anti-ENOS (NOS3) Antikörper

anti-Nitric Oxide Synthase 3 (Endothelial Cell) Antikörper (NOS3)
Auf finden Sie aktuell 478 Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) Antikörper von 31 unterschiedlichen Herstellern. Zusätzlich bieten wir Ihnen ENOS Kits (97) und ENOS Proteine (23) und viele weitere Produktgruppen zu diesem Protein an. Insgesamt sind aktuell 612 ENOS Produkte verfügbar.
2310065A03Rik, cNOS, EC-NOS, ecNOS, ENOS, NOS, Nos-3, NOS3, NOSIII

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Am meisten referenzierte anti-ENOS Antikörper

  1. Amphibian Polyclonal ENOS Primary Antibody für IHC (fro), IHC (p) - ABIN152659 : Guo, Comhair, Zheng, Dweik, Eissa, Thomassen, Calhoun, Erzurum: Molecular mechanisms of increased nitric oxide (NO) in asthma: evidence for transcriptional and post-translational regulation of NO synthesis. in Journal of immunology (Baltimore, Md. : 1950) 2000 (PubMed)
    Zeige alle 7 Referenzen für 152659

  2. Human Polyclonal ENOS Primary Antibody für IF (p), IHC (p) - ABIN725690 : Li, Han, Guo, Li, Sang: Oxidative stress, endothelial dysfunction and inflammatory response in rat heart to NO? inhalation exposure. in Chemosphere 2011 (PubMed)
    Zeige alle 7 Referenzen für 725690

  3. Human Polyclonal ENOS Primary Antibody für IF (p), IHC (p) - ABIN746468 : Ikemura, Yamamoto, Motomura, Yamaguchi, Zhao, Iwasaki, Iwamoto: Preventive effects of the anti-vasospasm agent via the regulation of the Rho-kinase pathway on the development of steroid-induced osteonecrosis in rabbits. in Bone 2013 (PubMed)
    Zeige alle 4 Referenzen für 746468

  4. Human Polyclonal ENOS Primary Antibody für EIA, FACS - ABIN953739 : Yanamandra, Napper, Pramanik, Bocchini, Dhanireddy: Endothelial nitric oxide synthase genotypes in the etiology of retinopathy of prematurity in premature infants. in Ophthalmic genetics 2010 (PubMed)
    Zeige alle 4 Referenzen für 953739

  5. Human Polyclonal ENOS Primary Antibody für IF, WB - ABIN319319 : Wadman: GM advisory panel is slanted, say critics. in Nature 1999 (PubMed)
    Zeige alle 3 Referenzen für 319319

  6. Human Polyclonal ENOS Primary Antibody für FACS, IF - ABIN655773 : Ren, Sun, Deng, Zhang, Wu, Wei, Mani, Dou, Wang: The anti-inflammatory effect and potential mechanism of cardamonin in DSS-induced colitis. in American journal of physiology. Gastrointestinal and liver physiology 2015 (PubMed)
    Zeige alle 2 Referenzen für 655773

  7. Dog (Canine) Monoclonal ENOS Primary Antibody für WB - ABIN968916 : Michell, Chen Zp, Tiganis, Stapleton, Katsis, Power, Sim, Kemp: Coordinated control of endothelial nitric-oxide synthase phosphorylation by protein kinase C and the cAMP-dependent protein kinase. in The Journal of biological chemistry 2001 (PubMed)

  8. Human Polyclonal ENOS Primary Antibody für IF (p), IHC (p) - ABIN703315 : Papinska, Mordwinkin, Meeks, Jadhav, Rodgers: Angiotensin-(1-7) administration benefits cardiac, renal and progenitor cell function in db/db mice. in British journal of pharmacology 2015 (PubMed)

  9. Human Polyclonal ENOS Primary Antibody für EIA, WB - ABIN453769 : Rikova, Guo, Zeng, Possemato, Yu, Haack, Nardone, Lee, Reeves, Li, Hu, Tan, Stokes, Sullivan, Mitchell, Wetzel, Macneill, Ren, Yuan, Bakalarski, Villen, Kornhauser, Smith, Li, Zhou, Gygi, Gu et al.: Global survey of phosphotyrosine signaling identifies oncogenic kinases in lung cancer. ... in Cell 2007 (PubMed)

  10. Human Polyclonal ENOS Primary Antibody für EIA - ABIN453267 : Greif, Kou, Michel: Site-specific dephosphorylation of endothelial nitric oxide synthase by protein phosphatase 2A: evidence for crosstalk between phosphorylation sites. in Biochemistry 2002 (PubMed)

Weitere Antikörper gegen ENOS Interaktionspartner

Human Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) Interaktionspartner

  1. In the subgroup analysis based on ethnicity, both T786C and 4a4b eNOS polymorphisms could influence the risk of male infertility in Asian and Caucasian.[meta-analysis].

  2. Phosphocreatine exerts an antiapoptotic effect in HUVECs exposed to oxidative stress by methylglyoxal through the mitochondrial pathway and the modulation of PI3K (zeige PIK3CA Antikörper)/Akt (zeige AKT1 Antikörper)/eNOS and NF-kappaB (zeige NFKB1 Antikörper) signaling.

  3. NOS2 (zeige NANOS2 Antikörper) rs2779248, NOS2 (zeige NANOS2 Antikörper) rs1137933, and NOS3 (zeige NANOS3 Antikörper) rs3918188 genetic polymorphisms are potentially related to the susceptibility to type 2 diabetes mellitus (T2DM), and the rs1800783 polymorphism might be considered as genetic risk factors for diabetic nephropathy.

  4. This study determined the genetic polymorphisms of the eNOS (Glu298Asp) and Cav-1 (zeige CAV1 Antikörper) (G14713A and T29107A) genes in association with susceptibility risk in patients who had suffered from a large artery atherosclerotic stroke. It demonstrated that the eNOS Glu298Asp in combination with 14713A/29107A at the CAV1 (zeige CAV1 Antikörper) locus was associated with a significant risk of an large artery atherosclerotic stroke.

  5. The eNOS T786C polymorphism was associated with ISR in Chinese Han patients treated with DES (zeige DES Antikörper). Genotyping may be helpful to identify patients with higher risks of ISR after drug-eluting stent implantation.

  6. Meta-analysis. eNOS gene G894T polymorphism was related to pregnancy-induced hypertension risk, especially for Asians.

  7. Data suggest that, in T-lymphocytes, nitric oxide generated by eNOS S-nitrosylates Cys374 on ACTB (zeige ACTB Antikörper) and thus regulates activation/recruitment of PRKCQ (zeige PRKCQ Antikörper) at immune synapse; S-nitrosylation of beta-actin (zeige ACTB Antikörper) impairs actin binding to PFN1 (zeige PFN1 Antikörper) and regulates protein transport in lamellipodia. (eNOS = nitric oxide synthase (zeige NOS Antikörper) 3; ACTB (zeige ACTB Antikörper) = beta-actin (zeige ACTB Antikörper); PRKCQ (zeige PRKCQ Antikörper) = protein kinase C-theta (zeige PRKCQ Antikörper); PFN1 (zeige PFN1 Antikörper) = profilin-1 (zeige PFN1 Antikörper))

  8. The GG eNOS allele showed greater susceptibility to atherosclerosis.

  9. NOS3 (zeige NANOS3 Antikörper) TT894 genotype protected against doxorubicin cardiotoxicity in survivors of childhood acute lymphoblastic leukemia.

  10. These findings indicated that miR (zeige MLXIP Antikörper)-126 protects HCMECs from H/R-induced injury and inflammatory response by activating the PI3K (zeige PIK3CA Antikörper)/Akt (zeige AKT1 Antikörper)/ eNOS signaling pathway.

Pig (Porcine) Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) Interaktionspartner

  1. NOS3 was lowest in kidneys removed from live pigs, greater in kidneys from pigs with brain death, and greatest in kidneys from pigs with cardiac arrest.

  2. Rapid atrial pacing increases ADMA and down-regulates eNOS expression in an ADMA-independent manner.

  3. Icariin and icariside II may increase the eNOS expression through activating EGF-EGFR (zeige EGFR Antikörper) pathway in porcine aortic endothelial cells.

  4. Data suggest that pig sperm contain bNOS (zeige NOS1 Antikörper), iNOS (zeige NOS2 Antikörper), and eNOS; up-regulation of NOS (zeige NOS Antikörper) by leptin (zeige LEP Antikörper) during acrosome reaction and inhibition of acrosome reaction by inhibitors of nitric oxide synthases suggests these enzymes are involved in acrosome reaction.

  5. Periodic acceleration (pGz) acutely increases endothelial and neuronal nitric oxide synthase (zeige NOS1 Antikörper) expression in endomyocardium of normal swine.

  6. Data suggest that angiotensin II regulates nNOS (zeige NOS1 Antikörper) and eNOS expression and NOS (zeige NOS Antikörper) activity in afferent arterioles of the developing kidney via angiotensin 1 and 2 receptors.

  7. Endothelial nitric oxide synthase mRNA expression was elevated in gestational day 50 intrauterine growth retardation placenta and areola compared to gd50 control.

  8. Oligonol prevented the impairment of eNOS activity induced by high glucose through reversing altered eNOS phosphorylation status.

  9. Exercise training significantly increased total eNOS and phosphorylated levels of eNOS (pSer(1179)) in collateral-dependent arteries of experimental minipigs.

  10. Data show that wall shear stress increases with a decrease in artery diameter; eNOS protein contents decrease with an increase in diameter.

Cow (Bovine) Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) Interaktionspartner

  1. These data define the mechanism responsible for the repressive effects of nitric oxide (NO) on the transcriptional activity of beta-catenin (zeige CTNNB1 Antikörper) and link eNOS-derived NO to the modulation by VEGF (zeige VEGFA Antikörper) of Wnt (zeige WNT2 Antikörper)/beta-catenin (zeige CTNNB1 Antikörper)-induced endothelial cell proliferation.

  2. Endothelial cell autophagy maintains shear stress-induced nitric oxide generation via glycolysis-dependent ATP/P2Y1 receptor (zeige P2RY1 Antikörper) signaling to endothelial nitric oxide synthase.

  3. TNFalpha (zeige TNF Antikörper)reduces eNOS activity in bovine endothelial cells through serine 116 phosphorylation and Pin1 binding.

  4. Pomanox supplementation hinders hyperlipemia-induced coronary endothelial dysfunction by activating the Akt (zeige AKT1 Antikörper)/endothelial nitric oxide-synthase pathway and favorably counteracting vascular inflammation and oxidative damage.

  5. Signals that activate and phosphorylate eNOS are transmitted through distinct membrane domains in endothelial cells. Cholesterol domains, but not individual caveolae, mediate HDL stimulation of eNOS. VEGF and shear stress may act through caveolae.

  6. eNOS serine 1179 phosphorylation, in addition to enhancing NO production, also profoundly affects superoxide generation

  7. In addition to the heme center of eNOS oxygenase domain, we confirmed another O2.- generation site in the eNOS reductase domain and characterized its regulatory properties.

  8. A dimer-destabilized mutant of bovine eNOS where cysteine 101 was replaced by alanine, cloned and introduced into an eNOS-deficient mouse strain and that results provide the first in vivo evidence that eNOS-dependent oxidative stress is unlikely to be an initial cause of impaired endothelium-dependent vasodilation and/or a pathologic factor promoting intimal hyperplasia.

  9. Data show that resveratrol (Res) reversed caveolin-1 (Cav-1 (zeige CAV1 Antikörper))/endothelial nitric oxide synthase (eNOS) expressions in high glucose cultured bovine aortic endothelial cells (BAECs).

  10. Fluorescence decays of fluorescently labeled CaM (zeige KRIT1 Antikörper) bound to eNOS reveal four distinct conformational states and single-molecule fluorescence trajectories show multiple fluorescence states with transitions between states

Rabbit Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) Interaktionspartner

  1. Kidneys from circulation-restricted fetuses showed endothelial nitric oxide synthase phosphorylation inhibition.

  2. Antidiabetic drug pioglitazone protects the heart via activation of endothelial nitric oxide synthase (eNOS/Nos3) pathway in a rabbit model of myocardial infarction.

  3. VEGFR2 (zeige KDR Antikörper) activation was not affected by Slit2 (zeige SLIT2 Antikörper), but eNOS phosphorylation was diminished

  4. Data suggest distinct localizations of eNOS at the spiral arteries/arterial sinuses and iNOS (zeige NOS2 Antikörper) along the radial arteries in the developing placenta.

  5. Pulmonary ischaemia-reperfusion up-regulates inducible nitric oxide synthesis and/activity, which coincides with reduced endothelial nitric oxide synthase activity.

  6. eNOS dysregulation may be a central mechanism of impaired cardioprotection during hyperglycemia.

  7. Quercetin inhibited myocardial ischemia-reperfusion-induced NOS3 mRNA and protein expression.

Mouse (Murine) Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) Interaktionspartner

  1. these data suggest that CD NOS3 may be involved in the diuretic response to a water load in a sex-specific manner; the mechanism of this effect remains to be determined.

  2. CAV1 (zeige CAV1 Antikörper) KO mice have elevated IOP and reduced conventional outflow facility when compared with WT mice. CAV2 (zeige CAV2 Antikörper) expression was absent in CAV1 (zeige CAV1 Antikörper) KO mice, but we observed increased expressions of eNOS

  3. TNF-alpha (zeige TNF Antikörper) does not regulate eNOS activity in murine endothelial cells through serine 116 phosphorylation and Pin1 (zeige PIN1 Antikörper) binding.

  4. The eNOS expressed in smooth muscle cells in FAs (zeige FAS Antikörper) attenuates alpha-adrenergic vasoconstriction.

  5. Thioredoxin (zeige TXN Antikörper)-mediated deglutathionylation of eNOS in the coronary artery in vivo protected against reperfusion injury, even in the presence of normal levels of glutathione.

  6. Eph (zeige EPHA1 Antikörper)-B4 stimulates eNOS phosphorylation in vitro and may mediate vein graft adaptation by regulation of eNOS activity in vivo.

  7. Loss of endothelial NO plays an important role in the generation of p25 (zeige CDK5R1 Antikörper) and resulting tau phosphorylation in neuronal tissue. Endothelial nitric oxide synthase (eNOS)-deficient (eNOS-/-) mice display increased levels of p25 (zeige CDK5R1 Antikörper), an aberrant activator of cyclin-dependent kinase 5 (zeige CDK5 Antikörper), which is one of the primary kinases responsible for tau hyperphosphorylation, and a statistically higher p25/p35 (zeige CDK5R1 Antikörper) ratio.

  8. Pulse pressure finely regulates eNOS, controlling cerebral artery reactivity.

  9. TNF-alpha (zeige TNF Antikörper) induces vascular insulin (zeige INS Antikörper) resistance by mechanisms that involve positive modulation of PTEN (zeige PTEN Antikörper) and inhibition of Akt (zeige AKT1 Antikörper)/eNOS/NO signaling.

  10. Beta2AR (zeige ADRB2 Antikörper) overexpression enhances endothelial progenitor cells (EPC (zeige TCF21 Antikörper)) functions in vitro and enhances the vascular repair abilities of EPCs in vivo via the beta2AR (zeige ADRB2 Antikörper)/Akt (zeige AKT1 Antikörper)/eNOS pathway.

Guinea Pig Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) Interaktionspartner

  1. The present evidence indicated that the customary HBOT protocol may increase constitutive NOS expression.

ENOS (NOS3) Antigen-Profil

Beschreibung des Gens

Nitric oxide is a reactive free radical which acts as a biologic mediator in several processes, including neurotransmission and antimicrobial and antitumoral activities. Nitric oxide is synthesized from L-arginine by nitric oxide synthases. Variations in this gene are associated with susceptibility to coronary spasm. Multiple transcript variants encoding different isoforms have been found for this gene.

Alternative names and synonyms associated with ENOS (NOS3)

  • nitric oxide synthase 3 (endothelial cell) (NOS3) Antikörper
  • nitric oxide synthase 3 (endothelial cell) (nos3) Antikörper
  • endothelial nitric oxide synthase (23B) Antikörper
  • nitric oxide synthase 3, endothelial cell (Nos3) Antikörper
  • 2310065A03Rik Antikörper
  • cNOS Antikörper
  • EC-NOS Antikörper
  • ecNOS Antikörper
  • ENOS Antikörper
  • NOS Antikörper
  • Nos-3 Antikörper
  • NOS3 Antikörper
  • NOSIII Antikörper

Bezeichner auf Proteinebene für NOS3

nitric oxide synthase 3 (endothelial cell) , nitric oxide synthase, endothelial-like , EC-NOS , NOS type III , NOSIII , cNOS , constitutive NOS , endothelial NOS , nitric oxide synthase, endothelial , eNOS , endothelial nitric oxide synthase , endothelial nitric oxide synthase NOS3 , endothelial nitric oxide synthase 3

714231 Macaca mulatta
100018035 Monodelphis domestica
100486016 Xenopus (Silurana) tropicalis
443077 Ovis aries
4846 Homo sapiens
403784 Canis lupus familiaris
397557 Sus scrofa
287024 Bos taurus
443073 Ovis aries
100063339 Equus caballus
100009498 Oryctolagus cuniculus
18127 Mus musculus
24600 Rattus norvegicus
100135577 Cavia porcellus
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