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anti-Human TLR4 Antikörper:
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Human Monoclonal TLR4 Primary Antibody für ChIP, CyTOF - ABIN252522
Kessel, Toubi, Pavlotzky, Mogilner, Coran, Lurie, Karry, Sukhotnik et al.: Treatment with glutamine is associated with down-regulation of Toll-like receptor-4 and myeloid differentiation factor 88 expression and decrease in intestinal mucosal injury caused by ... in Clinical and experimental immunology 2008
Show all 63 Pubmed References
Dog (Canine) Monoclonal TLR4 Primary Antibody für BP, CyTOF - ABIN4360113
Rallabhandi, Bell, Boukhvalova, Medvedev, Lorenz, Arditi, Hemming, Blanco, Segal, Vogel: Analysis of TLR4 polymorphic variants: new insights into TLR4/MD-2/CD14 stoichiometry, structure, and signaling. in Journal of immunology (Baltimore, Md. : 1950) 2006
Show all 45 Pubmed References
Dog (Canine) Monoclonal TLR4 Primary Antibody für FACS - ABIN252065
Konno, Wakabayashi, Akashi-Takamura, Ishii, Kobayashi, Takahashi, Kusumoto, Saitoh, Yoshizawa, Miyake: A molecule that is associated with Toll-like receptor 4 and regulates its cell surface expression. in Biochemical and biophysical research communications 2005
Show all 24 Pubmed References
Human Monoclonal TLR4 Primary Antibody für BP, CyTOF - ABIN4360167
Degraaf, Zasłona, Bourdonnay, Peters-Golden: Prostaglandin E2 reduces Toll-like receptor 4 expression in alveolar macrophages by inhibition of translation. in American journal of respiratory cell and molecular biology 2014
Show all 24 Pubmed References
Dog (Canine) Monoclonal TLR4 Primary Antibody für FACS - ABIN4360117
Mempel, Voelcker, Köllisch, Plank, Rad, Gerhard, Schnopp, Fraunberger, Walli, Ring, Abeck, Ollert et al.: Toll-like receptor expression in human keratinocytes: nuclear factor kappaB controlled gene activation by Staphylococcus aureus is toll-like receptor 2 but not toll-like receptor 4 or platelet ... in The Journal of investigative dermatology 2003
Show all 23 Pubmed References
Dog (Canine) Monoclonal TLR4 Primary Antibody für BP, CyTOF - ABIN4360119
Basak, Pathak, Bhattacharyya, Mandal, Pathak, Kundu et al.: NF-kappaB- and C/EBPbeta-driven interleukin-1beta gene expression and PAK1-mediated caspase-1 activation play essential roles in interleukin-1beta release from Helicobacter pylori ... in The Journal of biological chemistry 2005
Show all 22 Pubmed References
Dog (Canine) Monoclonal TLR4 Primary Antibody für FACS, ICC - ABIN4360164
Cognasse, Hamzeh, Chavarin, Acquart, Genin, Garraud: Evidence of Toll-like receptor molecules on human platelets. in Immunology and cell biology 2005
Show all 21 Pubmed References
Dog (Canine) Monoclonal TLR4 Primary Antibody für BP, ELISA - ABIN4360158
Scheel, Papavlassopoulos, Blunck, Gebert, Hartung, Zähringer, Seydel, Schromm: Cell activation by ligands of the toll-like receptor and interleukin-1 receptor family depends on the function of the large-conductance potassium channel MaxiK in human macrophages. in Infection and immunity 2006
Show all 18 Pubmed References
Human Monoclonal TLR4 Primary Antibody für FACS - ABIN4360193
Zanoni, Navone, Lunardi, Tridente, Bason, Sivori, Beri, Dolcino, Valletta, Corrocher, Puccetti: In celiac disease, a subset of autoantibodies against transglutaminase binds toll-like receptor 4 and induces activation of monocytes. in PLoS medicine 2006
Show all 16 Pubmed References
Human Monoclonal TLR4 Primary Antibody für FACS - ABIN4360159
Allam, Peng, Appel, Wenghoefer, Niederhagen, Bieber, Bergé, Novak: Toll-like receptor 4 ligation enforces tolerogenic properties of oral mucosal Langerhans cells. in The Journal of allergy and clinical immunology 2008
Show all 15 Pubmed References
The immunoenhancement effect of PSP (zeige MSMB Antikörper) against lung cancer is mediated by TLR4-MAPK (zeige MAPK1 Antikörper)/NF-kappaB (zeige NFKB1 Antikörper) signaling pathways
LPS (zeige IRF6 Antikörper) stimulation induced TLR4 expression and increased pigmentation. TLR4 expression was not detected after single-dose UVA or UVB treatment, but pigmentation increased. Repeated UV treatment induced TLR4 expression and increased pigmentation. LPS (zeige IRF6 Antikörper) stimulation and repeated UV treatment increased IL-6 (zeige IL6 Antikörper) secretion, and repeated UVB treatment increased IL-10 (zeige IL10 Antikörper) secretion.
data suggest that high-phosphate conditions directly induce vascular calcification via the activation of TLR4/NF-kappaB (zeige NFKB1 Antikörper) signaling in VSMCs
LncRNA MEG3 (zeige FAM129B Antikörper) ameliorates respiratory syncytial virus infection by suppressing TLR4 signaling.
Study shows that the toll-like receptor 4 gene rs1927914 polymorphism was associated with susceptibility to ischemic stroke in males. Moreover, the rs10759932 polymorphism may affect inflammatory response in ischemic stroke patients.
There was no difference found in MMP-2 (zeige MMP2 Antikörper), MMP-9 (zeige MMP9 Antikörper) or TLR-4 levels between non-thrombocytopenic and thrombocytopenic septic donors. PLA formation was increased in thrombocytopenic patients.
there was a negative correlation between YKL-40 (zeige CHI3L1 Antikörper) and TLR4 expression in chronic sinusitis patients with nasal polyps. YKL-40 (zeige CHI3L1 Antikörper) and TLR4 interacted with each other to activate NF-kappaB (zeige NFKB1 Antikörper) and promote disease progression.
High TLR4 expression is associated with hepatocellular carcinoma.
High expression of MMP-9 (zeige MMP9 Antikörper) and TLR4 in patients with COPD (zeige ARCN1 Antikörper) may promote inflammatory cell infiltration, induce proliferation of smooth muscle cells, degrade extracellular matrix, and play an important role in lung revascularization.
our findings confirm that in south Tunisian patients with IBD, the TLR4-Thr399Ile variant is strongly associated with susceptibility to CD and that the two polymorphisms of this receptor (TLR4-Thr399Ile and TLR4-Asp299Gly) may play a role in the clinical expression of UC.
Study showed the significance of TLR4 in upregulation of MAPK/ErK (zeige MAPK1 Antikörper) in NMDAR (zeige GRIN1 Antikörper) hypofunction schizophrenic-mouse model. This is characterized by a decrease in hippocampal IGF-1R (zeige IGF1R Antikörper)/CaMKIIalpha (zeige CAMK2 Antikörper), and social interaction behavioral deficits. Ultimately, hippocampal TLR4 knockdown rescued IGF-1R (zeige IGF1R Antikörper)/CaMKIIalpha (zeige CAMK2 Antikörper) decline and prevented behavioral deficits by reducing MAPK/ErK (zeige MAPK1 Antikörper) expression.
Study highlights the importance of Ca(2 (zeige CA2 Antikörper)+) signaling in macrophage activation and identifies the ion channel TRPM7 (zeige TRPM7 Antikörper) as a central component of TLR4 signaling.
The immunoenhancement effect of PSP (zeige BPIFA2 Antikörper) against lung cancer is mediated by TLR4-MAPK (zeige MAPK1 Antikörper)/NF-kappaB (zeige NFKB1 Antikörper) signaling pathways
knockout of TLR4 gene improved survival and cardiac function in severe sepsis induced by CLP by decreasing the myocardial levels of inflammatory cytokines, weakening neutrophil infiltration in myocardium, and attenuating the heart apoptosis.
beta-arrestin 2 (zeige ARRB2 Antikörper) can protect liver tissue from LPS-induced injury via inhibition of TLR4/NF-kappaB (zeige NFKB1 Antikörper) signaling pathway-mediated inflammation.
TLR4 mutation can decrease the expression of inflammatory factors and enhance the speed of recovery after sciatic nerve injury.
Toll-like receptor (TLR) 4, the expression of which was upregulated in B cells after ultraviolet B exposure, played an essential role in the induction of regulatory B cells.
type I and II IFN as in vivo modifiers of LIC fate that may significantly affect the impact of putative leukemia-driving activities such as the ETV6 (zeige ETV6 Antikörper)-RUNX1 (zeige RUNX1 Antikörper)-mediated growth advantage in the presence of TGF-beta (zeige TGFB1 Antikörper) and TLR4-upregulated mutagenic activity
Pudilan xiaoyan oral liquid prevents LPS-induced respiratory in fl ammation via effects on TLR4/NF-kappaB (zeige NFKB1 Antikörper) signaling.
NOD2 (zeige NOD2 Antikörper) Modulates Serotonin Transporter (zeige SLC6A4 Antikörper) and Interacts with TLR2 and TLR4 in Intestinal Epithelial Cells
Based on the impact of both candidate genes,TLR4 and CACNA2D1 (zeige CACNA2D1 Antikörper), on udder health, linear or generalized linear mixed models was applied for testing the associations of SNPs located in the genes and clinical mastitis
a single nucleotide polymorphism of the bovine toll like receptor 4 gene (TLR4) in New Zealand (NZ) Holstein-Friesian x Jersey (HF x J) cross dairy cows was associated with milk production traits
STA3 (zeige ARHGEF3 Antikörper) facilitates TLR4-dependent IL-6 (zeige IL6 Antikörper) and IL-8 (zeige IL8 Antikörper) production via IL-6 (zeige IL6 Antikörper) receptor-positive feedback in endometrial cells.
Studied genetic diversity of the Toll-like receptor gene TLR4 in Czech Red and Czech Red Pied cattle. Found 8 SNPs, which were grouped into 18 haplotypes.
TLR4 polymorphisms are associated with lower reproductive Performance.
As a pilot study, the present results revealed that identified SNPs in IL8 (zeige IL8 Antikörper) and TLR4 genes can be used as a genetic marker and predisposing factor for resistance/susceptibility to digital dermatitis in dairy cows. However, TLR4 gene may be a potential candidate for such disease.
Transcription levels of TLR2, TLR4, and CD14 (zeige CD14 Antikörper) in Holstein cows with retained placenta significantly decreased between the first and the seventh day postpartum.
Bovine viral diarrhea virus type 2 infection modulates TLR4 responsiveness in differentiated myeloid cells.
TLR2 and TLR4 mediate innate response against Cryptosporidium parvum in bovine intestinal epithelial cells.
TLR4 polymorphisms are associated with susceptibility to Mycobacterium avium ssp. paratuberculosis infection in Holsteins
TLR2, 3, 4, and 8 mRNA expression is strongly upregulated and correlates with the progression of atherosclerosis in the aorta. Fluvastatin significantly inhibited this progress and reduced inflammation via TLR downregulation.
The expression of TLR4 protein and mRNA, the level of activated NF-kappaB (zeige NFKB1 Antikörper) (p65 (zeige SYT1 Antikörper)) were respectively detected.
Lipopolysaccharide upregulates the expression of rabbit TLR2 and 4 in the uterine body and horn, and the expression of TLR4 in the ovary.
Polydatin might have a protective effect on lung ischemia/reperfusion injury by down-regulating TLR4 and NF-kappaB (zeige NFKB1 Antikörper) expression, then inhibiting the release of mediators of inflammation as ICAM-1 (zeige ICAM1 Antikörper).
TLR4 expression is upregulated in the brain after experimental subarachnoid haemorrhage
The elevated expression of TLR4 was detected after SAH (zeige ACSM3 Antikörper) and peaked on day 3 and 5. TLR4 is increasingly expressed in a parallel time course to the development of cerebral vasospasm in a rabbit experimental model of SAH (zeige ACSM3 Antikörper).
The TWEAK (zeige TNFSF12 Antikörper)-independent Fn14 (zeige TNFRSF12A Antikörper) activation augments TLR4-mediated inflammatory responses in the intestine of piglets.
These results further confirm the involvement of the TLR4 signaling pathway in resistance to E. coli F18 (zeige MAMLD1 Antikörper) in Meishan weaned piglets.
Data suggest expression of TLR4 and NFKB (nuclear factor kappa B) are regulated by dietary factors affecting innate immunity; here, Lactobacillus acidophilus in feed down-regulates expression of TLR4 and NFKB in mononuclear cells after LPS (zeige IRF6 Antikörper) challenge.
At 30 days after autotransplantation of a pig kidney, mRNA expression increases for TLR4.
Data suggest TLR2, TLR4, and calcium signaling in enterocytes play principal roles in mucosal immunity against enterotoxigenic Escherichia coli; probiotic Lactobacillus delbrueckii and its extracellular polysaccharides appear to stimulate TLR2/TLR4.
TLR2 is required for the suppression of TLR4 signaling activation.
The current study screened for single nucleotide polymorphisms (SNPs) in the TLR4 gene and tested their association with Salmonella fecal shedding.
The role of TLR2, TLR4 and RP105 (zeige CD180 Antikörper)/MD1 (zeige LY86 Antikörper) in the immunoregulatory effect of acidic exopolysaccharides from Lactobacillus plantarum N14 (zeige CLPTM1 Antikörper), is reported.
Data suggest expression of TLR4 in liver can be regulated by dietary factors; here, supplementation with aspartate down-regulates expression of TLR4 in liver in a model of liver disease.
Fish Oil attenuates the activation of the HPA (zeige HPSE Antikörper) axis induced by LPS (zeige IRF6 Antikörper) challenge. So it may be associated with decreasing the production of brain or peripheral proinflammatory cytokines through inhibition of TLR4 and NOD signaling pathways in weaned pigs.
The research findings suggest that Th17 cells are involved in active equine inflammatory bowel disease, and that TLR4 expression was increased in affected horses.
A low steady expression of TLR4, MD-2 (zeige LY96 Antikörper) and CD14 (zeige CD14 Antikörper) mRNA was demonstrated for the intestinal samples with no variation between the intestinal segments analysed.
In the present study, the authors show that TLR4 expression is significantly decreased following the exogenous expression of BPV-1 E2 and E7 in primary equine fibroblasts.
evidence that pulmonary intravascular macrophages are equipped with TLR4 to handle and rapidly respond to circulating endotoxins
TLR4/MD-2 (zeige LY96 Antikörper) complex is responsible for recognition of Rhodococcus spheroides lipopolysaccharide as an agonist in equine cells.
The protein encoded by this gene is a member of the Toll-like receptor (TLR) family which plays a fundamental role in pathogen recognition and activation of innate immunity. TLRs are highly conserved from Drosophila to humans and share structural and functional similarities. They recognize pathogen-associated molecular patterns that are expressed on infectious agents, and mediate the production of cytokines necessary for the development of effective immunity. The various TLRs exhibit different patterns of expression. This receptor has been implicated in signal transduction events induced by lipopolysaccharide (LPS) found in most gram-negative bacteria. Mutations in this gene have been associated with differences in LPS responsiveness. Multiple transcript variants encoding different isoforms have been found for this gene.
, homolog of Drosophila toll
, lipopolysaccharide response
, Toll-like receptor4 protein
, Toll-like receptor 4-like protein