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anti-Human FOXQ1 Antikörper:
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Human Polyclonal FOXQ1 Primary Antibody für ELISA, WB - ABIN547121
Samatar, Wang, Mirza, Koseoglu, Liu, Kumar: Transforming growth factor-beta 2 is a transcriptional target for Akt/protein kinase B via forkhead transcription factor. in The Journal of biological chemistry 2002
This study provides the first genetic mutant analyses of zebrafish foxq1a and foxq1b. Foxq1a, but not foxq1b, was transcriptionally regulated during a bacterial response, while the expression of foxq1a was detected in sorted macrophages and upregulated in foxq1a-deficient mutants.
inhibition of FOXQ1 restricted natural killer/T-cell lymphoma cell proliferation and growth but induced apoptosis
Findings indicate a positive feedback loop between cancer associated fibroblast (CAFs) and box Q1 (FOXQ1)/N-myc downstream-regulated gene 1 (NDRG1) axis in neoplastic cells to drive hepatocellular carcinoma (HCC) initiation, suggesting new potential therapeutic targets for HCC.
Results show that FOXQ1, a well-known oncogenic protein and promoter of gastric cancer (GC) metastasis, to be the direct downstream target of miR-345 in GC. Its mRNA and protein levels are up-regulated in GC tumors.
These data identify FOXQ1 as a melanoma suppressor.
Results revealed that, co-culture with TAMs promoted the invasion and migration of GC cells. Co-culture with TAMs induced EMT in GC cells. FOXQ1 is essential for TAM-induced EMT and metastasis in GC cells.
FOXQ1 remarkably inhibited the replicative senescence through depressing the expression of the inflammatory cytokines interleukin-6 (IL-6) and IL-8 via modulation of SIRT1-NF-kappaB pathway.
Cell viability and progression from G1 to S phase were both improved with overexpression of Foxq1, and microRNAs profiling study and dual-luciferase result suggested miR-320b contributed to the up-regulation of Foxq1 after calcium hydroxide stimulation. These results suggested that miR-320b mediated Foxq1 up-regulation promote proliferation of dental pulp stem cells.
We identified FOXM1 and FOXQ1 as novel prognostic biomarkers in colorectal cancer and miR-342 as a novel regulator of both FOXM1 and FOXQ1
Data indicate a role for the miR-320/SOX4/FOXM1/FOXQ1 axes in promoting colorectal cancer (CRC) development and suggest targeting those networks as potential therapeutic strategy for CRC.
MiR-133 directly targeted and down-regulated FOXQ1 expression, which in turn reduced TGF-beta level.
There are several modes of regulation of FOXQ1 expression that have been demonstrated in normal and tumor cells, such as microRNA and the Wnt signaling pathway. The activation of FOXQ1 affects downstream genes promoting the initiation, proliferation and invasion, in addition to the metastasis of tumor cells
FOXQ1 is a prognostic marker for patients with Gastric cancer, FOXQ1 over-expression is involved in acquisition of the mesenchymal phenotype of gastric cancer cells, and subsequent Snail expression is essential for induction of Epithelial-Mesenchymal Transition.
our study suggests that FOXQ1 regulates prostate cancer cell proliferation and apoptosis by regulating BCL11A/MDM2 expression and indicates that FOXQ1 may serve as a potential therapeutic target for prostate cancer.
The miR-506/FOXQ1 axis plays an important role in the pathogenesis of cervical cancer.
DATS administration inhibited ALDH1 activity in vivo in SUM159 xenografts. These results indicate that FoxQ1 is a novel target of bCSC inhibition by DATS.
Data suggest that forkhead box Q1 (FOXQ1) is a potential therapeutic target for the development of therapies for colorectal cancer.
we identified FOXQ1 as an oncogene to promote ESCC tumor cell proliferation and metastasis by negatively regulating CDH1 in esophageal squamous cell carcinoma cells
MiR-1271 inhibits cell proliferation, invasion, and epithelial-mesenchymal transition in gastric cancer by directly suppressing FOXQ1 expression.
Results demonstrate that miR-124 functions as a tumor-suppressive microRNA in nasopharyngeal carcinoma by repressing Foxq1 expression.
FoxQ1 expression is negatively associated with the overall survival of PC patients, and that this protein may therefore represent a novel molecular target and new prognostic biomarker for PC.
the Fox transcription factor binding motif was frequently observed within EWS-FLI1 peaks and Foxq1 was identified as the cooperative partner that interacts with the EWS portion of EWS-FLI1.
FOXQ1 is a novel factor involved in regulating hepatic gluconeogenesis, and the decreased FOXQ1 expression in liver may contribute to the development of type 2 diabetes.
R164C mutation in FOXQ1 H3 domain affects formation of the hair medulla in mice.
This study has elucidated the functional impact of FoxQ1 on epithelial differentiation.
satin hair mutant gene Foxq1 is among multiple and functionally diverse regulatory targets for Hoxc13 during hair follicle differentiation
Ultrastructural analysis suggests that the gastric acid secretion defect in Foxq1-deficient mice might be due to impairment in the fusion of cytoplasmic tubulovesicles to the apical membrane of secretory canaliculi.
Transcription factor foxq1 controls MUC5AC gene expression and granule content in mouse stomach surface mucous cells.
FOXQ1 is a member of the FOX gene family, which is characterized by a conserved 110-amino acid DNA-binding motif called the forkhead or winged helix domain. FOX genes are involved in embryonic development, cell cycle regulation, tissue-specific gene expression, cell signaling, and tumorigenesis (Bieller et al., 2001
forkhead box Q1
, forkhead transcription factor Q1
, fork-head box Q1 transcription factor
, HNF-3/forkhead-like protein 1
, forkhead box protein Q1
, hepatocyte nuclear factor 3 forkhead homolog 1
, winged helix/forkhead transcription factor
, HNF-3/forkhead homolog 1 like
, HNF-3/forkhead homolog-1