Use your antibodies-online credentials, if available.
Keine Produkte auf Ihrer Vergleichsliste.
Ihr Warenkorb ist leer.
Recognizes and binds the palindromic sequence 5'- TTGGCNNNNNGCCAA-3' present in viral and cellular promoters and in the origin of replication of adenovirus type 2. Zusätzlich bieten wir Ihnen NFIB Antikörper (54) und NFIB Proteine (6) und viele weitere Produktgruppen zu diesem Protein an.
Showing 3 out of 11 products:
low expression of NFIB was significantly associated with biologically more aggressive subtypes and, ultimately, poorer survival in lung adenocarcinoma patients
Data indicate that NFIB protein increases EZH2 (zeige EZH2 ELISA Kits) protein expression downstream of BRN2 (zeige POU3F2 ELISA Kits) protein, which further decreases MITF (zeige MITF ELISA Kits) protein levels.
Our study defines new molecular subclasses of ACC (zeige ACACA ELISA Kits) characterized by MYBL1 (zeige MYBL1 ELISA Kits) rearrangements and 5'-NFIB gene fusions.
In benign prostatic hyperplasia, luminal NFIB loss correlated with more severe disease. in androgen-dependent LNCaP prostate cancer cells demonstrated that 64.3% of NFIB binding sites are associated with AR and FOXA1 binding sites.
These data suggest that germline genetic variation at rs7034162 is important in osteosarcoma metastasis and that NFIB is an osteosarcoma metastasis susceptibility gene.
Further studies also discovered a conserved feedback regulatory circuitry formed by NFIB and miR (zeige MLXIP ELISA Kits)-365 in cutaneous squamous cell carcinoma development
multivariate analysis co-overexpression of NFIB, RANK and RANKL (zeige TNFSF11 ELISA Kits) significantly increased the risk of metastasis with an odds ratio of 13.59
TGF-beta (zeige TGFB1 ELISA Kits)-mediated suppression of ANT2 (zeige SLC25A5 ELISA Kits) through NF1 (zeige NF1 ELISA Kits)/Smad4 (zeige SMAD4 ELISA Kits) complexes contributes to oxidative stress and DNA damage during induction of cellular senescence.
Increased expression of NF1B is associated with pilocytic astrocytoma.
The data demonstrates that the autism spectrum disorder-associated A-C intronic haplotype of the ENGRAILED2 gene is a transcriptional activator, and both CUX1 (zeige CUX1 ELISA Kits) and NFIB mediate this activity.
Loss of Nfib resulted in a severe delay in intermediate progenitor cell generation while, conversely, overexpression of NFIX (zeige MLZE ELISA Kits) led to precocious intermediate progenitor cell generation.
Knockdown of NFIB in Drosha (zeige DROSHA ELISA Kits)-deficient hippocampal neural stem cells restores neurogenesis, suggesting that the Drosha (zeige DROSHA ELISA Kits)/NFIB mechanism robustly prevents oligodendrocyte fate acquisition in vivo.
Nfib is necessary and sufficient to increase chromatin accessibility at a large subset of the intergenic regions. Nfib promotes pro-metastatic neuronal gene expression programs and drives the metastatic ability of small cell lung cancer cells.
Loss of Nfib expression resulted in prostatic hyperplasia. NFIB knockdown studies demonstrated that loss of NFIB drives increased AR expression and superinduction of a subset of AR target genes.
Identify regulatory link between Nkx2-1 (zeige NKX2-1 ELISA Kits), miR (zeige MLXIP ELISA Kits)-200c, and the transcription factors Nfib and Myb (zeige MYB ELISA Kits), adding new players to the regulatory mechanisms driven by Nkx2-1 (zeige NKX2-1 ELISA Kits) in lung epithelial cells that may have implications in lung development and tumorigenesis.
NFIA (zeige NFIA ELISA Kits) and NFIB are endogenously expressed in gradients with high caudo-medial to low rostro-lateral expression ... this expression pattern deviates from that of PAX6 (zeige PAX6 ELISA Kits), suggesting that PAX6 (zeige PAX6 ELISA Kits) does not drive Nfi (zeige NFIC ELISA Kits) expression
NFIB-STAT5 (zeige STAT5A ELISA Kits) modules, possibly in conjunction with other transcription factors, control cell-specific genetic programs.
Study revealed both novel and broad functions of NFIB in regulating the generation of cortical projection neurons, including radial glial differentiation, neuronal migration and axon projection
downregulation of Ezh2 (zeige EZH2 ELISA Kits) transcription by NFIB is an important component of the process of neural progenitor cell differentiation during cortical development
transcription factor NFIB is an unanticipated coordinator of stem cell behaviour
Recognizes and binds the palindromic sequence 5'- TTGGCNNNNNGCCAA-3' present in viral and cellular promoters and in the origin of replication of adenovirus type 2. These proteins are individually capable of activating transcription and replication.
nuclear factor I/B
, nuclear factor 1 B-type-like
, nuclear factor I-B1
, CCAAT-box binding transcription factor
, CCAAT-box-binding transcription factor
, TGGCA-binding protein
, nuclear factor 1 B-type
, nuclear factor 1/B
, olfactory epithelium nuclear factor I-B