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Four SNPs in IFNGR2 (zeige IFNGR2 Proteine), IL12RB1 (zeige IL12RB1 Proteine), IL12RB2 (zeige IL12RB2 Proteine), and IL23R were found to be associated with the MAP infection status of the resource population.
We conclude that variants in IL-23A (zeige IL23A Proteine) and IL-23R genes were associated with the risk of multiple sclerosis or other inflammatory demyelinating diseases.
IL-23 (zeige IL23A Proteine) R (rs7517847) and LEP (zeige LEP Proteine) (rs7799039) polymorphisms were associated with an increased risk but not affecting the clinical presentation of HCC (zeige FAM126A Proteine) among Egyptian patients
In a Turkish population, IL23R polymorphism is a risk factor for UC and is protective against CD.
The current study emphasizes the lack of association of IL23R and IL17 (zeige IL17A Proteine) polymorphisms with rheumatoid arthritis susceptibility in the Algerian population. However, the data showed the relationship between IL23R and IL17A (zeige IL17A Proteine) polymorphisms and the production of the different RF isotypes in rheumatoid arthritis patients
Haplotype of non-synonymous IL-23R variants increase susceptibility to severe malarial anemia in children of a holoendemic P. falciparum transmission area.
there is a positive association between the GWAS reported rs3762318 and leprosy, and SLC35D1 and IL23R might be the causal genes
this study identified susceptibility single nucleotide polymorphisms in IL23R with Behcet's disease in Han Chinese
HLA-B51 is a primary association marker in predisposition to Behcet disease, with IL-23R and IL12A being the additional strongest loci.
Study identified a possible silencer downstream of IL23R that includes the ankylosing spondylitis (AS)-associated SNP rs924080, which appears to modulate the functional effects of this regulatory element; confirmed the primary association of AS with rs11209032 in this region, but suggest that there could be a possible additional effect from rs924080 in a putative silencer on the same haplotype.
Study demonstrated susceptible or protective character of the investigated IL23R SNPs on the phenotype of ulcerative colitis, confirming the genetic association.
Epithelial IL-23R signaling enables protective IL-22 (zeige IL22 Proteine) responses in experimental colitis.
Study highlights the importance of IL-23R expression level and the instability of Foxp3 (zeige FOXP3 Proteine)+ regulatory T cells in the development of inflammatory bowel diseases.
Differential splicing generates antagonistic soluble IL-23R (sIL-23R) variants, which might limit IL-23-mediated immune responses. Here, ectodomain shedding of IL-23R was identified as an alternative pathway for the generation of sIL-23R.
Estrogen and progesterone decrease let-7f microRNA expression and increase IL-23/IL-23 (zeige IL23A Proteine) receptor signaling and IL-17A (zeige IL17A Proteine) production in patients with severe asthma.
Data show the contribution of IL-23/IL-23 (zeige IL23A Proteine) receptor and IL-7/IL-7 (zeige IL7 Proteine) receptor signaling in Th17 and Th1 (zeige HAND1 Proteine) cell dynamics during experimental autoimmune encephalomyelitis (EAE).
Study provides evidence that IL-23 (zeige IL23A Proteine)/IL-23R plays a critical role in brain ischemic injury
IL-23 (zeige IL23A Proteine) plus T-cell receptor signalling results in significant upregulation of IL-23 receptor expressed predominantly on CD4 (zeige CD4 Proteine)(hi)CD8 (zeige CD8A Proteine)(hi)CD3 (zeige CD3E Proteine)(+)alphabetaTCR(+) thymocytes, and leads to RORgammat-dependent apoptosis.
Homeostatic IL-23 receptor signaling limits Th17 response through IL-22 (zeige IL22 Proteine)-mediated containment of commensal microbiota.
Characterization of potent IL-23R small-peptide modulator, 2305 (teeeqqly), that decreases inflammatory response.
Although they share a common subunit, IL-23 (zeige IL23A Proteine) and IL-12 (zeige IL12A Proteine) receptors are not expressed on the same cell populations
The protein encoded by this gene is a subunit of the receptor for IL23A/IL23. This protein pairs with the receptor molecule IL12RB1/IL12Rbeta1, and both are required for IL23A signaling. This protein associates constitutively with Janus kinase 2 (JAK2), and also binds to transcription activator STAT3 in a ligand-dependent manner.
, interleukin 23 receptor
, interleukin-23 receptor-like
, IL-23 receptor
, interleukin 23 receptor-like