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anti-Mouse (Murine) WNT10A Antikörper:
anti-Human WNT10A Antikörper:
anti-Rat (Rattus) WNT10A Antikörper:
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Human Polyclonal WNT10A Primary Antibody für ELISA, WB - ABIN1003487
Kelly, Lai, Moon: Expression of wnt10a in the central nervous system of developing zebrafish. in Developmental biology 1993
Show all 4 Pubmed References
Bat Polyclonal WNT10A Primary Antibody für IHC (p) - ABIN188908
Hakim, Kosmehl, Sieg, Trenkle, Jacobsen, Attila Benedek, Ribbat, Driemel: Altered expression of cell-cell adhesion molecules β-catenin/E-cadherin and related Wnt-signaling pathway in sporadic and syndromal keratocystic odontogenic tumors. in Clinical oral investigations 2011
Human Polyclonal WNT10A Primary Antibody für IHC, IHC (p) - ABIN4892990
Lee, Zheng, Kang, Kim, Oh, Cho: Therapeutic efficacy of autologous platelet-rich plasma and polydeoxyribonucleotide on female pattern hair loss. in Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society 2015
Human and mouse WNT10A mutant palmoplantar and tongue epithelia also display specific differentiation defects that are mimicked by loss of the transcription factor KLF4 (zeige KLF4 Antikörper).
Deletion of WNT10A resulted in the delayed wound healing associated with suppressed stromagenesis, especially in fibroblasts/myofibroblasts.
The authors conclude that inhibiting DNA methylation (zeige HELLS Antikörper) by 5-Aza-dC mutual-exclusively regulates the lineage determination of adipogenesis and osteoblastogenesis by demethylating Wnt10a gene and upregulating its expression.
Wnt10a/beta-catenin (zeige CTNNB1 Antikörper) signaling pathway is able to exacerbate keloid cell proliferation and inhibit the apoptosis of keloid cells through its interaction with TERT (zeige TERT Antikörper).
WNT10A plays an important role in the pathogenesis of IPF via TGF-beta (zeige TGFB1 Antikörper) activation and it may also be a sensitive predictor for the onset of an AE-IPF.
Wnt10a regulates proliferation and apoptosis of embryonic palatal mesenchymal cells at least partially through the canonical Wnt (zeige WNT2 Antikörper)/beta-catenin (zeige CTNNB1 Antikörper) signaling pathway.
Data indicate that that Wnt10a regulates Dspp (zeige DSPP Antikörper) expression in mesenchymal cells.
Histone methyltransferase G9a (zeige EHMT2 Antikörper) represses adipogenesis by inhibiting PPARgamma (zeige PPARG Antikörper) expression and facilitating Wnt10a expression.
Mechanisms downstream of beta-catenin (zeige CTNNB1 Antikörper) are required for Wnt6 (zeige WNT6 Antikörper), Wnt10a and Wnt10b (zeige WNT10B Antikörper) to influence differentiation of mesenchymal precursors.
WNT10A may be a novel angio/stromagenic growth factor
WNT10a rs201002930 significantly decreased the risk of cleft lip with cleft palate and cleft palate only.
WNT10A mutation is associated with tooth agenesis.
High WNT10A expression is associated with papillary thyroid carcinoma.
Mild to severe oligodontia was observed in all patients bearing biallelic WNT10A mutations.
The development of maxillary canine, maxillary second molar and mandibular second molar was statistically significantly delayed in patients with WNT10A variants compared with patients without variants. The impact of WNT10A variants on dental development increases with presence of the nonsense c.(321C>A p.(C107*)) variant and the number of missing teeth
Results from genetic analysis revealed that all seven individuals were homozygous or compound heterozygous for WNT10A mutations suggesting that tooth agenesis and/or peg (zeige PAEP Antikörper)-shaped crowns of primary mandibular incisors, severe oligodontia of permanent dentition as well as ectodermal symptoms of varying severity may be predictors of bi-allelic WNT10A mutations of importance for diagnosis, counselling and follow-up.
miR (zeige MLXIP Antikörper)-378a-3p suppresses hepatic stellate cell activation, at least in part, via targeting of Wnt10a, supporting its potential utility as a novel therapeutic target for liver fibrosis.
risk of hypodontia may be related to the WNT10A polymorphism. Our results also confirm the importance of the Wnt (zeige WNT2 Antikörper) pathway in tooth development.
The WNT gene family consists of structurally related genes which encode secreted signaling proteins. These proteins have been implicated in oncogenesis and in several developmental processes, including regulation of cell fate and patterning during embryogenesis. This gene is a member of the WNT gene family. It is strongly expressed in the cell lines of promyelocytic leukemia and Burkitt's lymphoma. In addition, it and another family member, the WNT6 gene, are strongly coexpressed in colorectal cancer cell lines. The gene overexpression may play key roles in carcinogenesis through activation of the WNT-beta-catenin-TCF signaling pathway. This gene and the WNT6 gene are clustered in the chromosome 2q35 region.
wingless-type MMTV integration site family, member 10A
, Wnt signaling ligand
, Wnt10a protein
, hypothetical protein
, WNT10A protein
, protein Wnt-10a-like
, protein Wnt-10a
, wingless related MMTV integration site 10a