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Rat (Rattus) Angiotensin II ELISA Kit für Competition ELISA - ABIN416068
Liu, Hu, Wang, Zhang, Ma, Feng, Wang, Wang, Dong, Gao, Zhang, Zhang: Angiotensin-converting enzyme (ACE) 2 overexpression ameliorates glomerular injury in a rat model of diabetic nephropathy: a comparison with ACE inhibition. in Molecular medicine (Cambridge, Mass.) 2011
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Human Angiotensin II ELISA Kit für Competition ELISA - ABIN414350
Zhang, Long, Sun, Wang, Li, Wu, Guo, Li, Niu, Li, Liu, Mei et al.: Evidence for the complementary and synergistic effects of the three-alkaloid combination regimen containing berberine, hypaconitine and skimmianine on the ulcerative colitis rats induced by ... in European journal of pharmacology 2010
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Mouse (Murine) Angiotensin II ELISA Kit für Competition ELISA - ABIN415355
Wang, Liu, Ye, Zhang, Tang, Chen, Zhang, Lou: Mesangial medium with IgA1 from IgA nephropathy inhibits nephrin expression in mouse podocytes. in European journal of clinical investigation 2009
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Rat (Rattus) Angiotensin II ELISA Kit für Sandwich ELISA - ABIN368044
Simic, Hermann, Shaw, Bigler, Stalder, Dörries, Besler, Lüscher, Ruschitzka: Torcetrapib impairs endothelial function in hypertension. in European heart journal 2011
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Mouse (Murine) Angiotensin II ELISA Kit für Sandwich ELISA - ABIN366511
Than, Leow, Chen: Control of adipogenesis by the autocrine interplays between angiotensin 1-7/Mas receptor and angiotensin II/AT1 receptor signaling pathways. in The Journal of biological chemistry 2013
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Human Angiotensin II ELISA Kit für Sandwich ELISA - ABIN366510
Mao, Cao, Lan, He, Chen, Wang, Hu, Lv, Wang, Yan: Therapeutic effect of forest bathing on human hypertension in the elderly. in Journal of cardiology 2012
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Autosomal dominant polycystic kidney disease (ADPKD), uniquely increases urinary angiotensinogen (zeige AGT ELISA Kits) and renin (zeige REN ELISA Kits) excretion despite their circulating levels being comparable with those in non-ADPKD chronic kidney disease.
Quaternary interactions and supercoiling modulate the cooperative DNA binding of AGT (zeige AGXT ELISA Kits).
results show that SNPs in the Hap (zeige SAFB ELISA Kits)-I of the hAGT gene promote high-fat diet-induced binding of transcription factors GR, CEBP-beta (zeige CEBPB ELISA Kits) and STAT3 (zeige STAT3 ELISA Kits), which lead to elevated expression of the hAGT gene in hepatic and adipose tissues
Angiotensinogen (zeige AGT ELISA Kits) import and subsequent trafficking to the mitochondria occurs in proximal kidney tubules.
Transgenic mice expressing human AGT (zeige AGXT ELISA Kits) in the subfornical organ AGT (zeige AGXT ELISA Kits) and possibly ANG I/ANG II (zeige AGT ELISA Kits) into the cerebral ventricles.
AngII could induce pulmonary injury by triggering endothelial barrier injury, and such process may be related to the dephosphorylation of Y685-VE-cadherin (zeige CDH5 ELISA Kits) and the endothelial skeletal rearrangement
Renin-angiotensin system transgenic mouse model suggests that renal injury in preeclampsia may be mediated through local VEGF.
endoplasmic reticulum stress induces apoptosis in human alveolar epithelial cells through mediation of unfolded protein response pathways, which in turn regulate the autocrine ANGII/ANG1 (zeige ANGPT1 ELISA Kits)-7 system.
Angiotensin II stimulates PYY secretion, in turn inhibiting epithelial anion fluxes, thereby reducing net fluid secretion into the colonic lumen.
NOXs had two time-dependent reactions in response to Ang II (zeige AGT ELISA Kits) stimulation via MAPK (zeige MAPK1 ELISA Kits) pathwa
this study demonstrated that Ang II (zeige AGT ELISA Kits) could increase TRPC6 (zeige TRPC6 ELISA Kits) induced Ca(2 (zeige CA2 ELISA Kits)+) influx and enhance autophagy through increasing reactive oxygen species levels in podocytes, and autophagy could protect Ang II (zeige AGT ELISA Kits)-treated podocytes.
These results implied that AngII could effectively induce EpiCs to differentiate into vascular smooth muscle-like cells through the AT1 receptor (zeige AGTRAP ELISA Kits).
Results suggest the involvement of angiotensin II (Ang II), through its angiotensin type-1 receptor (AT1R (zeige AGTRAP ELISA Kits)) in the inflammation induced by Aah (zeige ASPH ELISA Kits) venom, in the heart and the aorta.
expression of spinal ACE (zeige ACE ELISA Kits) increased in streptozotocin-induced diabetic mice, which in turn led to an increase in Ang II (zeige AGT ELISA Kits) levels and tactile allodynia.
the beneficial actions of insulin (zeige INS ELISA Kits) in diabetic nephropathy appear to be mediated, in part, by suppressing renal Nrf2 (zeige NFE2L2 ELISA Kits) and Agt (zeige AGXT ELISA Kits) gene transcription and preventing Nrf2 (zeige NFE2L2 ELISA Kits) stimulation of Agt (zeige AGXT ELISA Kits) expression via hnRNP F (zeige HNRNPF ELISA Kits)/K.
Angiotensinogen (zeige AGT ELISA Kits)-mediated downregulation of aquaporin 1 (zeige AQP1 ELISA Kits) and Nrf2 (zeige NFE2L2 ELISA Kits) signalling may play an important role in intrarenal renin (zeige REN ELISA Kits)-angiotensin system-induced hypertension and kidney injury.
This study suggests that deletion of AT2R decreases the expression of the beneficial ACE2/Ang-(1-7)/MasR.
an inverse correlation was found between Ang-(1 (zeige ANGPT1 ELISA Kits)-7) level and tau hyperphosphorylation, a pathological hallmark of Alzheimer's disease, in cerebral cortex and hippocampus of SAMP8 mice.
The inhibition of pathological autophagy in the heart in response to chronic Ang II (zeige AGT ELISA Kits) by Interleukin-10 (zeige IL10 ELISA Kits), and its implications, has been described.
The protein encoded by this gene, pre-angiotensinogen or angiotensinogen precursor, is expressed in the liver and is cleaved by the enzyme renin in response to lowered blood pressure. The resulting product, angiotensin I, is then cleaved by angiotensin converting enzyme (ACE) to generate the physiologically active enzyme angiotensin II. The protein is involved in maintaining blood pressure and in the pathogenesis of essential hypertension and preeclampsia. Mutations in this gene are associated with susceptibility to essential hypertension, and can cause renal tubular dysgenesis, a severe disorder of renal tubular development. Defects in this gene have also been associated with non-familial structural atrial fibrillation, and inflammatory bowel disease.
alpha-1 antiproteinase, antitrypsin
, angiotensin I
, angiotensin II
, serine (or cysteine) proteinase inhibitor
, serpin A8
, angiotensinogen (PAT)
, angiotensin ll