ENOS Proteine (NOS3)

Bezeichnung:
Nitric Oxide Synthase 3 (Endothelial Cell) Proteine (NOS3)
Auf www.antikoerper-online.de finden Sie aktuell 23 Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) Proteine von 7 unterschiedlichen Herstellern. Zusätzlich bieten wir Ihnen ENOS Antikörper (478) und ENOS Kits (97) und viele weitere Produktgruppen zu diesem Protein an. Insgesamt sind aktuell 612 ENOS Produkte verfügbar.
Synonyme:
2310065A03Rik, cNOS, EC-NOS, ecNOS, ENOS, NOS, Nos-3, NOS3, NOSIII
alle Proteine anzeigen Gen GeneID UniProt
NOS3 4846 P29474
NOS3 18127 P70313
NOS3 24600 Q62600

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Weitere Proteine zu ENOS Interaktionspartnern

Human Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) Interaktionspartner

  1. NOS3 (zeige NANOS3 Proteine) -786 C/T rs2070744 polymorphism in Dilated Cardiomyopathy may serve as a marker for more rapid progression of Heart Failure.

  2. This study shows that expression of SDF-1alpha, eNOS and VEGF (zeige VEGFA Proteine) were significantly higher in uterine leiomyoma than myometrium with a different expression pattern according to the size of uterine leiomyoma. However, expressions of visfatin (zeige NAMPT Proteine) and leptin (zeige LEP Proteine) had no significant differences between the two groups.

  3. The C allele of the eNOS single nucleotide polymorphism, is independently associated with increased risk for delayed cerebral ischemia following subarachnoid hemorrhage.

  4. In the subgroup analysis based on ethnicity, both T786C and 4a4b eNOS polymorphisms could influence the risk of male infertility in Asian and Caucasian.[meta-analysis].

  5. Phosphocreatine exerts an antiapoptotic effect in HUVECs exposed to oxidative stress by methylglyoxal through the mitochondrial pathway and the modulation of PI3K (zeige PIK3CA Proteine)/Akt (zeige AKT1 Proteine)/eNOS and NF-kappaB (zeige NFKB1 Proteine) signaling.

  6. NOS2 (zeige NANOS2 Proteine) rs2779248, NOS2 (zeige NANOS2 Proteine) rs1137933, and NOS3 (zeige NANOS3 Proteine) rs3918188 genetic polymorphisms are potentially related to the susceptibility to type 2 diabetes mellitus (T2DM), and the rs1800783 polymorphism might be considered as genetic risk factors for diabetic nephropathy.

  7. This study determined the genetic polymorphisms of the eNOS (Glu298Asp) and Cav-1 (zeige CAV1 Proteine) (G14713A and T29107A) genes in association with susceptibility risk in patients who had suffered from a large artery atherosclerotic stroke. It demonstrated that the eNOS Glu298Asp in combination with 14713A/29107A at the CAV1 (zeige CAV1 Proteine) locus was associated with a significant risk of an large artery atherosclerotic stroke.

  8. The eNOS T786C polymorphism was associated with ISR in Chinese Han patients treated with DES (zeige DES Proteine). Genotyping may be helpful to identify patients with higher risks of ISR after drug-eluting stent implantation.

  9. Meta-analysis. eNOS gene G894T polymorphism was related to pregnancy-induced hypertension risk, especially for Asians.

  10. Data suggest that, in T-lymphocytes, nitric oxide generated by eNOS S-nitrosylates Cys374 on ACTB (zeige ACTB Proteine) and thus regulates activation/recruitment of PRKCQ (zeige PRKCQ Proteine) at immune synapse; S-nitrosylation of beta-actin (zeige ACTB Proteine) impairs actin binding to PFN1 (zeige PFN1 Proteine) and regulates protein transport in lamellipodia. (eNOS = nitric oxide synthase 3; ACTB (zeige ACTB Proteine) = beta-actin (zeige ACTB Proteine); PRKCQ (zeige PRKCQ Proteine) = protein kinase C-theta (zeige PRKCQ Proteine); PFN1 (zeige PFN1 Proteine) = profilin-1 (zeige PFN1 Proteine))

Pig (Porcine) Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) Interaktionspartner

  1. NOS3 was lowest in kidneys removed from live pigs, greater in kidneys from pigs with brain death, and greatest in kidneys from pigs with cardiac arrest.

  2. Rapid atrial pacing increases ADMA and down-regulates eNOS expression in an ADMA-independent manner.

  3. Icariin and icariside II may increase the eNOS expression through activating EGF-EGFR (zeige EGFR Proteine) pathway in porcine aortic endothelial cells.

  4. Data suggest that pig sperm contain bNOS (zeige NOS1 Proteine), iNOS (zeige NOS2 Proteine), and eNOS; up-regulation of NOS by leptin (zeige LEP Proteine) during acrosome reaction and inhibition of acrosome reaction by inhibitors of nitric oxide synthases suggests these enzymes are involved in acrosome reaction.

  5. Periodic acceleration (pGz) acutely increases endothelial and neuronal nitric oxide synthase (zeige NOS1 Proteine) expression in endomyocardium of normal swine.

  6. Data suggest that angiotensin II regulates nNOS and eNOS expression and NOS activity in afferent arterioles of the developing kidney via angiotensin 1 and 2 receptors.

  7. Endothelial nitric oxide synthase mRNA expression was elevated in gestational day 50 intrauterine growth retardation placenta and areola compared to gd50 control.

  8. Oligonol prevented the impairment of eNOS activity induced by high glucose through reversing altered eNOS phosphorylation status.

  9. Exercise training significantly increased total eNOS and phosphorylated levels of eNOS (pSer(1179)) in collateral-dependent arteries of experimental minipigs.

  10. Data show that wall shear stress increases with a decrease in artery diameter; eNOS protein contents decrease with an increase in diameter.

Cow (Bovine) Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) Interaktionspartner

  1. S1179D substitution in eNOS increased the rate of flavin reduction, altered the conformational equilibrium of the reductase domain, and increased the rate of its conformational transitions.

  2. These data define the mechanism responsible for the repressive effects of nitric oxide (NO) on the transcriptional activity of beta-catenin (zeige CTNNB1 Proteine) and link eNOS-derived NO to the modulation by VEGF (zeige VEGFA Proteine) of Wnt (zeige WNT2 Proteine)/beta-catenin (zeige CTNNB1 Proteine)-induced endothelial cell proliferation.

  3. Endothelial cell autophagy maintains shear stress-induced nitric oxide generation via glycolysis-dependent ATP/P2Y1 receptor (zeige P2RY1 Proteine) signaling to endothelial nitric oxide synthase.

  4. TNFalpha (zeige TNF Proteine)reduces eNOS activity in bovine endothelial cells through serine 116 phosphorylation and Pin1 binding.

  5. Pomanox supplementation hinders hyperlipemia-induced coronary endothelial dysfunction by activating the Akt (zeige AKT1 Proteine)/endothelial nitric oxide-synthase pathway and favorably counteracting vascular inflammation and oxidative damage.

  6. Signals that activate and phosphorylate eNOS are transmitted through distinct membrane domains in endothelial cells. Cholesterol domains, but not individual caveolae, mediate HDL stimulation of eNOS. VEGF and shear stress may act through caveolae.

  7. eNOS serine 1179 phosphorylation, in addition to enhancing NO production, also profoundly affects superoxide generation

  8. In addition to the heme center of eNOS oxygenase domain, we confirmed another O2.- generation site in the eNOS reductase domain and characterized its regulatory properties.

  9. A dimer-destabilized mutant of bovine eNOS where cysteine 101 was replaced by alanine, cloned and introduced into an eNOS-deficient mouse strain and that results provide the first in vivo evidence that eNOS-dependent oxidative stress is unlikely to be an initial cause of impaired endothelium-dependent vasodilation and/or a pathologic factor promoting intimal hyperplasia.

  10. Data show that resveratrol (Res) reversed caveolin-1 (Cav-1 (zeige CAV1 Proteine))/endothelial nitric oxide synthase (eNOS) expressions in high glucose cultured bovine aortic endothelial cells (BAECs).

Rabbit Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) Interaktionspartner

  1. Kidneys from circulation-restricted fetuses showed endothelial nitric oxide synthase phosphorylation inhibition.

  2. Antidiabetic drug pioglitazone protects the heart via activation of endothelial nitric oxide synthase (eNOS/Nos3) pathway in a rabbit model of myocardial infarction.

  3. VEGFR2 (zeige KDR Proteine) activation was not affected by Slit2 (zeige SLIT2 Proteine), but eNOS phosphorylation was diminished

  4. Data suggest distinct localizations of eNOS at the spiral arteries/arterial sinuses and iNOS (zeige NOS2 Proteine) along the radial arteries in the developing placenta.

  5. Pulmonary ischaemia-reperfusion up-regulates inducible nitric oxide synthesis and/activity, which coincides with reduced endothelial nitric oxide synthase activity.

  6. eNOS dysregulation may be a central mechanism of impaired cardioprotection during hyperglycemia.

  7. Quercetin inhibited myocardial ischemia-reperfusion-induced NOS3 mRNA and protein expression.

Mouse (Murine) Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) Interaktionspartner

  1. these data suggest that CD NOS3 may be involved in the diuretic response to a water load in a sex-specific manner; the mechanism of this effect remains to be determined.

  2. CAV1 (zeige CAV1 Proteine) KO mice have elevated IOP and reduced conventional outflow facility when compared with WT mice. CAV2 (zeige CAV2 Proteine) expression was absent in CAV1 (zeige CAV1 Proteine) KO mice, but we observed increased expressions of eNOS

  3. TNF-alpha (zeige TNF Proteine) does not regulate eNOS activity in murine endothelial cells through serine 116 phosphorylation and Pin1 (zeige PIN1 Proteine) binding.

  4. The eNOS expressed in smooth muscle cells in FAs (zeige FAS Proteine) attenuates alpha-adrenergic vasoconstriction.

  5. Thioredoxin (zeige TXN Proteine)-mediated deglutathionylation of eNOS in the coronary artery in vivo protected against reperfusion injury, even in the presence of normal levels of glutathione.

  6. Eph (zeige EPHA1 Proteine)-B4 stimulates eNOS phosphorylation in vitro and may mediate vein graft adaptation by regulation of eNOS activity in vivo.

  7. Loss of endothelial NO plays an important role in the generation of p25 (zeige CDK5R1 Proteine) and resulting tau phosphorylation in neuronal tissue. Endothelial nitric oxide synthase (eNOS)-deficient (eNOS-/-) mice display increased levels of p25 (zeige CDK5R1 Proteine), an aberrant activator of cyclin-dependent kinase 5 (zeige CDK5 Proteine), which is one of the primary kinases responsible for tau hyperphosphorylation, and a statistically higher p25/p35 (zeige CDK5R1 Proteine) ratio.

  8. Pulse pressure finely regulates eNOS, controlling cerebral artery reactivity.

  9. TNF-alpha (zeige TNF Proteine) induces vascular insulin (zeige INS Proteine) resistance by mechanisms that involve positive modulation of PTEN and inhibition of Akt (zeige AKT1 Proteine)/eNOS/NO signaling.

  10. Beta2AR (zeige ADRB2 Proteine) overexpression enhances endothelial progenitor cells (EPC (zeige TCF21 Proteine)) functions in vitro and enhances the vascular repair abilities of EPCs in vivo via the beta2AR (zeige ADRB2 Proteine)/Akt (zeige AKT1 Proteine)/eNOS pathway.

Guinea Pig Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) Interaktionspartner

  1. The present evidence indicated that the customary HBOT protocol may increase constitutive NOS expression.

ENOS (NOS3) Protein Überblick

Protein Überblick

Nitric oxide is a reactive free radical which acts as a biologic mediator in several processes, including neurotransmission and antimicrobial and antitumoral activities. Nitric oxide is synthesized from L-arginine by nitric oxide synthases. Variations in this gene are associated with susceptibility to coronary spasm. Multiple transcript variants encoding different isoforms have been found for this gene.

Alternative names and synonyms associated with ENOS (NOS3)

  • nitric oxide synthase 3 (endothelial cell) (NOS3)
  • nitric oxide synthase 3 (endothelial cell) (nos3)
  • endothelial nitric oxide synthase (23B)
  • nitric oxide synthase 3, endothelial cell (Nos3)
  • 2310065A03Rik Protein
  • cNOS Protein
  • EC-NOS Protein
  • ecNOS Protein
  • ENOS Protein
  • NOS Protein
  • Nos-3 Protein
  • NOS3 Protein
  • NOSIII Protein

Bezeichner auf Proteinebene für NOS3

nitric oxide synthase 3 (endothelial cell) , nitric oxide synthase, endothelial-like , EC-NOS , NOS type III , NOSIII , cNOS , constitutive NOS , endothelial NOS , nitric oxide synthase, endothelial , eNOS , endothelial nitric oxide synthase , endothelial nitric oxide synthase NOS3 , endothelial nitric oxide synthase 3

GENE ID SPEZIES
714231 Macaca mulatta
100018035 Monodelphis domestica
100486016 Xenopus (Silurana) tropicalis
443077 Ovis aries
4846 Homo sapiens
403784 Canis lupus familiaris
397557 Sus scrofa
287024 Bos taurus
443073 Ovis aries
100063339 Equus caballus
100009498 Oryctolagus cuniculus
18127 Mus musculus
24600 Rattus norvegicus
100135577 Cavia porcellus
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