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anti-Mouse (Murine) Antikörper:
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Human Monoclonal LCN2 Primary Antibody für ELISA, WB - ABIN1045904
Devarajan: Neutrophil gelatinase-associated lipocalin (NGAL): a new marker of kidney disease. in Scandinavian journal of clinical and laboratory investigation. Supplementum 2008
Show all 18 Pubmed References
Human Monoclonal LCN2 Primary Antibody für ELISA, WB - ABIN1045905
Makris, Markou, Evodia, Dimopoulou, Drakopoulos, Ntetsika, Rizos, Baltopoulos, Haliassos: Urinary neutrophil gelatinase-associated lipocalin (NGAL) as an early marker of acute kidney injury in critically ill multiple trauma patients. in Clinical chemistry and laboratory medicine : CCLM / FESCC 2009
Show all 18 Pubmed References
Rat (Rattus) Polyclonal LCN2 Primary Antibody für IHC (p), WB - ABIN966671
Begley, Robb, Rockman, Visvader, Bockamp, Chan, Green: Structure of the gene encoding the murine SCL protein. in Gene 1994
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Human Polyclonal LCN2 Primary Antibody für IHC, IHC (p) - ABIN4331126
Rathore, Berard, Redensek, Chierzi, Lopez-Vales, Santos, Akira, David: Lipocalin 2 plays an immunomodulatory role and has detrimental effects after spinal cord injury. in The Journal of neuroscience : the official journal of the Society for Neuroscience 2011
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Human Polyclonal LCN2 Primary Antibody für IF (p), IHC (p) - ABIN685627
Xing, Wang, Cheng, Montaner, Mandeville, Leung, van Leyen, Lok, Wang, Lo: Neuronal production of lipocalin-2 as a help-me signal for glial activation. in Stroke; a journal of cerebral circulation 2014
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Human Polyclonal LCN2 Primary Antibody für IHC (p), IHC - ABIN250360
Yu, Wu, Ping, Huang, Shen: NGAL can alternately mediate sunitinib resistance in renal cell carcinoma. in The Journal of urology 2014
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Mouse (Murine) Polyclonal LCN2 Primary Antibody für ELISA, WB - ABIN408671
Venkatesha, Hanai, Seth, Karumanchi, Sukhatme: Lipocalin 2 antagonizes the proangiogenic action of ras in transformed cells. in Molecular cancer research : MCR 2006
Rat (Rattus) Monoclonal LCN2 Primary Antibody für ELISA, ICC - ABIN451573
Sultan, Ahmad, Rave-Fränk, Malik, Hess, Christiansen, Cameron: Induction of Lipocalin2 in a Rat Model of Lung Irradiation. in International journal of molecular sciences 2016
Our findings collectively support a potential role of LCN2 in cancer cell invasion through the epithelial-to- mesenchymal transition pathway and suggest that LCN2 could be effectively utilized as a lymph node metastasis marker in cervical cancer.
We conclude that low BDNF (zeige BDNF Antikörper) and high LCN2 and NF-L (zeige NEFL Antikörper) levels are associated with Multiple Sclerosis (MS) pathogenesis, and high IGFBP1level is a biomarker for female MS only, suggesting different MS progression pathways between the sexes. LCN2 is a candidate predictor of response to natalizumab treatment, and NF-L (zeige NEFL Antikörper) is a candidate predictor of clinically isolated syndrome's (CIS (zeige CISH Antikörper)) conversion into MS.
Ascites NGAL level may be a biomarker of peritonitis in hospitalized patients with cirrhosis and an independent predictor of short-term in-hospital mortality.
Circulating NGAL and MMP-9 (zeige MMP9 Antikörper)/NGAL are associated with plaque vulnerability in patients with carotid artery stenosis. Statin treatment could contribute to plaque stabilization by reducing circulating NGAL and MMP-9 (zeige MMP9 Antikörper)/NGAL levels.
our findings indicated that LCN2 negatively modulated proliferation, EMT (zeige ITK Antikörper) and energy metabolism in colorectal cancer (CRC (zeige CALR Antikörper)) cells. Accordingly, LCN2 may be a candidate metastasis suppressor and potential therapeutic target in CRC (zeige CALR Antikörper)
High plasma NGAL correlates with high mortality and myocardial dysfunction in severe sepsis and septic shock.
The analysis showed higher urinary NGAL and urinary ATF3 (zeige ATF3 Antikörper) in patients with sepsis-AKI in comparison with patients with sepsis-non-AKI and healthy volunteers.
Knockout of Lcn2 attenuated tumor-associated lymphangiogenesis and breast tumor metastasis
Lipocalin-2 Promotes Endoplasmic Reticulum Stress and Proliferation by Augmenting Intracellular Iron in Human Pulmonary Arterial Smooth Muscle Cells
Acute kidney injury is more common with sepsis than non-infectious systemic inflammatory response syndrome. Plasma NGAL was diagnostic of AKI in both subgroups.
Overexpression of exogenous kidney-specific Ngal reduced cystic progression and prolonged the lifespan in polycystic kidney disease mice, and was associated with reductions in interstitial fibrosis and proliferation, and augmented apoptosis.
Either Lcn2 deficiency or anti-Lcn2 antibody blockade limits abdominal aortic aneurysm expansion in mice by decreasing neutrophil infiltration in the aorta.
Results suggest that blood brain barrier leakage occurs in white matter after subarachnoid hemorrhage (SAH (zeige ACSM3 Antikörper)) and that LCN2 contributes to SAH (zeige ACSM3 Antikörper)-induced blood brain barrier disruption.
LCN2 overexpression in bones modifies the bone marrow microenvironment via modulation of the expression of key secreted factors and cytokines, which in turn regulate the hematopoietic stem cell niche behavior enhancing both HSC (zeige FUT1 Antikörper) homing in young mice and erythrocytes production in older mice.
LCN2 modulated the secretion of proinflammatory cytokines in PSC of the PDAC tumor microenvironment, whereas downregulation of LCN2-specific receptor SLC22A17 (zeige SLC22A17 Antikörper) blocked these effects. Our results reveal how LCN2 acts in the tumor microenvironment links obesity, inflammation, and PDAC development.
results identify lipocalin 2 as a bone-derived hormone with metabolic regulatory effects, which suppresses appetite in a MC4R (zeige MC4R Antikörper)-dependent manner, and show that the control of appetite is an endocrine function of bone
Lcn2 can suppress the beneficial cardiac autophagic response to ischemia and that this contributes to enhanced ischemia-induced cell death and cardiac dysfunction.
Plasma levels of LCN2 measured in patients one week after ischemic stroke contribute to the prediction of clinical outcome at 90 days and reflect the systemic response to post-stroke infections
may potentiate the development of psoriasis via the enhancement of Th17- and antimicrobial peptide (zeige cAMP Antikörper)-mediated inflammation
we demonstrated the presence of high molecular weight (HMW) complexes (130, 170, and 220 kDa) containing MMP9 (zeige MMP9 Antikörper), TIMP1 (zeige TIMP1 Antikörper), and NGAL (also MMP2 (zeige MMP2 Antikörper) in 220 kDa complex) without proteolytic activity.
porcine p25alpha (zeige Tppp Antikörper) is heavily modified with a variety of modifications: phosphorylation, di- and trimethylation, citrullination and a HexNAc group.
mouse homolog plays a role in IL3 withdrawal-induced apoptosis
, lipocalin 2 (oncogene 24p3)
, 25 kDa alpha-2-microglobulin-related subunit of MMP-9
, migration-stimulating factor inhibitor
, neutrophil gelatinase-associated lipocalin
, oncogene 24p3
, siderocalin LCN2
, SV-40-induced 24P3 protein
, secreted inducible protein 24
, alpha-2-microglobulin-related protein
, alpha-2U globulin-related protein