anti-Tricellulin (MARVELD2) Antikörper

anti-Tricellulin Antikörper (MARVELD2)
Auf finden Sie aktuell 30 Tricellulin (MARVELD2) Antikörper von 8 unterschiedlichen Herstellern. Zusätzlich bieten wir Ihnen Tricellulin Proteine (7) und Tricellulin Kits (1) und viele weitere Produktgruppen zu diesem Protein an. Insgesamt sind aktuell 40 Tricellulin Produkte verfügbar.
BC003296, DFNB49, MARVD2, MRVLDC2, Tric, Trica, Tricb, Tricc
Alle Antikörper anzeigen Gen GeneID UniProt
MARVELD2 365657  
MARVELD2 218518 Q3UZP0
MARVELD2 153562 Q8N4S9

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Meistgesuchte Reaktivitäten zu anti-Tricellulin (MARVELD2) Antikörper

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anti-Rat (Rattus) Tricellulin Antikörper:

anti-Mouse (Murine) Tricellulin Antikörper:

anti-Human Tricellulin Antikörper:

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Am meisten referenzierte anti-Tricellulin Antikörper

  1. Human Polyclonal Tricellulin Primary Antibody für EIA, IHC (p) - ABIN955328 : Tam, Spiess, Auyeung, Joachimiak, Chen, Poirier, Frydman: The chaperonin TRiC blocks a huntingtin sequence element that promotes the conformational switch to aggregation. in Nature structural & molecular biology 2009 (PubMed)
    Zeige alle 2 Referenzen für 955328

  2. Human Polyclonal Tricellulin Primary Antibody für ICC, IHC (fro) - ABIN261710 : Nguyen, Suzuki, Wakasugi, Hohchi, Hashida, Ohbuchi: Different expressions of erbB1/2 and tight junction proteins in hypertrophic inferior turbinates and nasal polyps. in European archives of oto-rhino-laryngology : official journal of the European Federation of Oto-Rhino-Laryngological Societies (EUFOS) : affiliated with the German Society for Oto-Rhino-Laryngology - Head and Neck Surgery 2013 (PubMed)
    Zeige alle 2 Referenzen für 261710

  3. Cow (Bovine) Polyclonal Tricellulin Primary Antibody für WB - ABIN2775617 : Chishti, Bhatti, Tamim, Lee, McDonald, Leal, Ahmad: Splice-site mutations in the TRIC gene underlie autosomal recessive nonsyndromic hearing impairment in Pakistani families. in Journal of human genetics 2008 (PubMed)

Weitere Antikörper gegen Tricellulin Interaktionspartner

Mouse (Murine) Tricellulin (MARVELD2) Interaktionspartner

  1. Findings suggest that both tricellular tight junctions proteins TRIC and LSR (zeige LSR Antikörper) have crucial roles for the differentiated cochlear cell survival.

  2. Tricellulin is a specific redox sensor and sealing element at 3-cell contacts and may compensate as a redox mediator for occludin (zeige OCLN Antikörper) loss at 2-cell contacts in vivo and in vitro.

  3. GFP-tagged angulin-1/LSR (zeige LSR Antikörper), in which serine 288 was substituted by alanine, was observed to be dispersed to bicellular junctions, indicating that phosphorylation of Ser288 is crucial for the exclusive localization of angulin-1/LSR (zeige LSR Antikörper) and tricellulin at tTJs.

  4. localization of angulin-1/LSR (zeige LSR Antikörper) and tricellulin at tricellular contacts of brain and retinal endothelial cells in vivo

  5. Loss of tricellulin prevented the coalition of the strands of the bicellular junction with the central element of the tricellular junction in the inner ear epithelia.

  6. The findings show the heterogeneity of the molecular organization of tTJs in terms of the content of LSR (zeige LSR Antikörper), ILDR1 (zeige ILDR1 Antikörper) or ILDR2, and suggest that ILDR1 (zeige ILDR1 Antikörper)-mediated recruitment of tricellulin to TCs is required for hearing.

  7. The tricellulin may be a component to maintain the integrity for PNS myelin function and morphology.

  8. marvelD3 (zeige MARVELD3 Antikörper), occludin (zeige OCLN Antikörper), and tricellulin define the tight junction-associated MARVEL protein family

  9. In this study, we identify tricellulin, the first integral membrane protein that is concentrated at the vertically oriented TJ strands of tricellular contacts.

  10. In the inner ear, tricellulin is concentrated at the tricellular tight-junctions in cochlear and vestibular epithelia, including the structurally complex and extensive junctions between supporting and hair cells.

Human Tricellulin (MARVELD2) Interaktionspartner

  1. Tricellulin-dependent macromolecule passage was comparably regulated in leaky and tight epithelia, but relative and absolute ion permeabilities of the tricellular tight junction (tTJ) were different.

  2. Data show that the E3 ubiquitin ligase (zeige MUL1 Antikörper) Itch forms a complex with tricellulin and thereby enhances its ubiquitination.

  3. we demonstrated that PLG (zeige PLG Antikörper) functions as a molecular bridge between tricellulin and streptococcal surface enolase (SEN (zeige MORF4 Antikörper)). The wild type strain efficiently translocated across the epithelial monolayer, accompanied by cleavage of transmembrane junctional proteins.

  4. The expressions of MARVELD2, CLDN1 (zeige CLDN1 Antikörper) and CLDN3 (zeige CLDN3 Antikörper) mRNA were significantly lower in cholesteatoma tissue and may be involved in epithelium permeability.

  5. High tricellulin expression is associated with hepatocellular carcinoma.

  6. MARVELD2 variants are responsible for about 1.5 % (95 % CI 0.8-2.6) of non-syndromic hearing loss in our cohort of 800 Pakistani families. The c.1331+2T>C allele is recurrent.

  7. The findings show the heterogeneity of the molecular organization of tTJs in terms of the content of LSR (zeige LSR Antikörper), ILDR1 (zeige ILDR1 Antikörper) or ILDR2, and suggest that ILDR1 (zeige ILDR1 Antikörper)-mediated recruitment of tricellulin to TCs is required for hearing.

  8. This study reveals the presence and subcellular distribution of tricellulin in brain endothelial cells.

  9. The dynamic behavior of tricellulin during the destruction and formation of tight junctions (TJ) under various extracellular calcium conditions seems to be closely associated with the barrier and fence functions of TJs.

  10. DFNB49 is an important cause of non-syndromic deafness in Czech Roma patients but not in the general Czech population.

Tricellulin (MARVELD2) Antigen-Profil

Beschreibung des Gens

The protein encoded by this gene is a membrane protein found at the tight junctions between epithelial cells. The encoded protein helps establish epithelial barriers such as those in the organ of Corti, where these barriers are required for normal hearing. Defects in this gene are a cause of deafness autosomal recessive type 49 (DFNB49). Two transcript variants encoding different isoforms have been found for this gene.

Alternative names and synonyms associated with Tricellulin (MARVELD2)

  • MARVEL domain containing 2 (Marveld2) Antikörper
  • MARVEL (membrane-associating) domain containing 2 (Marveld2) Antikörper
  • MARVEL domain containing 2 (MARVELD2) Antikörper
  • BC003296 Antikörper
  • DFNB49 Antikörper
  • MARVD2 Antikörper
  • MRVLDC2 Antikörper
  • Tric Antikörper
  • Trica Antikörper
  • Tricb Antikörper
  • Tricc Antikörper

Bezeichner auf Proteinebene für anti-Tricellulin (MARVELD2) Antikörper

MARVEL (membrane-associating) domain containing 2 , MARVEL domain-containing protein 2 , tricellulin

365657 Rattus norvegicus
218518 Mus musculus
153562 Homo sapiens
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