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Human KLF4 ELISA Kit für Sandwich ELISA - ABIN416925
Seipel, Marques, Bozzini, Meinken, Mueller, Pabst: Inactivation of the p53-KLF4-CEBPA Axis in Acute Myeloid Leukemia. in Clinical cancer research : an official journal of the American Association for Cancer Research 2016
zebrafih Klf4a is essential for the repression of intestinal cell proliferation
Proper heart valve formation in zebrafish critically depends on protein kinase D2 (zeige PKD2 ELISA Kits)-histone deacetylase 5 (zeige HDAC5 ELISA Kits)-Kruppel-like factor signaling.
PARP1 (zeige PARP1 ELISA Kits) recruits KLF4 to activate telomerase expression and stem cell pluripotency, indicating a positive regulatory role of the PARP1 (zeige PARP1 ELISA Kits)-KLF4 complex in telomerase expression in cancer and stem cells.
KLF2 (zeige KLF2 ELISA Kits) and KLF4 serve as important regulators that promote hemoglobin alpha (zeige HBA1 ELISA Kits) expression in the endothelium.
Loss of KLF4 expression is associated with T-cell acute lymphoblastic leukemia.
IRF4 (zeige IRF4 ELISA Kits) protects arteries against neointima formation by promoting the expression of KLF4 by directly binding to its promoter.
High KLF4 expression is associated with breast cancer tumorigenesis.
High KLF4 expression is associated with melanoma.
KLF4 transactivates Ch25h and LXR, thereby promoting the synergistic effects between ECs and macrophages to protect against atherosclerosis susceptibility.
findings evidence a positive correlation between SIRT1 (zeige SIRT1 ELISA Kits) and BCL6 (zeige BCL6 ELISA Kits) expression increase in follicular lymphomas (FL) . SIRT1 (zeige SIRT1 ELISA Kits) methylation decreases in FL and diffuse large-B cell lymphomas (DLBCL)and this parallels the increase of KLF4, DAPK1 (zeige DAPK1 ELISA Kits) and SPG20 (zeige SPG20 ELISA Kits) methylation
the use of KLF4 as a diagnostic and therapeutic target in cancer requires careful consideration of context.
KLF4 suppresses oncogenic TGF-beta (zeige TGFB1 ELISA Kits) signaling by activation of Smad7 (zeige SMAD7 ELISA Kits) transcription, and loss of KLF4 expression in primary hepatocellular carcinoma may contribute to activation of oncogenic TGF-beta (zeige TGFB1 ELISA Kits) signaling and subsequent tumor progression.
Klf4 is transcribed both maternally and zygotically and the transcript is ubiquitous in embryos during germ-layer formation
A long-range enhancer cluster controls Klf4 gene expression in mESCs
we revealed that one of the key functions of KLF4-induced TCL1 (zeige TCL1A ELISA Kits) during reprogramming is to promote the metabolic shift from oxidative phosphorylation to glycolysis.
KLF4 modulates development of BMI1 (zeige BMI1 ELISA Kits)-expressing intestinal stem cell-derived lineage following gamma-radiation-induced gut (zeige GUSB ELISA Kits) injury in mice.
dysregulation of Elovl6 (zeige ELOVL6 ELISA Kits)-driven long-chain fatty acid metabolism induces phenotypic switching of VSMC via ROS (zeige ROS1 ELISA Kits) production and AMPK (zeige PRKAA1 ELISA Kits)/KLF4 signaling that leads to growth arrest and downregulation of VSMC marker expression.
KLF2 (zeige KLF2 ELISA Kits) and KLF4 serve as important regulators that promote hemoglobin alpha (zeige HBA-A1 ELISA Kits) expression in the endothelium.
Together, our results suggest that KLF4 regulates asthma development in a TSLP (zeige TSLP ELISA Kits)- and fibrocyte-dependent manner.
Functional studies indicated that Klf4 mediates the self-renewal-promoting effects of Gbx2 (zeige GBX2 ELISA Kits), because knockdown of Klf4 expression abrogated the ability of Gbx2 (zeige GBX2 ELISA Kits) to maintain the undifferentiated state of mESCs.
Genetic inactivation of Klf4 in perivascular cells decreased formation of a pre-metastatic niche and metastasis. KLF4-dependent perivascular cell plasticity mediates pre-metastatic niche formation and metastasis.
miR (zeige MYLIP ELISA Kits)-92a coregulates KLF4 and KLF2 (zeige KLF2 ELISA Kits) expression in arterial endothelium and contributes to phenotype heterogeneity associated with regional atherosusceptibility and protection in vivo.
a transcription factor that works with Sp1 to activate the Laminin gamma1 chain gene
Krueppel-like factor 4
, Kruppel-like transcription factor 4a
, Kruppel-like factor 4 (gut)
, Kruppel-like factor 4
, endothelial Kruppel-like zinc finger protein
, epithelial zinc finger protein EZF
, gut-enriched krueppel-like factor
, Kruppel-like transcription factor
, blood island enriched kruppel like factor
, Kruppel-like factor 4 (Gut enriched kruppel-like factor) (Epithelial zinc-finger protein EZF)