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indicate that MIEN1 overexpression may facilitate migration and invasion in breast cancer
In conclusion, this study revealed that miR-26b suppresses NSCLC metastasis by targeting MIEN1 via NF-kappaB/MMP-9/VEGF pathways, implicating a potential prognostic biomarker and therapeutic target for NSCLC treatment.
MIEN1, a novel interactor of Annexin A2 (zeige ANXA2 Proteine), promotes tumor cell migration by enhancing AnxA2 (zeige ANXA2 Proteine) cell surface expression.
MIEN1 expression not only positively correlates with oral cancer progression but also seems to be a critical molecular determinant in migration and invasion of oral cancer cells.
C35 might serve as a biomarker or therapeutic target for management of colorectal cancer.
While MIEN1 is a direct target of miR (zeige MLXIP Proteine)-940, miR (zeige MLXIP Proteine)-940 alters MIEN1 RNA.
results suggest that DeltaNp73 might cooperate with C35 to promote tumour progression and contribute to cisplatin resistance in ovarian cancer cells.
Results suggest that MIEN1 may have an important regulatory role in phosphorylation of AKT (zeige AKT1 Proteine) with its redox potential.
prenylation is required for the function of the C17orf37 protein in cancer cells
Inhibition of C35 gene expression by small interfering RNA induces apoptosis of breast cancer cells.
Increases cell migration by inducing filopodia formation at the leading edge of migrating cells. Plays a role in regulation of apoptosis, possibly through control of CASP3. May be involved in a redox-related process (By similarity).
hypothetical protein LOC548750
, hypothetical protein LOC100049740
, HBV X-transactivated gene 4 protein
, HBV XAg-transactivated protein 4
, protein C17orf37
, protein C17orf37 homolog