Use your antibodies-online credentials, if available.
Keine Produkte auf Ihrer Vergleichsliste.
Ihr Warenkorb ist leer.
Alle Spezies anzeigen
Weitere Synonyme anzeigen
Wählen Sie die Spezies und Applikation aus
anti-Human PDE3B Antikörper:
anti-Mouse (Murine) PDE3B Antikörper:
anti-Rat (Rattus) PDE3B Antikörper:
Sie gelangen zu unserer vorgefilterten Suche.
Human Polyclonal PDE3B Primary Antibody für ELISA, WB - ABIN562146
Bazhin, Kahnert, Kimpfler, Schadendorf, Umansky: Distinct metabolism of cyclic adenosine monophosphate in regulatory and helper CD4+ T cells. in Molecular immunology 2010
Our results shed more light on phosphodiesterase 3A (zeige PDE3A Antikörper) activation and could be relevant to the treatment of severe hypertension in the general population.
Data indicate that genome-wide significant associations were found both at age 6 and 14 with single nucleotide polymorphisms (SNPs) on chromosome 11p15 in phosphodiesterase 3B, cGMP-inhibited protein PDE3B/cytochrome P-450 (zeige CYP Antikörper) CYP2R1 (zeige CYP2R1 Antikörper) genes.
PDE3B appears to be an important actor in regulation of energy metabolism and energy intake. Is inhibition of PDE3B a possible target for treatment of obesity or type 2 diabetes mellitus? [REVIEW]
PDE3B and the anti-lipolytic action of insulin (zeige INS Antikörper) may have a role in increased S-nitrosylation of adipose tissue in obesity
PDE3B is activated in response to agents relevant for beta-cell function and activation is linked to increased as well as decreased phosphorylation of the enzyme
PDE3B and EPAC1 (zeige RAPGEF3 Antikörper) bind directly through protein-protein interactions
Data suggest that five novel genes, LUM (zeige LUM Antikörper), PDE3B, PDGF-C (zeige PDGFC Antikörper), NRG1 (zeige NRG1 Antikörper) and PKD2 (zeige PKD2 Antikörper), have great potential for predicting the efficacy of cisplatin-based chemotherapy against OSCC.
Inhibition of PDE3B augments PDE4 (zeige PDE4A Antikörper) inhibitor-induced apoptosis in a subset of patients with chronic lymphocytic leukemia
Results explain the dual cyclic AMP (zeige APRT Antikörper)/cyclic GMP (zeige NT5C2 Antikörper) binding capabilities of phosphodiesterase 3B and provide the molecular basis for inhibitor specificity.
p87PIKAP (zeige PIK3R6 Antikörper) is a novel regulatory subunit of phosphoinositide 3-kinase gamma that is highly expressed in heart and interacts with PDE3B [p87PIKAP (zeige PIK3R6 Antikörper)]
Study concludes that CUGBP1 (zeige CELF1 Antikörper) is a critical regulator of insulin (zeige INS Antikörper) secretion via activating PDE3B.
Insulin (zeige INS Antikörper) induction of hypothalamic phosphodiesterase 3B activity and the reversal of the anorectic and body weight-reducing effects and stimulatory effect of insulin (zeige INS Antikörper) on hypothalamic proopiomelanocortin (zeige POMC Antikörper) gene expression by cilostamide suggest that activation of phosphodiesterase 3B is a novel mechanism of insulin (zeige INS Antikörper) signaling in the hypothalamus.
These results suggest the possibility for a direct role for the PDE3B pathway in mediating leptin (zeige LEP Antikörper) action in the hypothalamus.
Suggest that by reducing production of cAMP in adipocytes, PDE3B, IKKepsilon (zeige IKBKE Antikörper) and TBK1 (zeige TBK1 Antikörper) may contribute to the repression of energy expenditure during obesity.
Lack of PDE3B in brown adipocytes impairs the action of insulin (zeige INS Antikörper) on lipolysis.
these findings indicate that PDE3B deletion confers cardioprotective effects because of cAMP/PKA-induced preconditioning, which is associated with the accumulation of proteins with cardioprotective function in ICEFs.
Data suggest that Pde3b is involved in regulation of adipogenesis of adult stem cells into white adipocytes versus brown adipocytes; knockout mice lacking Pde3b form epididymal white adipose tissue with characteristics of brown adipose tissue.
The variation of PDE3B-expression in the brain might play a crucial pathophysiological role, and regulation of PDE3B production might protect against ischemic brain damage.
Calcineurin regulates degradation of phosphodiesterase 3B, in addition to phosphodiesterase 4D (zeige PDE4D Antikörper).
The PDE3B pathway has a direct role in mediating leptin (zeige LEP Antikörper) signaling in proopiomelanocortin (zeige POMC Antikörper) and neuropeptide Y (zeige NPY Antikörper) neurons, via a potential mechanism of leptin (zeige LEP Antikörper) signaling in the hypothalamus.
catalyzes the hydrolysis of cAMP and cGMP; regulates insulin secretory processes
phosphodiesterase 3B, cGMP-inhibited
, cGMP-inhibited 3',5'-cyclic phosphodiesterase B-like
, CGI-PDE B
, cGMP-inhibited 3',5'-cyclic phosphodiesterase B
, cyclic GMP-inhibited phosphodiesterase B
, cyclic nucleotide phosphodiesterase
, phosphodiesterase 3B cGMP inhibited