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anti-Mouse (Murine) Ankyrin G Antikörper:
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Mammalian Monoclonal Ankyrin G Primary Antibody für ISt, IHC - ABIN1304536
Komulainen, Zdrojewska, Freemantle, Mohammad, Kulesskaya, Deshpande, Marchisella, Mysore, Hollos, Michelsen, Mågard, Rauvala, James, Coffey: JNK1 controls dendritic field size in L2/3 and L5 of the motor cortex, constrains soma size, and influences fine motor coordination. in Frontiers in cellular neuroscience 2014
Show all 74 Pubmed References
Mammalian Monoclonal Ankyrin G Primary Antibody für ISt, IHC - ABIN1304537
Winterflood, Platonova, Albrecht, Ewers: Dual-Color 3D Superresolution Microscopy by Combined Spectral-Demixing and Biplane Imaging. in Biophysical journal 2015
Show all 13 Pubmed References
Mammalian Monoclonal Ankyrin G Primary Antibody für ISt, IHC - ABIN1304534
Yamankurt, Wu, McCarthy, Cunha: Exon organization and novel alternative splicing of Ank3 in mouse heart. in PLoS ONE 2015
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Our data suggest that alterations of Ank3 expression and function may contribute to the effects of ELS on the development of psychiatric disorders.
Interaction of Ankyrin-G with EB1 (zeige MAPRE1 Antikörper) protein drives axon initial segment formation and neuronal polarity.
Ank3 is subject to complex alternative splicing regulation resulting in a diverse population of ankyrin-G isoforms in heart.
This study found that ankyrin-G regulates canonical Wnt (zeige WNT2 Antikörper) signaling by altering the subcellular localization and availability of beta-catenin (zeige CTNNB1 Antikörper) in proliferating cells.
The data support a model where ankyrinG-binding is required for preferential Nav1.6 (zeige SCN8A Antikörper) insertion into the axon initial segment plasma membrane during development.
CK2 (zeige CSNK2A1 Antikörper)-regulated IQJC-SCHIP-1 association with AnkG contributes to axon initial segment maintenance.
the structures of ANK (zeige ANKH Antikörper) repeats in complex with an inhibitory segment from the C-terminal regulatory domain and with a sodium channel Nav1.2 (zeige SCN2A Antikörper) peptide, are reported.
The giant exon of AnkG is required for assembly of the AIS (zeige AR Antikörper) and nodes of Ranvier and was a transformative innovation in evolution of the vertebrate nervous system that now is a potential target in neurodevelopmental disorders.
giant ankyrin-G promotes GABAergic synapse stability through opposing endocytosis of GABAA (zeige GABRg1 Antikörper) receptors, and requires a newly described interaction with GABARAP (zeige GABARAP Antikörper), a GABAA receptor-associated protein (zeige GABARAP Antikörper).
In embryonic mouse axons, AnkG is expressed at the beginning of axonogenesis at E9.5 and initially distributed homogeneously along the entire growing axon. From E11.5 the protein progressively becomes restricted to the proximal axon.
Ankyrin-G targets Nav1.5 (zeige SCN5A Antikörper) to the intercalated disc. Ank3 KO mice display conduction defects and arrhythmias.
ANK3 expression is associated with androgen receptor (zeige AR Antikörper) stability, invasiveness, and lethal outcome in prostate cancer patients.
Nonsense mutation in ANK3 was identified in a patient with speech impairment, intellectual disability and autistic features.
Decreases in UF FA were observed among BD subjects carrying the ANK3 rs9804190 risk allele (T), compared to CC and T-carrier control subjects and BD CC homozygotes.
This study identified a SNP in ANK3 with a strong protective effect for Bipolar Disorder and Schizophrenia.
The haplotype analysis results suggest that ANK3 variants rs1938526 and rs10994336 may confer susceptibility for BD in the Korean population. Association analysis revealed a probable genetic difference between Korean and Caucasian populations in the degree of ANK3 involvement in BD pathogenesis.
The ankyrin 3 genotype may be associated with pathogenesis of age-related neurodegeneration, and, in part, of bipolar disorder.
Whole-exome sequencing in ASD (zeige ARSD Antikörper) patients from each family identified a second rare inherited genetic variant, affecting either the ANK3 genes encoding NLGN4X (zeige NLGN4X Antikörper) interacting proteins expressed in inhibitory or in excitatory synapses.
we investigated the association of CACNA1C (zeige CACNA1C Antikörper) and ANK3 with SZ using meta-analytic techniques.
ankyrin-G associates with and inhibits the endocytosis of VE-cadherin (zeige CDH5 Antikörper) cis (zeige CISH Antikörper) dimers.
ANK3 rs10761482 showed a significant association with bipolar disorder
Ankyrins are a family of proteins that are believed to link the integral membrane proteins to the underlying spectrin-actin cytoskeleton and play key roles in activities such as cell motility, activation, proliferation, contact, and the maintenance of specialized membrane domains. Multiple isoforms of ankyrin with different affinities for various target proteins are expressed in a tissue-specific, developmentally regulated manner. Most ankyrins are typically composed of three structural domains: an amino-terminal domain containing multiple ankyrin repeats\; a central region with a highly conserved spectrin binding domain\; and a carboxy-terminal regulatory domain which is the least conserved and subject to variation. Ankyrin 3 is an immunologically distinct gene product from ankyrins 1 and 2, and was originally found at the axonal initial segment and nodes of Ranvier of neurons in the central and peripheral nervous systems. Multiple transcript variants encoding different isoforms have been found for this gene.
, ankyrin 3, node of Ranvier (ankyrin G)
, ankyrin 3, node of Ranvier (ankyrin G)-like
, brain-specific ankyrin-G
, ankyrin G119