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Human Monoclonal HMOX1 Primary Antibody für IF, IP - ABIN968130
Foresti, Clark, Green, Motterlini: Thiol compounds interact with nitric oxide in regulating heme oxygenase-1 induction in endothelial cells. Involvement of superoxide and peroxynitrite anions. in The Journal of biological chemistry 1997
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Hamster Polyclonal HMOX1 Primary Antibody für IP, IHC - ABIN223113
Song, Shih, Chan, Zhang: Suppression of annexin A11 in ovarian cancer: implications in chemoresistance. in Neoplasia (New York, N.Y.) 2009
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Human Monoclonal HMOX1 Primary Antibody für IF, IP - ABIN968129
Ishikawa, Navab, Leitinger, Fogelman, Lusis: Induction of heme oxygenase-1 inhibits the monocyte transmigration induced by mildly oxidized LDL. in The Journal of clinical investigation 1997
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Cow (Bovine) Monoclonal HMOX1 Primary Antibody für ICC, IF - ABIN361697
Brydun, Watari, Yamamoto, Okuhara, Teragawa, Kono, Chayama, Oshima, Ozono: Reduced expression of heme oxygenase-1 in patients with coronary atherosclerosis. in Hypertension research : official journal of the Japanese Society of Hypertension 2007
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Human Monoclonal HMOX1 Primary Antibody für BP, ELISA - ABIN257888
Machado, Malheiros, Adamy, Santos, Silva Filho, Nahas, Lemos: Protective response in renal transplantation: no clinical or molecular differences between open and laparoscopic donor nephrectomy. in Clinics (São Paulo, Brazil) 2013
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Cow (Bovine) Polyclonal HMOX1 Primary Antibody für WB - ABIN2782158
Hausmann, Paul, Kellermeier, Frey, Schölmerich, Falk, Menzel, Fried, Herfarth, Rogler: (GT)N dinucleotide repeat polymorphism of haem oxygenase-1 promotor region is not associated with inflammatory bowel disease risk or disease course. in Clinical and experimental immunology 2008
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Hamster Polyclonal HMOX1 Primary Antibody für IHC, IP - ABIN4319615
Heeba, Hamza, Hassanin: Induction of heme oxygenase-1 with hemin alleviates cisplatin-induced reproductive toxicity in male rats and enhances its cytotoxicity in prostate cancer cell line. in Toxicology letters 2016
Human Polyclonal HMOX1 Primary Antibody für ICC, IF - ABIN4319619
Fouad, Albuali, Jresat: Simvastatin treatment ameliorates injury of rat testes induced by cadmium toxicity. in Biological trace element research 2013
Human Polyclonal HMOX1 Primary Antibody für ELISA, WB - ABIN561294
Hong, Kim, Kim, Lee, Shin, Son, Han, Sung, Kwon: Apoptosis induction of 2'-hydroxycinnamaldehyde as a proteasome inhibitor is associated with ER stress and mitochondrial perturbation in cancer cells. in Biochemical pharmacology 2007
Human Polyclonal HMOX1 Primary Antibody für ICC, IF - ABIN441348
Bakkar, Kousari, Kovalik, Li, Bowser: RBM45 Modulates the Antioxidant Response in Amyotrophic Lateral Sclerosis through Interactions with KEAP1. in Molecular and cellular biology 2015
study evaluated the time-course of HO-1 and catalase (zeige CAT Antikörper) gene expressions in nodules and roots of soybean plants subjected to cadmium treatment
Data suggest expression of hsp32 is up-regulated in kidney epithelial cells upon in vitro exposure to heavy metal water pollutants (Cd, As) or proteasomal inhibitors (MG132, withaferin A, celastrol); heat shock may be synergistic factor.
this study provides a better understanding of the role of the HO-1/Carbon monoxide system in controlling heart function in lower vertebrates.
The induced expression of HO-1 by fenofibrate appeared to be essential for mediating the protective effects of fenofibrate, as the inhibition of HO-1 activity significantly diminished the protective effects of fenofibrate against the GM-mediated death of sensory hair cells in cochlea
Suggest that Gata-1 (zeige GATA1 Antikörper) and Nrf2a (zeige NFE2L2 Antikörper) play differential roles in regulating the heme degradation enzymes hmox1a/bvra (zeige BLVRA Antikörper)/bvrb (zeige BLVRB Antikörper) during an early developmental period of heightened cellular stress.
Bach1 (zeige BACH1 Antikörper) regulates the liver specificity and transience of the Nrf2a (zeige NFE2L2 Antikörper)-dependent induction of hmox1a and that heme mediates this regulation through Bach1 (zeige BACH1 Antikörper) inhibition based on its level in each tissue.
Data indicate that Heme oxygenase 1 (HY1) functioned negatively and acted upstream of ABSCISIC ACID-INSENSITIVE4 (ABI4) in drought signaling.
HY1 confers cadmium tolerance by decreasing nitric oxide production and improving iron homeostasis.
Data indicate that AtHO1 (HY1; heme oxygenase-1)-overexpressing plants generated more NO, whereas knock-down of AtHO1 expression reduced the level of nitric oxide (NO) in plants.
HY1 mutant exhibited progressive salt hypersensitivity.
Disrupting the binding of the AtHBP5 to haem oxygenase 1 (HY1) leads to oxidative stress.
Mutation of HY1 causes UV-C hypersensitivity by impairing carotenoid and flavonoid biosynthesis and the down-regulation of antioxidant defence.
HY1 and HY5 additively regulate the expression of light regulated genes and accumulation of chlorophyll and anthocyanin during early seedling development.
HY1 (Heme oxygenase 1) plays an important role in salt acclimation.
all members of the HO1 subfamily (HY1, HO3 and HO4) are active monomeric HOs and can convert haem to BV IXalpha using spinach Fd (ferredoxin) as an electron donor
HO2 (zeige HMOX2 Antikörper) has an activity high enough to substitute for HO1 under aerobic conditions.
Longer poly-GT repeats in the HMOX1 gene are more common in African Americans with gastroparesis.
the expression of HO-1 gene was modulated by the nuclear factor (erythroid-derived 2)-like 2 (Nrf2 (zeige NFE2L2 Antikörper)), which translocated from cytoplasm to nucleus after priming dose radiation and enhance the antioxidant level of cells.
Downregulation of inducible NO synthetase (iNOS (zeige NOS2 Antikörper)) resulted in downregulation of heme oxygenase 1 (HO-1), and, conversely, upregulation of iNOS (zeige NOS2 Antikörper) enhanced HO-1 activity.
The cessation of HO-1 induction occurred at the transcriptional and translational levels, and was mediated by the activation of the heme-binding transcriptional repressor Bach1 (zeige BACH1 Antikörper) and translational factor HRI (zeige EIF2AK1 Antikörper).
The induction of HO-1 expression by proteasome inhibition was reduced in PINK1 (zeige PINK1 Antikörper) G309D mutant cells.
there was little difference in HMOX1 genotypes distribution in patients with chronic obstructive pulmonary disease with different severity.
investigation of the possible role of HMOX1 in adipose tissue, adipogenesis and iron metabolism; adipose tissue HMOX1 was increased in obese participants and positively associated with obesity-related metabolic disturbances, and markers of iron accumulation, inflammation and oxidative stress
the HO-1 interactome in prostate cancer, is identified.
induction of HO1 by M. tuberculosis infection may be a mycobacterial virulence mechanism to enhance inflammation and bacterial growth
Data (including data from studies using cells from knockout mice) confirm that the NRF2 (zeige GABPA Antikörper)/HMOX1 axis is a critical pathway in the hyperglycemic-mediated dysregulation and inflammatory features of chondrocytes in type 2 diabetes-associated osteoarthritis. (NRF2 (zeige GABPA Antikörper) = nuclear factor [erythroid-derived 2]-like 2 protein; HMOX1 = heme oxygenase [decycling] 1)
findings collectively suggest that miR (zeige MYLIP Antikörper)-506 acts as a tumor suppressor via regulation of ROCK1 (zeige ROCK1 Antikörper) expression and may thus be a promising therapeutic target for HCC (zeige FAM126A Antikörper)
Exogenous administration of CO exacerbated allergic symptoms, resulting in higher levels of both CO and heme oxygenase-1 expression, and a further reduction in H2S levels and CSE (zeige CSE Antikörper) expression.
Down-regulation of HO-1 is associated with pulmonary arterial hypertension and right ventricular failure.
The study revealed the involvement of HO-1 in classical swine fever virus proliferation.
The protective properties of flavonoids, such as EGCG, against endothelial inflammation may be regulated in part though induction of HO-1 and subsequent activator protein-1 signaling.
Glutamate (zeige GRIN2C Antikörper) regulates Ca2 (zeige CA2 Antikörper)+ signals in smooth muscle cells of newborn piglet brain slice arterioles through astrocyte- and heme oxygenase-dependent mechanisms.
obalt protoporphyrin prevents postictal cerebral vascular dysfunction by upregulating HO-1.
Data demonstrate that lipopolysaccharides evoke a heat shock response, with an increase heat shock proteins 70 and Hsp32) and of VEGF (zeige VEGFA Antikörper), a specific endothelial cell growth factor (zeige FGF1 Antikörper).
Interaction of soluble factors in plasma possibly generated during PICSO are not responsible for upregulation of HO-1 and VEGF mRNA.
the data were consistent with HO-1 acting as an anti-viral factor and these findings suggested that induction of HO-1 may be a useful prevention and treatment strategy against BVDV infection.
These findings suggest that bronchiolar epithelial cells and macrophages up-regulate Nrf2 (zeige NFE2L2 Antikörper) expression early in the course of infection, which results in increased expression of HO-1 within these cells.
investigation of molecular mechanisms of microvascular complications in diabetes/hyperglycemia: regulation of HO-1 gene expression in aortic endothelial cells by advanced glycation end products
Sickle blood increases endothelial heme oxygenase activity.
data provide evidence for the involvement of the thioredoxin/thioredoxin (zeige TXN Antikörper) receptor system, in the regulation of haem oxygenase-1 expression in aortic endothelial cells during pro-oxidant challenge
Heme oxygenase-1 induction modulates hypoxic pulmonary vasoconstriction through upregulation of ecSOD/SOD3 (zeige SOD3 Antikörper).
HO-1 influences granulopoiesis through regulation of myelocyte proliferation. It is accompanied by changes in expression of transcriptionally active C/EBPbeta (zeige CEBPB Antikörper) protein.
EET-mediated increase in HO-1 levels require PGC-1alpha (zeige PPARGC1A Antikörper) expression
Data suggest the a decrease in migration of hematopoietic stem progenitor cells (HSPCs) can be explained by impaired calcium release in phospholipase C (zeige PLC Antikörper)-beta2 (PLC (zeige HSPG2 Antikörper)-beta2) knockout (KO) mice and a high baseline level of heme oxygenase 1 (HO-1), an enzyme that negatively regulates cell migration.
HO-1 expression is upregulated by hydrogen-rich water in a mouse model of in fl ammatory bowel disease, which also inhibits inflammatory factors, and oxidative stress and ER stress
Results establish an essential role for the receptor CX3CR1 (zeige CX3CR1 Antikörper) in gut (zeige GUSB Antikörper) macrophages in resolving inflammation in the intestine, where it helps protects against colitis-associated cancer by regulating HMOX-1 expression.
study demonstrates that human respiratory syncytial virus (hRSV) infection can be modulated by the expression of HO-1 both in vitro and in vivo; thus, HO-1 activity may play a critical role during hRSV infection
This is the first study to dissect the mechanism by which the antiadipogenic and anti-inflammatory lipid, Epoxyeicosatrienoic acid, induces the PGC-1alpha (zeige PPARGC1A Antikörper) signaling cascade and reprograms the adipocyte phenotype by regulating mitochondrial function and HO-1 expression.
The findings suggest that HO-1 by modulating TLR9 (zeige TLR9 Antikörper) expression in PMCs promotes pleural innate immunity in MRSA (zeige MSR1 Antikörper) empyema.
HO-1 regulates not only cytokine/chemokine (zeige CCL1 Antikörper) production in pregnant uteri but also myeloid cell receptor numbers.
The expression of oxidative stress markers were upregulated after light exposure but attenuated by cyanidin-3-glucoside and ferulic acid, which may be attributed to the elevated secretion and expression of heme oxygenase (HO-1) and nuclear factor erythroid-2 related factor 2 (Nrf2 (zeige NFE2L2 Antikörper)).
Kidneys from circulation-restricted fetuses showed reduced heme oxygenase-1 mRNA.
Ligustrazine injection possesses notable protective effects on ischemia/reperfusion injury in rabbits by increasing the expression of HO-1 in lung.
Results add new evidence for the importance of HO-1 in the genesis and development of atherosclerosis and provide several possible mechanisms underlying the anti-atherosclerosis effects of HO-1
the effect of HO-1 on the progression and stabilization of vulnerable plaques and the possible mechanism
Heme-L-lysinate could attenuate atherosclerotic progression through upregulating HO-1 and HSP70 (zeige HSP70 Antikörper) expression and increasing CO production.
These results suggest that HO-1 is important in limiting in-stent stenosis and can be regarded as a new therapeutic target.
Statins showed anti-atherosclerotic effects mediated by HO-1/eNOS (zeige NOS3 Antikörper), restoring the [NO]/[ONOO(-)] imbalance and reducing lipid peroxidation.
HO-1/CO system was activated and may be one of the protective signal pathway during pulmonary ischemia-reperfusion injury in rabbits.
HO-1 contributes to vascular repair by increasing circulating endothelial progenitor cells (EPCs) derived from the bone marrow.
Heme oxygenase, an essential enzyme in heme catabolism, cleaves heme to form biliverdin, which is subsequently converted to bilirubin by biliverdin reductase, and carbon monoxide, a putative neurotransmitter. Heme oxygenase activity is induced by its substrate heme and by various nonheme substances. Heme oxygenase occurs as 2 isozymes, an inducible heme oxygenase-1 and a constitutive heme oxygenase-2. HMOX1 and HMOX2 belong to the heme oxygenase family.
heme oxygenase (decycling) 1
, heme oxygenase 1
, heme oxygenase
, Heme oxygenase
, heat shock protein, 32-kD
, heat shock protein 32
, heme oxygenase (decyclizing) 1
, P32 protein
, heme oxygenase-1