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The Arabidopsis Malectin (zeige MLEC ELISA Kits)-Like/LRR-RLK (zeige TXK ELISA Kits) IOS1 Is Critical for BAK1-Dependent and BAK1-Independent Pattern-Triggered Immunity
These findings show that BAK1 contains an additional AtRALF1 binding site, indicating that this protein may be part of a AtRALF1-containing complex as a co-receptor, and it is required for the negative regulation of cell expansion
the existence of partial redundancy between SERK3 and SERK1 as well as the promoting or repressive activity of a single coreceptor in multiple simultaneously active pathways, is reported.
The found that in an otherwise "active" BAK1 the alphaC helix is highly disordered, a hallmark of deactivation, whereas the BRI1 alphaC helix is moderately disordered and displays swinging behavior similar to numerous animal kinases.
These data suggest that BIK1 cooperates with BAK1 to regulate constitutive immunity and cell death.
Oligogalacturonides are sensed through multiple and partially redundant perception/transduction complexes, some targeted by AvrPto, but not necessarily comprising BAK1 and BKK1.
These results suggest that pleiotropic phenotypic alterations shown in the BAK1- overexpressing transgenic plants result from the constitutive activation of SA-mediated defense responses.
The study reveals the signaling activation mechanism in floral organ abscission by IDA-induced HAE/HSL2-SERK complex formation accompanied by transphosphorylation.
Receptor like kinases (RLP)-mediated resistance and endocytosis require ligand-induced recruitment of BAK1/SERK3, reminiscent of BAK1/SERK3 interaction and subcellular fate of the FLAGELLIN (zeige FliC ELISA Kits) SENSING 2 (FLS2) RLK (zeige TXK ELISA Kits).
G protein signaling is directly activated by the pathogen-associated molecular pattern flagellin (zeige FliC ELISA Kits) peptide 22 through its LRR RLK (zeige TXK ELISA Kits), FLS2, and co-receptor BAK1.
interleukin-6 (zeige IL6 ELISA Kits), endothelin ET-1 (zeige EDN1 ELISA Kits), and apoptotic Bak and Bcl-XL (zeige BCL2L1 ELISA Kits) genes have roles in small bowel transplantation, in a swine model of ischemia and reperfusion injury
Bak mRNA was significantly increased in haploid parthenotes compared with the diploid parthenotes. These results suggest that the haploid state affects apoptosis-related gene expression which results in increased apoptosis
UMI-77 enhances TRAIL-induced apoptosis by unsequestering Bim (zeige BCL2L11 ELISA Kits) and Bak, which provides a novel therapeutic strategy for the treatment of gliomas.
after the induction of apoptosis, Bak switches from its association with Mtx2 (zeige MTX2 ELISA Kits) and VDAC2 (zeige VDAC2 ELISA Kits) to a closer association with Mtx1 (zeige MTX1 ELISA Kits).
Conversion of Bim (zeige BCL2L11 ELISA Kits)-BH3 from Activator to Inhibitor of Bak through Structure-Based Design.
The results strongly suggest that the direct apoptosis-activation activities of Bid (zeige BID ELISA Kits), Bim (zeige BCL2L11 ELISA Kits), Puma (zeige BBC3 ELISA Kits), and p53 (zeige TP53 ELISA Kits) are not essential for activating Bax (zeige BAX ELISA Kits)/Bak once the anti-apoptotic Bcl-2 (zeige BCL2 ELISA Kits) proteins are neutralized.
Studied BAK1, SPRY4 (zeige SPRY4 ELISA Kits) and GAB2 (zeige GAB2 ELISA Kits) SNPs in pediatric germ cell tumors(GCT (zeige QPCT ELISA Kits)); found a variant in SPRY4 (zeige SPRY4 ELISA Kits) was associated with reduced risk of GCT (zeige QPCT ELISA Kits); a variant in BAK1 was positively associated with GCT (zeige QPCT ELISA Kits) with a strong estimated effect for testis tumors; and a SNP in GAB2 (zeige GAB2 ELISA Kits) was associated with increased risk for GCT (zeige QPCT ELISA Kits).
These results not only establish Bok (zeige BOK ELISA Kits) as a Bak- and Bax (zeige BAX ELISA Kits)-independent apoptosis inducer, but also suggest a potential impact of Bok (zeige BOK ELISA Kits) expression in ovarian cancer therapy.
The low-level constitutive production of platelets caused by the G allele of rs5745568 in BAK1 protein seems to increase the risk of bleeding in dengue infection
the expression of Bak1 was downregulated in CRC (zeige CALR ELISA Kits) tumor tissues and was reversely correlated with the expression of miR410, which provided further support that Bak1 was regulated by miR410.
Low expression of BAX (zeige BAX ELISA Kits) is associated with docetaxel resistance in gastric cancer.
Polymorphisms of KITLG (zeige KITLG ELISA Kits), SPRY4 (zeige SPRY4 ELISA Kits), and BAK1 genes in patients with testicular germ cell tumors and individuals with infertility associated with AZFc deletion of the Y chromosome
Postnatal synaptic rearrangement needed for acquisition of skilled behaviors requires the activity-dependent, non-apoptotic Bax (zeige BAX ELISA Kits)/Bak-caspase (zeige CASP3 ELISA Kits) signaling cascade. Adult Bax (zeige BAX ELISA Kits)/Bak mutant mice exhibit aberrant co-activation of antagonistic muscle pairs and skilled grasping deficits but normal reaching and retrieval behaviors.
The authors show that Bak is activated and that activated Bak is bound to p53 (zeige TP53 ELISA Kits) during reovirus encephalitis.
Pancreatic beta-Cell Death due to Pdx-1 (zeige PDX1 ELISA Kits) Deficiency Requires Multi-BH Domain Protein Bax (zeige BAX ELISA Kits) but Not Bak.
lipid profile in the absence of the proapoptotic proteins BAX (zeige BAX ELISA Kits) and BAK in mouse embryonic fibroblasts
alpha1 dissociation is a key step in unfolding Bak into three major components, the N terminus, the core (alpha2-alpha5) and the latch (alpha6-alpha8) that is required for apoptosis.
BAX (zeige BAX ELISA Kits) and BAK have a nonapoptotic role in eicosanoid metabolism in inflammation.
Puma (zeige BBC3 ELISA Kits) is the major mediator of virus-induced Bax (zeige BAX ELISA Kits)/Bak activation and mitochondrial membrane permeabilization induction.
Motifs of VDAC2 (zeige VDAC2 ELISA Kits) required for mitochondrial Bak import and tBid-induced apoptosis.
dual ablation of Bax (zeige BAX ELISA Kits) and Bak suppressed ureteral obstruction induced inflammation and kidney fibrosis with decreased tubular cell cycle arrest
regulation of A1 and Bak contribute to mitochondrial membrane stability, reduced caspase-9 (zeige CASP9 ELISA Kits) activity, and delayed apoptosis in bovine neutrophils exposed to glucocorticoids
The protein encoded by this gene belongs to the BCL2 protein family. BCL2 family members form oligomers or heterodimers and act as anti- or pro-apoptotic regulators that are involved in a wide variety of cellular activities. This protein localizes to mitochondria, and functions to induce apoptosis. It interacts with and accelerates the opening of the mitochondrial voltage-dependent anion channel, which leads to a loss in membrane potential and the release of cytochrome c. This protein also interacts with the tumor suppressor P53 after exposure to cell stress.
BCL2-like 7 protein
, apoptosis regulator BAK
, bcl-2 homologous antagonist/killer
, bcl-2-like protein 7
, pro-apoptotic protein BAK
, Bcl2 homologous antagonist/killer
, Bcl-2-like 7