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Cardiac transgenic vascular endothelial growth factor-B overexpression failed to protect heart transplants from ischemia-reperfusion injury.
renal VEGF-B expression correlates with the severity of Diabetic Kidney Disease.
Data show that metformin treatment reduces serum vascular endothelial growth factor B (VEGF-B) levels and ameliorates insulin (zeige INS Proteine) resistance.
VEGFA (zeige VEGFA Proteine) and VEGFB associated sub-network, kinase insert domain receptor (KDR (zeige KDR Proteine)), fibronectin 1 (FN1 (zeige FN1 Proteine)), transforming growth factor beta induced (TGFBI) and proliferating cell nuclear antigen (PCNA (zeige PCNA Proteine)) found to interact with at least two of the three hub genes.
Frameshift mutations of VEGFB gene is associated with stomach and colorectal cancers.
plasma VEGF levels before treatment were lower in patients with schizophrenia and that their VEGF levels increased after treatment.
fluid shear stress induces the synthesis of Insulin growth factor-2 and vascular endothelial growth factor (VEGF) B and D, which in turn transactivate MMP-12.
MMP9 (zeige MMP9 Proteine) may activate VEGF-B via PI3K (zeige PIK3CA Proteine)/Akt (zeige AKT1 Proteine) signaling pathway.
Roles of vascular endothelial growth factor in amyotrophic lateral sclerosis.
Low VEGFB and VEGFD (zeige Figf Proteine) gene expression is associated with early-stage non-small cell lung cancer.
Data suggest that dietary fatty acids create epigenetic/nutrigenomic changes in methylation levels of CpG island at Vegfb promoter and changes in Vegfb expression in white adipocytes in vivo and in vitro, with particular up-regulation of DNA methylation (zeige HELLS Proteine) by linoleic acid; Vegfb promoter methylation levels are closely related to Vegfb gene expression in visceral and subcutaneous adipose tissue.
PGC-1alpha is a master metabolic sensor that regulates the expression of Vegfa (zeige VEGFA Proteine) and Vegfb, coordinates blood vessels growth and fatty acid uptake with mitochondrial fatty acid oxidation.
These data therefore support a tightly controlled, paracrine signaling mechanism of VEGF-B to VEGFR1 (zeige FLT1 Proteine).
VEGFB-Induced Vascular Remodeling in Adipose Tissue Requires VEGF (zeige VEGFA Proteine)/VEGFR2 (zeige KDR Proteine). VEGFB-Induced Vascular Remodeling Improves Insulin (zeige INS Proteine) Supply, Signaling, and Function in Obese Mice.
VEGF-B is dispensable for normal cardiac function under unstressed conditions and for high fat diet-induced metabolic changes.
VEGF-B is a vascular remodeling factor promoting cancer metastasis.
Data indicate that vascular endothelial growth factor B knockout (Vegf-b-/-) mice showed impaired nerve repair with concomitant impaired trophic function.
Data indicate that VEGF-B is a high-affinity VEGFR-1 (zeige FLT1 Proteine) ligand that, unlike PlGF (zeige PGF Proteine), cannot efficiently induce signaling downstream of VEGFR-1 (zeige FLT1 Proteine).
results demonstrate that the vascular endothelium can function as an efficient barrier to excess muscle lipid uptake even under conditions of severe obesity and type 2 diabetes, and that this barrier can be maintained by inhibition of VEGF-B signalling
PCR-SSCP and DNA sequencing methods were applied to reveal 3 SNPs and a duplication in the bovine VEGF-B gene among 675 samples belonging to three native Chinese cattle breeds.
This gene encodes a member of the PDGF (platelet-derived growth factor)/VEGF (vascular endothelial growth factor) family. The VEGF family members regulate the formation of blood vessels and are involved in endothelial cell physiology. This member is a ligand for VEGFR-1 (vascular endothelial growth factor receptor 1) and NRP-1 (neuropilin-1). Studies in mice showed that this gene was co-expressed with nuclear-encoded mitochondrial genes and the encoded protein specifically controlled endothelial uptake of fatty acids. Alternatively spliced transcript variants encoding distinct isoforms have been identified.