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Human Polyclonal BCLAF1 Primary Antibody für ICC, IF - ABIN151225
Liu, Lu, Miki, Yoshida: Protein kinase C delta induces transcription of the TP53 tumor suppressor gene by controlling death-promoting factor Btf in the apoptotic response to DNA damage. in Molecular and cellular biology 2007
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Constitutive expression of the anti-apoptotic molecule Bfl1 (zeige BCL2A1 Antikörper) (A1) in murine vascular Endothelial Cells leads to prolonged allograft survival due to modifying inflammation.
Bclaf1 facilitates the differentiation of early retinal precursors into retinal ganglion cells, amacrine cells, and horizontal cells rather than into cone photoreceptors.
Sirt1 (zeige SIRT1 Antikörper) negatively regulates T cell activation via H3K56 deacetylation at the promoter region to inhibit transcription of Bclaf1
Bclaf1 expression was found to be strongly upregulated during the saccular stage of murine embryonic lung development and was dispensable for thymocyte development.
LTBR (zeige LTBR Antikörper) and BCLAF1 showed higher DNA methylation (zeige HELLS Antikörper) percentages in the marsupialized OKCs, but this difference did not affect gene expression (P > .05).
Results indicate a role for the RNA processing factors THRAP3 (zeige THRAP3 Antikörper) and BCLAF1 in the regulation of the cellular DNA damage response (DDR (zeige DDR1 Antikörper)) pathway.
findings link treatment responsiveness to re-instatement of miR (zeige MLXIP Antikörper)-194-5p/BCLAF1 balance
SMYD3 (zeige SMYD3 Antikörper) physically interacts with the promoter of BCLAF1 and upregulates its expression by accumulating di- and trimethylation of H3K4 at the BCLAF1 locus. BCLAF1 depletion inhibits SMYD3 (zeige SMYD3 Antikörper)-induced autophagy.
The Bclaf1 can interact with the leucine zipper region of C/EBPbeta (zeige CEBPB Antikörper) and cooperate with C/EBPbeta (zeige CEBPB Antikörper) to upregulate IL-8 (zeige IL8 Antikörper).
both cytoplasmic BCLAF1 expression and nuclear BCLAF1 expression are increased in post-neoadjuvant therapy rectal cancer, and that negative and weak nuclear BCLAF1 expression are independently associated with a poor prognosis
SRSF10 (zeige SRSF10 Antikörper) is a key regulator of BCLAF1 pre-mRNA splicing and the maintenance of oncogenic features in human colon cancer cells
findings showed that FXR1P (zeige FXR1 Antikörper) interacts with BTF in vivo and proved that FXR1P (zeige FXR1 Antikörper) and BTF can co-localize mainly in the cytoplasm around the nucleus
BTF has functions distinct from TRAP150 (zeige THRAP3 Antikörper) in regulating the subcellular distribution of mRNAs in human cells.
BCLAF1 co-localized with gammaH2AX (zeige H2AFX Antikörper) foci in nuclei and stabilized the Ku70 (zeige XRCC6 Antikörper)/DNA-PKcs (zeige PRKDC Antikörper) complex therein, facilitating non-homologous end joining (NHEJ)-based DSB repair in surviving cells.
This gene encodes a transcriptional repressor that interacts with several members of the BCL2 family of proteins. Overexpression of this protein induces apoptosis, which can be suppressed by co-expression of BCL2 proteins. The protein localizes to dot-like structures throughout the nucleus, and redistributes to a zone near the nuclear envelope in cells undergoing apoptosis. Multiple transcript variants encoding different isoforms have been found for this gene.
bcl-2-associated transcription factor 1
, mitochondrial fission regulator 2
, protein FAM54A
, BCL2-associated transcription factor 1