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These data identify suppression of CXCL2 (zeige CXCL2 ELISA Kits) and CXCL5 (zeige CXCL5 ELISA Kits) chemoattractant expression by 11beta-HSD1 (zeige HSD11B1 ELISA Kits) as a novel mechanism with potential for regulation of neutrophil recruitment to the injured myocardium, and cardiac fibroblasts as a key site for intracellular glucocorticoid regeneration during acute inflammation following myocardial injury.
IL-17RA (zeige IL17RA ELISA Kits) regulates CXL-1 and 5 production in the lungs during the adaptive response.
STAT3 (zeige STAT3 ELISA Kits) is required for maximal OSM (zeige OSM ELISA Kits)-induced CXCL5 (zeige CXCL5 ELISA Kits) expression.
CXCL5 (zeige CXCL5 ELISA Kits) has a role in neutrophil recruitment in TH17-mediated glomerulonephritis
Since adaptive villus growth occurs despite impaired CXCL5 (zeige CXCL5 ELISA Kits) expression and enhanced angiogenesis, this suggests that the growth of new blood vessels is not needed for resection-induced mucosal surface area expansion following massive SBR (zeige NXF1 ELISA Kits).
CXCL5 (zeige CXCL5 ELISA Kits) regulates pulmonary responses to infection and plays a central role in conferring clock control of inflammation.
findings demonstrated that CXCL1 (zeige CXCL1 ELISA Kits) and CXCL5 (zeige CXCL5 ELISA Kits) are increased in circulation with onset of T2D, are produced by islets under stress, and synergistically affect islet function, suggesting that these chemokines participate in pathogenesis of T2D.
TLR2 (zeige TLR2 ELISA Kits)-induced epithelial-derived CXCL5 (zeige CXCL5 ELISA Kits) is critical for polymorphonuclear leukocyte-driven destructive inflammation in pulmonary tuberculosis.
Our data suggest that the differential regulation of the chemokine CXCL5 between osteoblasts and endothelial cells upon FGF2 treatment is involved in Hematopoietic stem cell mobilization from the osteoblast niche or bone marrow to peripheral blood.
CXCL5 (zeige CXCL5 ELISA Kits) modulated macrophage activation, increased expression of the cholesterol efflux regulatory protein ABCA1 (zeige ABCA1 ELISA Kits), and enhanced cholesterol efflux activity in macrophages.
CXCL6 level is high in the serum of chronic hepatitis B patients.CXCL6 promotes human hepatocyte proliferation through the CXCR1-NFkappaB pathway and inhibits collagen I secretion by hepatic stellate cells.
The role of PITX2 in glaucoma may be mediated partly by regulating the expression of CXCL6 and BBS5 and thus affecting immune functions and intraocular pressure.
This is the first study to note that elevated systemic CCL5 (zeige CCL5 ELISA Kits) and CXCL6 were associated with moderate/severe lumbar disc degeneration. These chemokines may be systemic biomarkers for the diagnosis and monitoring of disc degeneration.
The neutrophil-recruiting chemokine (zeige CCL1 ELISA Kits) GCP-2/CXCL6 is expressed in cystic fibrosis (zeige S100A8 ELISA Kits) airways and retains its functional properties after binding to extracellular DNA.
HIF-1alpha (zeige HIF1A ELISA Kits) promotes HCC (zeige FAM126A ELISA Kits) progression and metastasis by upregulating CXCL6 transcription in HCC (zeige FAM126A ELISA Kits) cells.
Expression of the CXCL8 (zeige IL8 ELISA Kits), CXCL6 and CXCL1 (zeige CXCL1 ELISA Kits) genes are under the primary control of 1,25-dihydroxyvitamin D3 and its receptor.
Data suggest that expression of CXCL6 in trophoblasts is up-regulated during pregnancy development/placentation; CXCL6 expression inhibits trophoblast cell migration and invasion by down-regulating activity of MMP2 (matrix metalloproteinase 2 (zeige MMP2 ELISA Kits)).
Overexpression of GCP-2 (zeige FOLH1 ELISA Kits) in mesenchymal stem cells has the potential to enhance their angiogenic and survival properties.
Studies show that functional blocking of GCP-2 (zeige FOLH1 ELISA Kits) inhibits tumor growth and metastases.
The aim of this study was to investigate the role of the transcription factors, AP-1 (zeige FOSB ELISA Kits) and NF-kappaB (zeige NFKB1 ELISA Kits), in IL-6 (zeige IL6 ELISA Kits) and CXCL8 (zeige IL8 ELISA Kits) regulation in Jurkat T-cells.
mouse homolog is a member of the CXC chemokine family, is a neutrophil chemoattractant and is rapidly induced in response to muscle injury
C-X-C motif chemokine 5
, CXC chemokine LIX
, chemokine (C-X-C motif) ligand 6 (granulocyte chemotactic protein 2)
, cytokine LIX
, small-inducible cytokine B5
, granulocyte chemotactic protein-2
, small inducible cytokine B subfamily, member 5
, small inducible cytokine subfamily B, member 15
, C-X-C motif chemokine 6
, Small inducible cytokine subfamily B (Cys-X-Cys), member b
, chemokine alpha 3
, granulocyte chemotactic protein 2
, small inducible cytokine subfamily B (Cys-X-Cys), member 6 (granulocyte chemotactic protein 2)
, small-inducible cytokine B6
, chemokine (C-X-C motif) ligand 5
, inducible cytokine subfamily B (Cys-X-Cys), member 6