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Rat (Rattus) NOX1 ELISA Kit für Sandwich ELISA - ABIN431571
El-Naga: Pre-treatment with cardamonin protects against cisplatin-induced nephrotoxicity in rats: impact on NOX-1, inflammation and apoptosis. in Toxicology and applied pharmacology 2013
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Human NOX1 ELISA Kit für Sandwich ELISA - ABIN417371
Abdelmageed, El-Naga, El-Demerdash, Elmazar: Indole-3- carbinol enhances sorafenib cytotoxicity in hepatocellular carcinoma cells: A mechanistic study. in Scientific reports 2016
Rat (Rattus) NOX1 ELISA Kit für Sandwich ELISA - ABIN1081786
El-Naga, Mahran: Indole-3-carbinol protects against cisplatin-induced acute nephrotoxicity: role of calcitonin gene-related peptide and insulin-like growth factor-1. in Scientific reports 2016
These results are consistent with the hypothesis that antioxidants or NOX1/4 inhibition may be useful in blocking profibrotic effects of TGFbeta (zeige TGFB1 ELISA Kits) on dermal and gingival fibroblasts and warrant consideration for further development as potential antifibrotic agents
We demonstrated that rapid deletion of p22phox (zeige CYBA ELISA Kits) is possible and that the activity of Nox1 and Nox4 (zeige NOX4 ELISA Kits) but not Nox5 (zeige NOX5 ELISA Kits) exclusively depends on p22phox (zeige CYBA ELISA Kits).
PAK4 (zeige PAK4 ELISA Kits) downregulation decreased PPARgamma (zeige PPARG ELISA Kits)-mediated Nox1 expression and suppressed EMT (zeige ITK ELISA Kits) in IR-treated glioma cells.
Nox1-SHH (zeige SHH ELISA Kits)-Grem1 (zeige GREM1 ELISA Kits) signaling axis in pulmonary vascular endothelium that is likely to contribute to Pulmonary hypertension.
5-HT1B receptor-dependent cellular Src (zeige SRC ELISA Kits)-related kinase-Nox1-pathways contribute to vascular remodeling in pulmonary arterial hypertension.
NOX1 has a role in maintaining the proliferative phenotype of some colon cancers and has potential as a therapeutic target in this disease
NOX1 mRNA was undetectable in the gastric mucosa.
P38 MAPK (zeige MAPK14 ELISA Kits), phosphorylated P38 MAPK (zeige MAPK14 ELISA Kits), and RAC2 (zeige RAC2 ELISA Kits) regulated in mutual feedback and negative feedback regulatory pathways, resulting in the radioresistance of G0 cells.
NS5A contributes to reactive oxygen species production by activating expression of NADPH (zeige NQO1 ELISA Kits) oxidases 1 and 4 as well as cytochrome P450 2E1 (zeige CYP2E1 ELISA Kits).
our results highlight that the Nox1/AKT (zeige AKT1 ELISA Kits) signaling pathway plays an important role in cell survival in oral squamous cell carcinoma (OSCC) cells.
Both Nox1 and Duox2 (zeige DUOX1 ELISA Kits) induce exfoliation of crypt epithelium, but only Nox1 induces apoptosis. NOX1 and DUOX2 (zeige DUOX1 ELISA Kits) may be potential therapeutic targets for treating ileocolitis in human patients suffering inflammatory bowel disease (IBD).
we used the Ang II (zeige AGT ELISA Kits) infused hph-1 (zeige EGLN2 ELISA Kits) mice to examine the roles of NOX isoforms in the development of AAA (zeige AAAS ELISA Kits). We generated double mutants of hph-1 (zeige EGLN2 ELISA Kits)-NOX1, hph-1 (zeige EGLN2 ELISA Kits)-NOX2 (zeige CYBB ELISA Kits), hph-1 (zeige EGLN2 ELISA Kits)-p47phox, and hph-1 (zeige EGLN2 ELISA Kits)-NOX4 (zeige NOX4 ELISA Kits)
C-kit (zeige KIT ELISA Kits)-positive hematopoietic stem/progenitor cells expressed significantly higher of Nox1 and catalase (zeige CAT ELISA Kits), but less of lactoperoxidase (zeige LPO ELISA Kits) than in matured mononuclear cells.
Nox1 deletion reduces oxidant load and restores microvascular health in obese mice.
Data suggests that ROS (zeige ROS1 ELISA Kits) produced during primitive endoderm differentiation is dependent in part on increased NOX1 and NOX4 (zeige NOX4 ELISA Kits) levels, which is under the control of GATA6 (zeige GATA6 ELISA Kits). Furthermore, these results suggest that the combined activity of multiple NOX proteins is necessary for the differentiation of F9 cells to primitive endoderm.
Gp91phox (zeige CYBB ELISA Kits) NADPH oxidase modulates litter size by up-regulating mucin1 (zeige MUC1 ELISA Kits) expression in the uterus of mice.
NOX4 (zeige NOX4 ELISA Kits)- and NOX1-derived ROS (zeige ROS1 ELISA Kits) contribute to atherosclerosis in the aortic sinus of diabetic ApoE (zeige APOE ELISA Kits) knockout mice.
Data show that pan-NOX-inhibitor APX (zeige SHROOM1 ELISA Kits)-115 treatment decreased NADPH oxidase (Nox) Nox1, Nox2 (zeige CYBB ELISA Kits), and Nox4 (zeige NOX4 ELISA Kits) protein expression in the kidney.
NOX1 is selectively important in GPCR (zeige GPBAR1 ELISA Kits)-dependent intracellular ROS (zeige ROS1 ELISA Kits) generation but dispensable for GPVI (zeige GP6 ELISA Kits)-ITAM-dependent ROS (zeige ROS1 ELISA Kits) generation in blood platelets..
NADPH oxidase plays an important role in proMMP-2 expression and activation and MMP-2 (zeige MMP2 ELISA Kits) mediated SMC (zeige DYM ELISA Kits) proliferation occurs through the involvement of Spm (zeige NPC1 ELISA Kits)-Cer (zeige CBLN1 ELISA Kits)-S1P (zeige MBTPS1 ELISA Kits) signaling axis under ANG II (zeige AGT ELISA Kits) stimulation of PASMCs
Differential Roles of Protein Complexes NOX1-NOXO1 and NOX2-p47phox in Mediating Endothelial Redox Responses to Oscillatory and Unidirectional Laminar Shear Stress.
The current study was designed to determine mechanisms underlying 20-hydroxyeicosatetraenoic acid -stimulated nitric oxide (NO) release, and particularly the role of NADPH oxidase, reactive oxygen species, and PI3-kinase (zeige PIK3CA ELISA Kits) in stimulated NO release.
NEP (zeige MME ELISA Kits) expression is down-regulated in vascular endothelial cells by physiological laminar shear, possibly via a mechanotransduction mechanism involving NADPH oxidase-induced reactive oxygen species production.
The nox1 expression in zebrafish during early nervous system development from 12 to 48 hours post fertilization.
Over inhibition of the NADPH oxidase by the NADPH (zeige NQO1 ELISA Kits) Inhibitor DPI (zeige DSP ELISA Kits) may reduce the cell even the tissue in the progress of healing after the injury, in zebrafish liver cells.
This gene encodes a member of the NADPH oxidase family of enzymes responsible for the catalytic one-electron transfer of oxygen to generate superoxide or hydrogen peroxide. Alternatively spliced transcript variants encoding multiple isoforms have been observed for this gene.
NADH/NADPH mitogenic oxidase subunit P65-MOX
, NADPH oxidase homolog-1
, mitogenic oxidase (pyridine nucleotide-dependent superoxide-generating)
, mitogenic oxidase 1
, NADH/NADPH mitogenic oxidase subunit p65-mox
, GP91 phox homolog
, NADPH oxidase 1 alpha
, NADPH oxidase-1
, predicted NADPH oxidase-1
, NADPH oxidase 1