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anti-Human VEGF Antikörper:
anti-Mouse (Murine) VEGF Antikörper:
anti-Rat (Rattus) VEGF Antikörper:
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Human Polyclonal VEGF Primary Antibody für IHC (p), WB - ABIN3044494
Jiang, Han, Li, Yang, Liu: Carboxymethyl chitosan represses tumor angiogenesis in vitro and in vivo. in Carbohydrate polymers 2015
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Human Polyclonal VEGF Primary Antibody für IHC (p), WB - ABIN3042324
Song, Yue, Li, Li, Zhao, Zhang: Study of the mechanism of sonodynamic therapy in a rat glioma model. in OncoTargets and therapy 2014
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Guinea Pig Polyclonal VEGF Primary Antibody für IF (p), IHC (p) - ABIN707186
Wang, Liao, Wang, Deng, Yu: Transplantation of bone marrow stromal cells overexpressing human vascular endothelial growth factor 165 enhances tissue repair in a rat model of radiation-induced injury. in Chinese medical journal 2014
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Human Monoclonal VEGF Primary Antibody für Neut, ELISA - ABIN1003427
Kim, Li, Houck, Winer, Ferrara: The vascular endothelial growth factor proteins: identification of biologically relevant regions by neutralizing monoclonal antibodies. in Growth factors (Chur, Switzerland) 1992
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Human Polyclonal VEGF Primary Antibody für IHC, ELISA - ABIN1585685
Dai, Feng, Ye, Wu, Liu, Liao, Cao: Exogenous avian leukosis virus-induced activation of the ERK/AP1 pathway is required for virus replication and correlates with virus-induced tumorigenesis. in Scientific reports 2016
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Human Polyclonal VEGF Primary Antibody für ELISA, WB - ABIN541854
Gu, Brännström, Jiang, Bergh, Wester: Vascular endothelial growth factor-A and -C protein up-regulation and early angiogenesis in a rat photothrombotic ring stroke model with spontaneous reperfusion. in Acta neuropathologica 2001
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Human Monoclonal VEGF Primary Antibody für PLA, ELISA - ABIN521322
Arai, Kawachi, Setiawan, Kobayashi: Hypoxia-selective growth inhibition of cancer cells by furospinosulin-1, a furanosesterterpene isolated from an Indonesian marine sponge. in ChemMedChem 2010
Human Polyclonal VEGF Primary Antibody für IF (p), IHC (p) - ABIN675893
Ryzhov, Goldstein, Novitskiy, Blackburn, Biaggioni, Feoktistov: Role of A2B adenosine receptors in regulation of paracrine functions of stem cell antigen 1-positive cardiac stromal cells. in The Journal of pharmacology and experimental therapeutics 2012
Rat (Rattus) Polyclonal VEGF Primary Antibody für IHC, ELISA - ABIN802829
Zhang, Kong, Chen, Zhang, Lian, Zhu, Lu, Zheng: Peroxisome proliferator-activated receptor-? interrupts angiogenic signal transduction by transrepression of platelet-derived growth factor-? receptor in hepatic stellate cells. in Journal of cell science 2014
Human Monoclonal VEGF Primary Antibody für CyTOF, FACS - ABIN4900874
Banerjee, Lin, Skinner, Schiffhauer, Peacock, Hicks, Redmond, Morrow, Huston, Shayne, Langstein, Miller-Graziano, Strickland, ODonoghue, De: Heat shock protein 27 differentiates tolerogenic macrophages that may support human breast cancer progression. in Cancer research 2011
miR (zeige MYLIP Antikörper)-9 modulation of neuronal VEGF-A controls brain angiogenesis in vivo.
Timelapse analysis of individual cardiomyocyte trajectories reveals misdirected cells in zebrafish pdgfra (zeige PDGFRA Antikörper) mutants, suggesting that PDGF (zeige PDGFA Antikörper) signaling steers cardiomyocytes toward the midline during cardiac fusion. Intriguingly, the ligand pdgfaa is expressed in the endoderm medial to the pdgfra (zeige PDGFRA Antikörper)-expressing myocardial precursors.
This study demonstrates the involvement of VEGF signaling in regulating sustained liver injuries after acute alcohol exposure.
we determined that radial glia control this process via the Vegf decoy receptor sFlt1 (zeige FLT1 Antikörper): sflt1 (zeige FLT1 Antikörper) mutants exhibit the venous over-sprouting observed in radial glia-ablated larvae, and sFlt1 (zeige FLT1 Antikörper) overexpression rescues it. Genetic mosaic analyses show that sFlt1 (zeige FLT1 Antikörper) function in trunk endothelial cells can limit their over-sprouting.
High VEGFA expression is associated with Ectopic Proliferation and Retinal Dysplasia in Zebrafish Optic Pathway Tumors.
Rspo1 (zeige RSPO1 Antikörper) is required for hematopoietic stem cell specification through control of parallel signaling pathways controlling HSC (zeige FUT1 Antikörper) specification: Wnt16 (zeige WNT16 Antikörper)/DeltaC/DeltaD and Vegfa/Tgfbeta1 (zeige TGFB1 Antikörper)
Tmem2 (zeige TMEM2 Antikörper) regulates hyaluronic acid turnover to promote normal Vegf signaling during developmental angiogenesis.
low levels of Vegf signaling promote overall vascular endothelial differentiation and cell survival by upregulating etv2 (zeige ETV2 Antikörper) expression, while high levels of Vegf signaling promote arterial and inhibit venous specification.
altering the amount of VEGF signaling in endothelial cells by stimulating them with different VEGF concentrations triggered distinct and mutually exclusive dynamic Ca(2 (zeige CA2 Antikörper)+) signaling responses that correlated with different cellular behaviors.
Bis (zeige BAG3 Antikörper)(2,3-dibromo-4,5-dihydroxybenzyl) ether downregulate the expression of VEGF and VEGFR (zeige KDR Antikörper), and simultaneously regulates VEGF signaling pathway to prevent angiogenesis.
VEGFA produced in the somites is required to initiate adult haemangioblast programming in the adjacent dorsal lateral plate mesoderm.
sequential, isoform-specific VEGFA signaling successively induces the endothelial, arterial, and hematopoietic stem cell programs in the dorsal aorta.
VEGF induces stromal cell migration or recruitment that are required for blood vessel formation.
increased VEGF(170) levels disturb Hand-1 (zeige HAND1 Antikörper) expression in the region required for normal heart morphogenesis
data for the first time demonstrate a calpain/PTP1B/VEGFR2 negative feedback loop in the regulation of VEGF-induced angiogenesis. Modulation of local PTP1B and/or calpain activities may prove beneficial in the treatment of impaired wound healing in diabetes.
Studied the effect of cyclic uniaxial stretch (20%, 1 Hz), with and without the stimulation of vascular endothelial growth factor (VEGF (zeige VEGF Antikörper)), on sprouting angiogenesis by employing a stretchable three-dimensional cell culture model.
Study shows that VEGFA mRNA in mammalian endothelial cells undergoes programmed translational readthrough (PTR (zeige PTCHD3 Antikörper)) generating VEGF-Ax, an isoform containing a unique 22-amino-acid C terminus extension.
Data indicate that superoxide dismutase (SOD) inhibited high glucose (HG)-induced expression of uPAR and VEGF in bovine retinal microvascular endothelial cell (REC).
Exposure of endothelial cells to VEGF, high glucose, or hydrogen peroxide up-regulated the XBP1 (zeige XBP1 Antikörper)/IRE1 alpha (zeige ERN1 Antikörper) and ATF6 (zeige ATF6 Antikörper) arms of the unfolded protein response compared with untreated cells.
Microvascular endothelial cells are more susceptible than aortic cells to advanced glycation end products-enhanced permeability and that AGE-enhanced permeability is dependent on VEGF expression induced by reactive oxygen species.
VEGF supports germ cell survival and sperm production in bulls.
analysis of polymorphisms of bovine VEGF gene and their associations with growth traits in Chinese cattle
VEGF mRNA expression at estrus was higher than at the early I, early II and late luteal stages (P<0.05), whereas VEGF protein content was greatest at the early I luteal stage and decreased thereafter
Alterations in the expression of VEGF-A and bFGF (zeige FGF2 Antikörper) systems suggest that angiogenic factors are involved in abnormal placental development in cloned gestations, contributing to impaired fetal development and poor survival rates.
The VEGF/sVEGF-R1 ratio in follicular fluid on the day of oocyte retrieval in women undergoing IVF (zeige SCN5A Antikörper) procedure, regardless of the type of stimulation protocol, might predict the risk of developing ovarian hyperstimulation syndrome (OHSS). To the best of our knowledge this is the first paper in the literature to show interplay among VEGF, EG-VEGF (zeige Prok1 Antikörper) and sVEGF-R1 and the correlation between their concentration and OHSS risk.
STAT1 (zeige STAT1 Antikörper) knockdown using an inhibitor and siRNA attenuated the IL-17 (zeige IL17A Antikörper)-mediated increases in IL-6 (zeige IL6 Antikörper), IL-8 (zeige IL8 Antikörper) and VEGF expression in A549 and H292 cells.
Smad4 (zeige SMAD4 Antikörper) expression negatively correlated with VEGF-A and VEGF-C (zeige VEGFC Antikörper) in colon cancer
Mendelian randomization suggests that observed positive association of VEGF with ischemic heart disease is unlikely to be causal.
In primary hip OA, angiogenesis may be induced by a combined mechanism: hypoxia-related VEGF-dependent vasculogenesis and endothelial differentiation of the activated pluripotent cells, which are released from the hyperplastic synovial cells layer.
Lung cancer patients showed significantly lower levels of serum VEGF (1.9 fold) and IL-8 (zeige IL8 Antikörper) (~9 fold) than COPD (zeige ARCN1 Antikörper) patients. VEGF level was significantly higher (2.6 fold) in metastatic than non-metastatic cancer patients. An increase in MMP-9 (zeige MMP9 Antikörper) (~1.6 fold) levels was observed in lung cancer patients.
results demonstrated that mechanical stretch and CoCl2-induced HIF-1alpha (zeige HIF1A Antikörper) together increased the level of MMP-2 (zeige MMP2 Antikörper) and decreased the levels of VEGF and CTGF (zeige CTGF Antikörper) in cultured ACL (zeige ACLY Antikörper) fibroblasts.
Cyr61 (zeige CYR61 Antikörper) and VEGF expressions were independent prognostic indicators of overall survival and may serve as important prognostic predictors in patients with osteosarcoma.
Studied association of papillary thyroid carcinoma susceptibility and vascular endothelial growth factor (zeige VEGF Antikörper) single nucleotide polymorphisms.
miR (zeige MLXIP Antikörper)-9 modulation of neuronal VEGF-A controls brain angiogenesis in vivo.
The present data suggest that Ischemic preconditioning transiently increases plasma VEGF levels by downregulating miR (zeige MLXIP Antikörper)-762 and miR (zeige MLXIP Antikörper)-3072-5p in CD34 (zeige CD34 Antikörper)-positive BM cells, leading to protection against organ ischemia.
findings are the first to demonstrate the importance of CdGAP (zeige ARHGAP31 Antikörper) in embryonic vascular development and VEGF-induced signaling, and highlight CdGAP (zeige ARHGAP31 Antikörper) as a potential therapeutic target to treat pathological angiogenesis and vascular dysfunction
TGF-beta1 (zeige TGFB1 Antikörper)/TbetaRII/Smad3 (zeige SMAD3 Antikörper) signaling pathway increased VEGF expression in in oral squamous cell carcinoma tumor-associated macrophages (TAMs). TAMs can promote the tumor angiogenesis by secreting VEGF.
VEGF causes extensive neural stem cell (NSC) remodelling manifested in transition of the enigmatic NSC terminal arbor onto long cytoplasmic processes engaging with and spreading over even remote blood vessels, a configuration reminiscent of early postnatal "juvenile" NSCs.
effect of dox on VEGF-A levels might at least partly explain its previously reported beneficial effects on myocardial and brain ischemia. Also, this effect on VEGF-A should be taken into account in all studies using dox-regulated vectors
Genetic depletion experiments revealed that VEGFR2 (zeige KDR Antikörper), but not VEGFR3 (zeige FLT4 Antikörper), is indispensable for maintenance of thyroid vascular integrity. Notably, blockade of VEGF-A or VEGFR2 (zeige KDR Antikörper) not only abrogated vascular remodeling but also inhibited follicular hypertrophy, which led to the reduction of thyroid weights during goitrogenesis.
Findings suggest that VEGF gene expression can be suppressed by TNFSF15 (zeige TNFSF15 Antikörper)-stimulated activation of the JNK (zeige MAPK8 Antikörper)-GATA3 (zeige GATA3 Antikörper) signaling pathway which gives rise to up-regulation of miR (zeige MLXIP Antikörper)-29b.
The low-molecular-weight heparin (LMWH) Tinzaparin inhibited Von Willebrand factor (VWF (zeige VWF Antikörper)) fiber formation and vessel occlusion in tumor vessels by blocking thrombin (zeige F2 Antikörper)-induced endothelial cells (ECs) activation and vascular endothelial growth factor-A (VEGF-A)-mediated VWF (zeige VWF Antikörper) release.
These data suggest that VEGF expressed by skeletal myofibers may directly or indirectly regulate both hippocampal blood flow and neurogenesis.
Results show that apoE4-driven brain pathology and cognitive impairments in young apoE4 TR mice are associated with down regulation of the VEGF system and can be reversed by upregulation of the expression of VEGF in the hippocampus. These animal model findings suggest that the VEGF system is a promising target for the treatment of apoE4 carriers in Alzheimers disease.
It was concluded that VEGF and factor VIII are important growth factors associated with fetal development in pigs and are identified in all uterine segments.
results are consistent with a participation of (VEFG) in the regulation of the dynamics of oviductal fluid secretion and the oviduct contractibility
Here we demonstrate that VEGF-165 mediates MSC (zeige MSC Antikörper) differentiation into ECs via VEGFR-2 (zeige KDR Antikörper)-dependent induction of Sox18 (zeige SOX18 Antikörper), which ultimately coordinates the transcriptional upregulation of specific markers of the EC phenotype.
VEGF was significantly downregulated 7 days after cryotherapy of the sclera and stayed at that level until day 14. It returned to baseline by day 21.
VEGF production, blood vessel network and follicle remodeling are impaired by the antiprogesterone RU486.
findings suggest that the augmented neovascular response seen with VEGF administration was through the VEGF-induced upregulation of Notch (zeige NOTCH1 Antikörper) signaling.
interleukin-1beta-induced vascular endothelial growth factor (zeige VEGF Antikörper) in airway smooth muscle cells
Upregulation of VEGF during hypoxia in chondrocyte is mediated partially through HIF-1alpha (zeige HIF1A Antikörper).
data shows that members of the VEGF-VEGFR (zeige KDR Antikörper) system are temporally and spatially well localized for playing key roles during umbilical cord formation and its intensive growth observed after day 75 of pregnancy
cardiac-specific and hypoxia-induced coexpression of VEGF and Ang1 (zeige ANGPT1 Antikörper) improves the perfusion and function of porcine MI heart through the induction of angiogenesis and cardiomyocyte proliferation
Peripheral Blood-Derived Mesenchymal Stromal Cells Promote Angiogenesis via Paracrine Stimulation of Vascular Endothelial Growth Factor (zeige VEGF Antikörper) Secretion
TNF (zeige TNF Antikörper) may up-regulate VEGF and stimulate angiogenesis in the mare (zeige C16orf35 Antikörper) early corpus luteum.
After acoustic trauma, vascular endothelial growth factor (zeige VEGF Antikörper) was up-regulated both in the cochlea and vestibule. Expression in both structures suggests a reparative role with potentially therapeutic implications.
Studied the role of T help 17 cells (Th17) and STAT3 (zeige STAT3 Antikörper)-VEGF pathway in pathogenesis of psoriasis.
ghrelin (zeige GHRL Antikörper) can inhibit intraplaque angiogenesis and promote plaque stability by down-regulating VEGF and VEGFR2 (zeige KDR Antikörper) expression, inhibiting the plaque content of macrophages, and reducing MCP-1 (zeige CCL2 Antikörper) expression at an advanced stage of atherosclerosis in rabbits
VEGF mRNA abundance was present at low levels at 16 h post-ovulation and remained low at 72 h, but the level increased at 144 h in uterus.
correlation was observed between CT perfusion parameters (BF, BV, PS, and MAV (zeige KLHL2 Antikörper)) and expression of VEGF and MVD (zeige MVD Antikörper) in tumor tissue
Physiologic ischemic training stimulates VEGF-mediated mobilization of endothelial progenitor cells as well as angiogenesis.
Suggest supplemental oxygen inhibits HIF-1alpha (zeige HIF1A Antikörper)/VEGF signaling to reduce smooth muscle proliferation in rabbit arteriovenous fistula model.
Therefore, it was reasonable to speculate that the increased expression of VEGF and MMP-9 (zeige MMP9 Antikörper) in residual hepatic tumor cells and tumor angiogenesis post-embolization would be responsible for the increased metastatic potentiality and invasiveness.
VEGF expression peaked at 8 weeks after meniscus transplantation
Studied the biocompatibility and neovascularization of the PLGA nanospheres wrapped with vascular endothelial growth factor (VEGF (zeige VEGF Antikörper)).
VEGF induces TGF-beta1 (zeige TGFB1 Antikörper) expression and myofibroblast transformation after glaucoma surgery.
The results of this study are promising concerning the role of VEGF as a diagnostic marker in moderate osteoarthritis
Data suggest that microRNA-126 plays role in diabetic retinopathy; here, choroid-retinal endothelial cells exposed to high glucose exhibit down-regulation of microRNA-126 and up-regulation of VEGFA and PIK3R2; overexpression of microRNA-126 down-regulates expression of VEGFA and PIK3R2. (VEGFA = vascular endothelial growth factor A; PIK3R2 = class Ia phosphoinositol-3 kinase regulatory subunit 2 (zeige CCT2 Antikörper))
VEGF plays an important role in choroid-retinal endothelial cell proliferation and tube formation.
These findings suggest that FBLN5 (zeige FBLN5 Antikörper) may interfere with choroidal neovascularization by downregulating VEGF, CXCR4 (zeige CXCR4 Antikörper), and TGFB1 (zeige TGFB1 Antikörper) expression and inhibiting choroidl endothelial cell proliferation.
Apelin (zeige APLN Antikörper) may play a role in the development of central retinal vein occlusion (CRVO). Apelin (zeige APLN Antikörper) has a unique upstream signaling pathway, independent of the VEGF pathway.
Dll4 (zeige DLL4 Antikörper) play an important role in choroidal neovascularization (CNV) angiogenesis, which appears to be regulated by HIF-1alpha (zeige HIF1A Antikörper) and VEGF during the progression of CNV under hypoxic conditions.
A functional network involving VEGF, IGF2, and MMP9 (zeige MMP9 Antikörper) in early placental trophoblast cells and maternal endometrium appears to be important for normal placentation.
This gene is a member of the PDGF/VEGF growth factor family and encodes a protein that is often found as a disulfide linked homodimer. This protein is a glycosylated mitogen that specifically acts on endothelial cells and has various effects, including mediating increased vascular permeability, inducing angiogenesis, vasculogenesis and endothelial cell growth, promoting cell migration, and inhibiting apoptosis. Elevated levels of this protein is linked to POEMS syndrome, also known as Crow-Fukase syndrome. Mutations in this gene have been associated with proliferative and nonproliferative diabetic retinopathy. Alternatively spliced transcript variants, encoding either freely secreted or cell-associated isoforms, have been characterized. There is also evidence for the use of non-AUG (CUG) translation initiation sites upstream of, and in-frame with the first AUG, leading to additional isoforms.
, vascular endothelial growth factor A
, vascular endothelial growth factor A-A
, vascular endothelial growth factor A-a
, vascular endothelial growth factor 164
, vascular permeability factor
, vascular endothelial growth factor 188
, angiogenic factor
, Vascular permeability factor
, endothelial growth factor-164
, vascular endothelial growth factor VEGF