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Data show that TEAD family of transcription factors Tead1 and Tead4 (zeige TEAD4 Proteine)-regulated gene expression in differentiating primary myoblasts.
Cells with reduced Tead activity became losers, whereas cells with increased Tead activity became super-competitors. Tead directly regulated Myc (zeige MYC Proteine) RNA expression, and cells with increased Myc (zeige MYC Proteine) expression also became super-competitors.
The PDZ-binding motif of YAP (zeige YAP1 Proteine) is critical for YAP (zeige YAP1 Proteine)-mediated oncogenesis, and that this effect is mediated by YAP's co-activation of TEAD-mediated CTGF (zeige CTGF Proteine) transcription.
TEAD1 regulates C2C12 differentiation through negatively regulating the expression of Ccne1 (zeige CCNE1 Proteine), which can explain the transition between proliferation and differentiation.
TEAD1 is shown to be a mediator of skeletal muscle development.
increased TEAD-1 can induce characteristics of cardiac remodeling associated with cardiomyopathy and heart failure.
These results are consistent with two plausible models of cryptic MCAT (zeige MCAT Proteine) enhancer regulation by Pur alpha (zeige PURA Proteine), Pur beta (zeige PURB Proteine), and MSY1 (zeige YBX1 Proteine) involving either competitive single-stranded DNA binding or masking of MCAT (zeige MCAT Proteine)-bound transcription enhancer factor-1.
Transcription enhancer factor 1 binds multiple muscle MEF2 (zeige MEF2C Proteine) and A/T-rich elements during fast-to-slow skeletal (zeige MYL3 Proteine) muscle fiber type transitions
VITO-1 (zeige VGLL2 Proteine), a new scalloped interaction domain-containing protein, binds to TEF1 in vitro and strongly stimulates transcription of a reporter plasmid together with TEF-1
p38 (zeige CRK Proteine) MAPKs regulate TEF-1 and C/EBPbeta (zeige CEBPB Proteine) transcriptional activity in the absence of environmental stress.
MYC (zeige MYC Proteine) and TEAD activity is able to stratify different breast cancer subtypes in large panels of breast cancer patients.
Collectively, these results indicate that human papillomavirus 16 E6 induces upregulation of APOBEC3B (zeige APOBEC3B Proteine) through increased levels of TEADs, highlighting the importance of the TEAD-APOBEC3B (zeige APOBEC3B Proteine) axis in carcinogenesis.
Upregulation of transcriptional enhancer activator domain 1 was found in hepatocellular carcinoma tissues and inversely correlated with miR (zeige MLXIP Proteine)-590-3p. Our results indicate a tumor suppressor role of miR (zeige MLXIP Proteine)-590-3p in hepatocellular carcinoma through targeting transcriptional enhancer activator domain 1 and suggest its use in the diagnosis and prognosis of liver cancer.
TEAD1 could enhance the expression levels of SP1 (zeige PSG1 Proteine), by directly binding to its promoter.
TEAD1 mediates YAP1 (zeige YAP1 Proteine) chromatin-binding genome-wide.
show that the proangiogenic microfibrillar-associated protein 5 (MFAP5 (zeige MFAP5 Proteine)) is a direct transcriptional target of YAP (zeige YAP1 Proteine)/TEAD in cholangiocarcinoma cells transcription factors.
Melanoma reprogramming involves thousands of genomic regulatory regions underlying the proliferative and invasive states, identifying SOX10 (zeige SOX10 Proteine)/MITF (zeige MITF Proteine) and AP-1 (zeige FOSB Proteine)/TEAD as regulators, respectively.
TAZ (zeige TAZ Proteine) negatively regulate transcription of DeltaNp63 through TEAD1,2,3 and 4 transcription factors.
Our data suggest that AIC is a genetically heterogeneous disease and is not restricted to the X chromosome, and that TEAD1 mutations may be present in male patients.
Our findings suggest that genetic variants of Hippo pathway genes, particularly YAP1 (zeige YAP1 Proteine) rs11225163, TEAD1 rs7944031 and TEAD4 (zeige TEAD4 Proteine) rs1990330, may independently or jointly modulate survival of CM patients.
the XNTEF-1 and XDTEF-1 (zeige TEAD4 Proteine) mRNAS are predominantly detected in eye, embryonic brain, somites and heart; in animal cap assay, the two genes are activated by bFGF (zeige FGF2 Proteine) but are differently regulated by BMP4 (zeige BMP4 Proteine), and the muscle regulatory factor Mef2d (zeige MEF2D Proteine)
This gene encodes a ubiquitous transcriptional enhancer factor that is a member of the TEA/ATTS domain family. This protein directs the transactivation of a wide variety of genes and, in placental cells, also acts as a transcriptional repressor. Mutations in this gene cause Sveinsson's chorioretinal atrophy. Additional transcript variants have been described but their full-length natures have not been experimentally verified.
TEA domain family member 1
, TEA domain family member 1 (SV40 transcriptional enhancer factor)
, TEA domain family member 1-like
, transcriptional enhancer factor TEF-1-like
, transcription factor 13
, transcriptional enhancer factor TEF-1
, protein GT-IIC
, transcriptional enhancer factor 1