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myocardin is an essential component of the regulatory pathway for myocardial differentiation
several transcription factors, that is SRF, myocardin, and GATA6, that induce the expression of SM-MHC in animal cap cells
YAP (zeige YAP1 Antikörper) negatively regulates differentiation of Vascular smooth muscle cells (VSMCs) derived from cardiovascular progenitor cell (CVPC) by decreasing transcription of myocardin in a NKX2.5 (zeige NKX2-5 Antikörper)-dependent manner.
Myocardin inhibits estrogen receptor alpha (zeige ESR1 Antikörper)-mediated proliferation of human breast cancer cells via regulating MicroRNA expression.
Key role for miR (zeige MLXIP Antikörper)-214 in modulation of MEF2C (zeige MEF2C Antikörper)-MYOCD-LMOD1 (zeige LMOD1 Antikörper) signaling.
Study demonstrates that myocardin indirectly down-regulates Cx43 (zeige GJA1 Antikörper) through its repressive action on miR (zeige MLXIP Antikörper)-206 to regulate vascular smooth muscle cell phenotypic switch.
These results have revealed the roles for atrogin-1 (zeige FBXO32 Antikörper) in the regulation of smooth muscle contractility through enhancement of myocardin ubiquitylation/degradation and its transcriptional activity.
our study reveals that Ang II (zeige AGT Antikörper) downregulates miR (zeige MLXIP Antikörper)-145 to regulate Klf4 (zeige KLF4 Antikörper) and myocardin expression in HCASMCs under high glucose conditions. Ang II (zeige AGT Antikörper) plays a critical role in the regulation of miR (zeige MLXIP Antikörper)-145 under hyperglycemic conditions
Human expression data disclosed correlations of MYOCD with CAV1 in a majority of human tissues and in the heart, correlation with MKL2 (MRTF-B) was observed.
inhibition of GSK-3beta reduces myocardin transcriptional activity, suggesting a role for GSK-3beta in myocardin transcriptional activity and smooth muscle differentiation
STAT3 protein regulates vascular smooth muscle cell phenotypic switch by interaction with myocardin and SRF.
TMEM16A (zeige ANO1 Antikörper) and myocardin form a positive feedback loop that is disrupted by KLF5 (zeige KLF5 Antikörper) during Ang II (zeige AGT Antikörper)-induced vascular remodeling.
Long noncoding RNAs cardiac autophagy inhibitory factor (lncRNA CAIF) regulates autophagy through controlling p53 (zeige TP53 Antikörper) and myocardin.
TAZ (zeige TAZ Antikörper) and MyoC856 physically interact. SynergyTAZ and Myocardin (MyoC856), in regulating smooth muscle gene activation was observed in primary aortic vascular smooth muscle cells.
two MEF2 (zeige MEF2C Antikörper) sites in the enhancer function cooperatively due to bridging of the MEF2C (zeige MEF2C Antikörper)-bound sites by the SAP (zeige APCS Antikörper) domain-containing co-activator protein myocardin
c-Myb (zeige MYB Antikörper) regulates proliferation/differentiation of adventitial Sca1 (zeige ATXN1 Antikörper)+ vascular smooth muscle progenitor cells by transcriptional activation of myocardin.
Knock down of the serum response factor (SRF), which mediates many of the effects of myocardin, decreased cavin-1 but increased caveolin-1 and -2 mRNAs.
Cholesterol loading of vascular smooth muscle cells converts them to a macrophage-appearing state by downregulating the miR (zeige MLXIP Antikörper)-143/145-myocardin axis.
Our data illustrate a novel mechanism that connects MRTF-A (zeige MKL1 Antikörper) dependent histone H3K4 methylation to HSC (zeige FUT1 Antikörper) activation.
DKK3 (zeige DKK3 Antikörper) induces stem cell differentiation into smooth muscle lineage via ATF6 (zeige ATF6 Antikörper) signaling and myocardin expression.
a moderate inhibition (e.g., normalization) of the activated MYOCD signaling in the diseased heart may be promising from a therapeutic point of view
MYOCD DeltaExon 11 may participate in modulating smooth muscle cell phenotype, potentially acting as a dominant-negative repressor of contraction-related genes.
Data show that forced myocd-A expression in the LVFW caused abnormal ECG.
Thrombin (zeige F2 Antikörper) stimulates swine smooth muscle cell differentiation from peripheral blood mononuclear cells via protease-activated receptor-1 (zeige F2R Antikörper), RhoA (zeige RHOA Antikörper), and myocardin.
This gene encodes a nuclear protein, which is expressed in heart, aorta, and in smooth muscle cell-containing tissues. It functions as a transcriptional co-activator of serum response factor (SRF) and modulates expression of cardiac and smooth muscle-specific SRF-target genes, and thus may play a crucial role in cardiogenesis and differentiation of the smooth muscle cell lineage. Alternatively spliced transcript variants encoding different isoforms have been found for this gene.
, SRF co-factor protein (cardiac and smooth muscle)
, SRF cofactor protein
, basic SAP coiled-coil transcription activator 2
, transcription factor myocardin