyltransferase 4 (Dombrock Blood Group) Proteine (14) und viele weitere Produktgruppen zu diesem Protein an.
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These results suggested that RIOK2 and NOB1 (zeige NOB1 Antikörper) may be potential targets in the treatment of Non-small cell lung cancer (NSCLC), and miR145 may be considered a therapeutic inhibitor of both genes.
our results indicate that miR (zeige MLXIP Antikörper)-330-5p inhibits non-small-cell lung cancer (NSCLC) cell growth through downregulation of NOB1 (zeige NOB1 Antikörper) expression. Our study suggests that miR (zeige MLXIP Antikörper)-330-5p may serve as a potential therapeutic target for the treatment of NSCLC
Authors identified that SNHG1 increased human nin one binding protein (NOB1 (zeige NOB1 Antikörper)), an oncogene (zeige RAB1A Antikörper), through sponging miR (zeige MLXIP Antikörper)-326 as competing endogenous RNA (ceRNA), finally prompting cell growth, migration and invasion in OS.
RIOK2 and NOB1 (zeige NOB1 Antikörper) were highly expressed in NSCLC cells and tissues, and their expression profiles were significantly associated with the Tumour Node Metastasis (TNM (zeige ODZ1 Antikörper)) clinical stage, lymph node metastasis, and differentiation. RIOK2 expression was correlated with NOB1 (zeige NOB1 Antikörper).
The proto-oncogene (zeige RAB1A Antikörper) NOB1 (zeige NOB1 Antikörper) as a direct target of miR (zeige MLXIP Antikörper)-326 in gastric cancer.
The expression of NOB1 (zeige NOB1 Antikörper) was also found to be higher in multidrug-resistant gastric cancer cells than that of sensitive cells. This novel MAb will be valuable for investigating the role of NOB1 (zeige NOB1 Antikörper) in carcinogenesis and multidrug resistance of gastric cancer.
CONCLUSION: Our results suggest that enhanced expression of NOB1 (zeige NOB1 Antikörper) related with poor early response to cisplatin-based chemotherapy in patients with advanced non-small cell lung cancer
NOB1 (zeige NOB1 Antikörper) plays an oncogenic role in laryngeal cancer cells through the regulation of JNK (zeige MAPK8 Antikörper) signaling pathway.
miR (zeige MLXIP Antikörper)-139-3p may act as a tumor suppressor that can inhibitcervical cancer cell proliferation, migration and invasion and induce cell apoptosis through down-regulation of NOB1 (zeige NOB1 Antikörper) expression.
we suggest that targeting miR (zeige MLXIP Antikörper)-192 and NOB1 (zeige NOB1 Antikörper) is a novel strategy which will assist in the development of new therapeutics that will be used in the future to prevent and treat prostate cancer.
findings demonstrate that Dok-3 (zeige DOK3 Antikörper) and Dok-1 (zeige DOK1 Antikörper)/-2 play distinctive but cooperative roles in osteoclastogenesis and protect mice from osteopenia, providing physiological and pathophysiological insight into bone homeostasis.
Platelets from Dok-1 (zeige DOK1 Antikörper)-/- mice displayed normal aggregation, activation of integrin alphaIIbbeta3, P-selectin (zeige SELP Antikörper) surface expression, and soluble fibrinogen binding. These findings indicate that Dok-1 (zeige DOK1 Antikörper) does not affect "inside-out" platelet signalling.
in the absence of PAG, Csk becomes more associated with alternative partners; i.e., phosphatase PTPN22 and Dok adaptors. Combining PAG deficiency with PTPN22 or Dok adaptor deficiency further enhances effector T cell responses. Unlike PAG, Cbl ubiquitin ligases inhibit the activation of naive, but not of effector, T cells.
These results reveal the critical involvement of Dok-1 (zeige DOK1 Antikörper) and Dok-2 in a negative-feedback loop that prevents overactivation of CD8 (zeige CD8A Antikörper)(+) T cells and promotes memory formation.
Thus, Dok-1 (zeige DOK1 Antikörper) and Dok-2 promote survival of glycoprotein B-specific CD8 (zeige CD8A Antikörper)(+) T cells in trigeminal ganglia latently infected with herpes simplex virus 1.
this study shows that Dok1 and Dok2 proteins are involved in the control of hematopoietic stem cell cycle regulation
Taken together, our results demonstrate that Dok-1 (zeige DOK1 Antikörper) and Dok-2 negatively regulate intestinal inflammation, apparently through the induction of IL-17A (zeige IL17A Antikörper) and IL-22 (zeige IL22 Antikörper) expression.
Dok1 (zeige DOK1 Antikörper) knockdown attenuated TLR2-induced NF-kappaB (zeige NFKB1 Antikörper) activation and IL-6 (zeige IL6 Antikörper) production in microglia.
Triple Dok1 (zeige DOK1 Antikörper) Doc2 (zeige DOC2A Antikörper) Doc3 knockout leads to spontaneous pulmonary inflammation with hallmarks of asthma.
Dok-1 (zeige DOK1 Antikörper) overexpression promotes the generation of an innate-like CD8 (zeige CD8A Antikörper)(+) T-cell population that expresses Eomesodermin (zeige EOMES Antikörper).
This gene encodes a protein that contains a mono-ADP-ribosylation (ART) motif. It is a member of the ADP-ribosyltransferase gene family but enzymatic activity has not been demonstrated experimentally. Antigens of the Dombrock blood group system are located on the gene product, which is glycosylphosphatidylinosotol-anchored to the erythrocyte membrane. Allelic variants, some of which lead to adverse transfusion reactions, are known.
ADP-ribosyltransferase 4 (DO blood group)
, ADP-ribosyltransferase C2 and C3 toxin-like 4
, Dombrock blood group carrier molecule
, NAD(P)(+)--arginine ADP-ribosyltransferase 4
, ecto-ADP-ribosyltransferase 4
, mono(ADP-ribosyl)transferase 4
, mono-ADP-ribosyltransferase 4
, NAD(P)(+)--arginine ADP-ribosyltransferase
, mono (ADP-ribosyl)transferase
, Dombrock blood group
, Dombrock glycoprotein
, dombrock molecule 1