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Human AMH Protein expressed in HEK-293 Cells - ABIN2714759
Signorile, Petraglia, Baldi: Anti-mullerian hormone is expressed by endometriosis tissues and induces cell cycle arrest and apoptosis in endometriosis cells. in Journal of experimental & clinical cancer research : CR 2014
nuclear receptor subfamily 5, group A, member 1b is a new candidate for sex determination and differentiation in a way similar to steroidogenic factor 1 (zeige NR5A1 Proteine), possibly involving AMH
zebrafish Anti-Mullerian hormone (Amh) is regulated by sox9a, sox9b, and cyp19a1a during gonad development
amh is a candidate gene down-regulating cyp19a1a, leading to "juvenile ovary-to-testis" transformation.
It was shown that the male-to-female sex reversal phenotype in hotei medaka mutants is not a direct consequence of anti-Mullerian hormone signaling in supporting cells, but is instead mediated by germ cells.
Data suggest that young men and young women initially have similar levels of circulating levels of AMH, but women lose their AMH in parallel with the decrease in their ovarian reserve. Circulating AMH in men is negatively associated with all-cause mortality; men with higher levels of AMH may outlive men with lower levels of AMH. [EDITORIAL]
In young women with polycystic ovary syndrome, low AMH levels predict a greater risk of metabolic syndrome.
MIS/AMH inhibits ovarian cancer by deregulating the Wnt (zeige WNT2 Proteine) signal pathway via the beta-catenin interacting protein (ICAT (zeige CTNNBIP1 Proteine)). MIS/AMH upregulated ICAT (zeige CTNNBIP1 Proteine) in ovarian cancer cell line which caused decreased cell viability, cell cycle arrest and apoptosis.
The AMH level peaked at or before ovulation in most women, trended down with natural pregnancies, and consistently increased or decreased in women with a viable pregnancy after therapy. Nonviable pregnancies showed erratic AMH patterns.
Before 35 years of age, women with type 1 diabetes have lower AMH levels than women without diabetes.
Suggest that activation of AMH by proteolytic enzymes is largely stable throughout the ovarian cycle. However, there is a subtle but robust decrease in the level of proAMH relative to AMHN,C in the acute postovulatory period.
Age is correlated with AMH, but it accounts for only a portion of the variation seen in reproductive age African American women.
AMH was inversely correlated with age in both the fertile and infertile populations and there was no significant difference between the fertile and infertile populations in terms of AMH.
Data suggest that, in adolescents (as with adults) with PCOS (polycystic ovary syndrome), serum levels of 17-hydroxyprogesterone are variable as a characteristic of the disorder; 17-hydroxyprogesterone responsiveness to r-hCG (zeige CGA Proteine) (recombinant human chorionic gonadotropin, a fertility agent) is not correlated to serum levels of AMH (anti-Mullerian hormone).
Anti-Mullerian hormone (AMH) and inhibin-A (zeige INHA Proteine) (INH-A) levels were found to be significantly higher in the polycystic ovary syndrome (PCOS) group compared to the controls.
AMH increases GnRH-dependent LH pulsatility and secretion, supporting a central action of AMH on GnRH neurons.
AMH and FOXL2 (zeige FOXL2 Proteine) collaboratively work to reserve ovarian follicles. AMH is an endogenous target gene of FOXL2 (zeige FOXL2 Proteine).
Up-regulation of SOX9 (zeige SOX9 Proteine) in sertoli cells from testiculopathic patients accounts for increasing anti-mullerian hormone expression via impaired androgen receptor (zeige AR Proteine) signaling.
Male mice require AMH to undergo normal social development.
Data show that Purkinje cells express receptors for Mullerian inhibiting substance (MIS), and that MIS(-/-) male mice have female-like numbers of Purkinje cells and a female-like size to other parts of their cerebellum.
MIS may be involved in anterograde rather than autocrine or retrograde regulation of neurons.
FSH (zeige BRD2 Proteine) and cAMP stimulate AMH transcription by granulosa cells. FSH (zeige BRD2 Proteine) and LH have an additive effect, which may be important in polycystic ovary syndrome.
This suggests that MIS is one of the determinants of "boy"-specific behavior.
Role of anti-Mullerian hormone (AMH) as a regulator and marker of ovarian function.
Administration of MIS to male mice induced IEX-1S mRNA in the prostate in vivo, suggesting that MIS may function as an endogenous hormonal regulator of NF-kappaB (zeige NFKB1 Proteine) signaling and growth in the prostate gland.
The effects of castration and other surgical intervention on the blood levels of anti-Muellerian hormone, inhibin A (zeige INHA Proteine), gonadotropins, and gonadotropin receptors in bull calves are reported.
Results from these studies indicate that AMH signaling plays a role in both regulating granulosa cell proliferation and preventing granulosa cells from 5- to 8-mm follicles from undergoing premature differentiation before follicle selection.
These findings indicate the followings: AMH mRNA levels decrease in both dominant and secondary follicles during follicular deviation; granulosa cells from heathy follicles express more AMH mRNA compared to subordinate follicles undergoing atresia and FSH (zeige BRD2 Proteine) stimulates AMH and AMHR2 (zeige AMHR2 Proteine) mRNA expression in granulosa cells of co-dominant follicles.
Measurement of AMH concentration in the plasma of cows can help to predict their capacity for embryo production in response to gonadotrophin treatment.
In first study to investigate the blood profile and immunohuistochemistry of anti-Mullerian hormone in bovine granulosa-theca cell tumors, the findings indicated that anti-Mullerian hormone is a novel biomarker for granulosa-theca cell tumors in cattle.
Regulation of anti-Mullerian hormone production in the cow.
Intrafollicular AMH was not a marker of cystic development in the cow, but low AMH concentrations in cysts were associated with luteinization.
AMH expression is modulated by androgens in bovine granulosa cells from small follicles.
Anti-Mullerian hormone is a member of the transforming growth factor-beta gene family which mediates male sexual differentiation. Anti-Mullerian hormone causes the regression of Mullerian ducts which would otherwise differentiate into the uterus and fallopian tubes. Some mutations in the anti-Mullerian hormone result in persistent Mullerian duct syndrome.
, anti-Mullerian hormone
, anti-mullerian hormone
, Mullerian inhibiting factor
, Mullerian inhibiting substance
, anti-Muellerian hormone
, muellerian-inhibiting substance
, Mullerian inhibitory substance
, Anti - Mullerian hormone (Mulerian inhibiting substance)