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anti-Human TLR4 Antikörper:
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Human Monoclonal TLR4 Primary Antibody für ChIP, CyTOF - ABIN252522
Kessel, Toubi, Pavlotzky, Mogilner, Coran, Lurie, Karry, Sukhotnik et al.: Treatment with glutamine is associated with down-regulation of Toll-like receptor-4 and myeloid differentiation factor 88 expression and decrease in intestinal mucosal injury caused by ... in Clinical and experimental immunology 2008
Show all 63 Pubmed References
Dog (Canine) Monoclonal TLR4 Primary Antibody für BP, CyTOF - ABIN4360113
Rallabhandi, Bell, Boukhvalova, Medvedev, Lorenz, Arditi, Hemming, Blanco, Segal, Vogel: Analysis of TLR4 polymorphic variants: new insights into TLR4/MD-2/CD14 stoichiometry, structure, and signaling. in Journal of immunology (Baltimore, Md. : 1950) 2006
Show all 44 Pubmed References
Human Monoclonal TLR4 Primary Antibody für BP, CyTOF - ABIN4360167
Degraaf, Zasłona, Bourdonnay, Peters-Golden: Prostaglandin E2 reduces Toll-like receptor 4 expression in alveolar macrophages by inhibition of translation. in American journal of respiratory cell and molecular biology 2014
Show all 24 Pubmed References
Dog (Canine) Monoclonal TLR4 Primary Antibody für FACS - ABIN252065
Konno, Wakabayashi, Akashi-Takamura, Ishii, Kobayashi, Takahashi, Kusumoto, Saitoh, Yoshizawa, Miyake: A molecule that is associated with Toll-like receptor 4 and regulates its cell surface expression. in Biochemical and biophysical research communications 2005
Show all 24 Pubmed References
Dog (Canine) Monoclonal TLR4 Primary Antibody für FACS - ABIN4360117
Mempel, Voelcker, Köllisch, Plank, Rad, Gerhard, Schnopp, Fraunberger, Walli, Ring, Abeck, Ollert et al.: Toll-like receptor expression in human keratinocytes: nuclear factor kappaB controlled gene activation by Staphylococcus aureus is toll-like receptor 2 but not toll-like receptor 4 or platelet ... in The Journal of investigative dermatology 2003
Show all 23 Pubmed References
Human Monoclonal TLR4 Primary Antibody für FACS - ABIN251961
Allam, Peng, Appel, Wenghoefer, Niederhagen, Bieber, Bergé, Novak: Toll-like receptor 4 ligation enforces tolerogenic properties of oral mucosal Langerhans cells. in The Journal of allergy and clinical immunology 2008
Show all 22 Pubmed References
Dog (Canine) Monoclonal TLR4 Primary Antibody für BP, CyTOF - ABIN4360119
Basak, Pathak, Bhattacharyya, Mandal, Pathak, Kundu et al.: NF-kappaB- and C/EBPbeta-driven interleukin-1beta gene expression and PAK1-mediated caspase-1 activation play essential roles in interleukin-1beta release from Helicobacter pylori ... in The Journal of biological chemistry 2005
Show all 22 Pubmed References
Dog (Canine) Monoclonal TLR4 Primary Antibody für FACS, ICC - ABIN4360164
Cognasse, Hamzeh, Chavarin, Acquart, Genin, Garraud: Evidence of Toll-like receptor molecules on human platelets. in Immunology and cell biology 2005
Show all 21 Pubmed References
Dog (Canine) Monoclonal TLR4 Primary Antibody für BP, ELISA - ABIN4360158
Scheel, Papavlassopoulos, Blunck, Gebert, Hartung, Zähringer, Seydel, Schromm: Cell activation by ligands of the toll-like receptor and interleukin-1 receptor family depends on the function of the large-conductance potassium channel MaxiK in human macrophages. in Infection and immunity 2006
Show all 18 Pubmed References
Human Monoclonal TLR4 Primary Antibody für FACS - ABIN4360193
Zanoni, Navone, Lunardi, Tridente, Bason, Sivori, Beri, Dolcino, Valletta, Corrocher, Puccetti: In celiac disease, a subset of autoantibodies against transglutaminase binds toll-like receptor 4 and induces activation of monocytes. in PLoS medicine 2006
Show all 16 Pubmed References
Cerebrospinal fluid soluble TLR2 and TLR4 may play a role in HIV/SIV-related neuroinflammation and subsequent neuropathology.
data confirm that engineered human cells expressing TLR4, MD2 (zeige LY96 Antikörper) and CD14 (zeige NDUFA2 Antikörper) can respond to CMP (zeige MATN1 Antikörper) with NF-kappaB (zeige NFKB1 Antikörper) activation and the response can be influenced by variations in CMP (zeige MATN1 Antikörper)-mannosylation
RBP4 (zeige POLR2D Antikörper)-induced inflammation is largely mediated by TLR4.
novel observation that sCD14 compared with lipopolysaccharide binding protein (zeige LBP Antikörper), offers a preferred target to ameliorate TLR especially TLR4-induced inflammation and insulin (zeige INS Antikörper) resistance in human obesity and metabolic syndrome
Data suggest that dendritic cells are sensitive to environmentally relevant level of aflatoxin B1 (AFB1); TLR2 (zeige TLR2 Antikörper) and TLR4 are involved in sensing AFB1; expression levels of both TLR2 (zeige TLR2 Antikörper) and TLR4 mRNA and proteins were significantly up-regulated in AFB1-treated dendritic cells.
For asthma, there was suggestive evidence of a gene-by-environment interaction for the CD14 (zeige NDUFA2 Antikörper) variant rs2569190 (Interaction P = 0.16) but not for the TLR4 variants rs4986790 and rs4986791.
Toll-like receptor 4 may have a role in non-small cell lung cancer
Activation of TLR3 (zeige TLR3 Antikörper) and TLR4 stimulated the expression of HIF-1 (zeige HIF1A Antikörper) through NF-kappaB (zeige NFKB1 Antikörper) in oral squamous cell carcinoma
essential role of TLR4/ROS (zeige ROS1 Antikörper)/miR (zeige MLXIP Antikörper)-21 pathway in LPS (zeige IRF6 Antikörper)-induced outgrowth of primary human lung cancer
An association with IL-1beta (zeige IL1B Antikörper)-511 locus and IL-1beta (zeige IL1B Antikörper)-511-TLR4-896 diplotype (CC-AA) and Type 2 Diabetes Mellitus is discussed.
analysis of TLR2 (zeige TLR2 Antikörper) rs5743708, TLR2 (zeige TLR2 Antikörper) rs4696480, TLR4 rs4986790, TLR9 (zeige TLR9 Antikörper) rs5743836 and TLR9 (zeige TLR9 Antikörper) rs352140 SNPs in children with pneumococcal and meningococcal meningitis and their family members; study did not show an association between the analyzed SNPs in TLR2 (zeige TLR2 Antikörper), TLR4, and TLR9 (zeige TLR9 Antikörper) and susceptibility to develop bacterial meningitis
TLR4 deletion does not protect from cast immobilization-induced muscle atrophy.
PCSK9 (zeige PCSK9 Antikörper) may act as an inflammatory mediator in the pathogenesis of atherosclerosis via TLR4/NF-kappaB (zeige NFKB1 Antikörper) signaling pathway.
ESAT6 may induce renal injury by promoting miR (zeige MLXIP Antikörper)-155 expression through the TLR-4/MyD88 (zeige MYD88 Antikörper) signaling pathway in MTB (zeige NCAPG2 Antikörper)-infected mice.
Ultraviolet radiation engages TLR4/MyD88 (zeige MYD88 Antikörper) as a death signaling complex in macrophages.
These results suggest that TLR2 (zeige TLR2 Antikörper) and TLR4 play cooperative roles in lung innate immune responses and bacterial dissemination, resulting in systemic inflammation during Klebsiella pneumoniae infection.
Findings identify a critical role for IL1Ra (zeige IL1RN Antikörper) in maintaining the natural diversity and composition of intestinal microbiota, and suggest a role for TLR4 in mucosal Th17 cell induction associated with the development of autoimmune disease in mice.
TLR4-NF-kappaB (zeige NFKB1 Antikörper) signal pathway is inhibited by paeonol in endotoxin-induced acute kidney injury
The present study shows that TLR-4 deficiency and genetic background influence the outcome of muscular tissue repair in aseptic lesions and yet still maintaining some level of signaling in the TLR4-mutant mice.
These data indicate that TLR4 plays a protective role in stress-induced gastric ulcer that is exerted via impacting synthesis of glucocorticoid in the adrenal gland.
STA3 (zeige ARHGEF3 Antikörper) facilitates TLR4-dependent IL-6 (zeige IL6 Antikörper) and IL-8 (zeige IL8 Antikörper) production via IL-6 (zeige IL6 Antikörper) receptor-positive feedback in endometrial cells.
Studied genetic diversity of the Toll-like receptor gene TLR4 in Czech Red and Czech Red Pied cattle. Found 8 SNPs, which were grouped into 18 haplotypes.
TLR4 polymorphisms are associated with lower reproductive Performance.
As a pilot study, the present results revealed that identified SNPs in IL8 (zeige IL8 Antikörper) and TLR4 genes can be used as a genetic marker and predisposing factor for resistance/susceptibility to digital dermatitis in dairy cows. However, TLR4 gene may be a potential candidate for such disease.
Transcription levels of TLR2 (zeige TLR2 Antikörper), TLR4, and CD14 (zeige CD14 Antikörper) in Holstein cows with retained placenta significantly decreased between the first and the seventh day postpartum.
Bovine viral diarrhea virus type 2 infection modulates TLR4 responsiveness in differentiated myeloid cells.
TLR2 (zeige TLR2 Antikörper) and TLR4 mediate innate response against Cryptosporidium parvum in bovine intestinal epithelial cells.
TLR4 polymorphisms are associated with susceptibility to Mycobacterium avium ssp. paratuberculosis infection in Holsteins
positive correlation between lower neutrophil apoptosis and higher expression of TLR2 (zeige TLR2 Antikörper) and TLR4 with the formation of NETs and change in surface architecture.
Studied SNPs in the bovine toll-like receptor 4 (TLR4) and monocyte chemo attractant protein-1(CCL2 (zeige CCL2 Antikörper)) genes.
TLR2 (zeige TLR2 Antikörper), 3, 4, and 8 mRNA expression is strongly upregulated and correlates with the progression of atherosclerosis in the aorta. Fluvastatin significantly inhibited this progress and reduced inflammation via TLR downregulation.
The expression of TLR4 protein and mRNA, the level of activated NF-kappaB (zeige NFKB1 Antikörper) (p65 (zeige SYT1 Antikörper)) were respectively detected.
Lipopolysaccharide upregulates the expression of rabbit TLR2 (zeige TLR2 Antikörper) and 4 in the uterine body and horn, and the expression of TLR4 in the ovary.
Polydatin might have a protective effect on lung ischemia/reperfusion injury by down-regulating TLR4 and NF-kappaB (zeige NFKB1 Antikörper) expression, then inhibiting the release of mediators of inflammation as ICAM-1 (zeige ICAM1 Antikörper).
TLR4 expression is upregulated in the brain after experimental subarachnoid haemorrhage
The elevated expression of TLR4 was detected after SAH (zeige ACSM3 Antikörper) and peaked on day 3 and 5. TLR4 is increasingly expressed in a parallel time course to the development of cerebral vasospasm in a rabbit experimental model of SAH (zeige ACSM3 Antikörper).
These results further confirm the involvement of the TLR4 signaling pathway in resistance to E. coli F18 (zeige MAMLD1 Antikörper) in Meishan weaned piglets.
Data suggest expression of TLR4 and NFKB (nuclear factor kappa B) are regulated by dietary factors affecting innate immunity; here, Lactobacillus acidophilus in feed down-regulates expression of TLR4 and NFKB in mononuclear cells after LPS (zeige IRF6 Antikörper) challenge.
At 30 days after autotransplantation of a pig kidney, mRNA expression increases for TLR4.
Data suggest TLR2 (zeige TLR2 Antikörper), TLR4, and calcium signaling in enterocytes play principal roles in mucosal immunity against enterotoxigenic Escherichia coli; probiotic Lactobacillus delbrueckii and its extracellular polysaccharides appear to stimulate TLR2 (zeige TLR2 Antikörper)/TLR4.
TLR2 (zeige TLR2 Antikörper) is required for the suppression of TLR4 signaling activation.
The current study screened for single nucleotide polymorphisms (SNPs) in the TLR4 gene and tested their association with Salmonella fecal shedding.
The role of TLR2 (zeige TLR2 Antikörper), TLR4 and RP105 (zeige CD180 Antikörper)/MD1 (zeige LY86 Antikörper) in the immunoregulatory effect of acidic exopolysaccharides from Lactobacillus plantarum N14 (zeige CLPTM1 Antikörper), is reported.
Data suggest expression of TLR4 in liver can be regulated by dietary factors; here, supplementation with aspartate down-regulates expression of TLR4 in liver in a model of liver disease.
Fish Oil attenuates the activation of the HPA (zeige HPSE Antikörper) axis induced by LPS (zeige IRF6 Antikörper) challenge. So it may be associated with decreasing the production of brain or peripheral proinflammatory cytokines through inhibition of TLR4 and NOD signaling pathways in weaned pigs.
Single nucleotide polymorphisms in TLR4 is associated with immune response to gram-negative bacterial infections.
The research findings suggest that Th17 cells are involved in active equine inflammatory bowel disease, and that TLR4 expression was increased in affected horses.
A low steady expression of TLR4, MD-2 (zeige LY96 Antikörper) and CD14 (zeige CD14 Antikörper) mRNA was demonstrated for the intestinal samples with no variation between the intestinal segments analysed.
In the present study, the authors show that TLR4 expression is significantly decreased following the exogenous expression of BPV-1 E2 and E7 in primary equine fibroblasts.
evidence that pulmonary intravascular macrophages are equipped with TLR4 to handle and rapidly respond to circulating endotoxins
TLR4/MD-2 (zeige LY96 Antikörper) complex is responsible for recognition of Rhodococcus spheroides lipopolysaccharide as an agonist in equine cells.
The protein encoded by this gene is a member of the Toll-like receptor (TLR) family which plays a fundamental role in pathogen recognition and activation of innate immunity. TLRs are highly conserved from Drosophila to humans and share structural and functional similarities. They recognize pathogen-associated molecular patterns that are expressed on infectious agents, and mediate the production of cytokines necessary for the development of effective immunity. The various TLRs exhibit different patterns of expression. This receptor has been implicated in signal transduction events induced by lipopolysaccharide (LPS) found in most gram-negative bacteria. Mutations in this gene have been associated with differences in LPS responsiveness. Multiple transcript variants encoding different isoforms have been found for this gene.
, homolog of Drosophila toll
, lipopolysaccharide response
, Toll-like receptor4 protein
, Toll-like receptor 4-like protein