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anti-Mouse (Murine) SERPING1 Antikörper:
anti-Human SERPING1 Antikörper:
anti-Rat (Rattus) SERPING1 Antikörper:
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Cow (Bovine) Polyclonal SERPING1 Primary Antibody für IHC (p), IHC - ABIN442446
Hayashi, Ushizawa, Hosoe, Takahashi: Differential gene expression of serine protease inhibitors in bovine ovarian follicle: possible involvement in follicular growth and atresia. in Reproductive biology and endocrinology : RB&E 2011
Studied the association of postmortem pH with SNPs in the fourth member of the short-chain dehydrogenase/reductase family (DHRS4), the first member of serpin peptidase inhibitor, clade G (complement inhibitor) (SERPING1), and the apolipoprotein R precursor (APOR) genes in Berkshire pigs. Results suggest that DHRS4, SERPING1, and APOR snps may be potential genetic markers for meat quality.
C1INH prevented myocardial ischemia reperfusion injury in mice.
Increased vascular permeability in C1 inhibitor-deficient mice mediated by the bradykinin type 2 receptor.
Transgenic mice have been generated that express human C1 inhibitor mRNA and protein under the control of the human promoter and regulatory elements; C1nh production is demonstrated in mouse brain, spleen, liver, heart, kidney, and lung.
In vivo, C1 inhibitor (C1INH) reduces the number of viable bacteria in the blood and peritoneal fluid and accelerates killing of bacteria by blood neutrophils and peritoneal macrophages.
C1-inhibitor limits neointimal plaque formation and inflammation. This may involve blockade of complement activation, inhibition of leukocyte recruitment, and reduced triglyceride levels
In addition to the protective activities mediated via inhibition of the complement system, these studies indicate that C1INH also plays a direct role in suppression of leukocyte transmigration into reperfused tissue.
Study identified that peripheral mRNA expression levels of two complement genes (C5, SERPING1) made unique contributions to the variance in superior frontal cortical thickness. Vertex-wise maps of the association between gene expression levels and thickness across the cortex suggested that this relationship was especially strong with SERPING1 in the superior frontal region.
Our analysis shows that SERPING1 mRNA is overexpressed in monocytes from HIV-1+ patients and the expression levels correlate positively with viral load and negatively with the CD4 (zeige CD4 Antikörper)(+) T-cell count.
A rare non-conservative missense mutation was newly identified in exon 9 of the PLG (zeige PLG Antikörper) gene.
plasma-derived C1 esterase inhibitor concentrate has limited effect on house dust mite-induced allergic lung inflammation in mice
Data suggest that hereditary angioedema (HAE) may be caused by the deficiency of complement component 1 inhibitor protein (C1 (zeige PFDN4 Antikörper)-inhibitor; SERPING1).
findings show that P. falciparum merozoites recruit C1-INH from human serum through an interaction with one of the merozoite surface proteins
PIC1 (zeige SUMO1 Antikörper) inhibits the peroxidase activity of myeloperoxidase (zeige MPO Antikörper) in cystic fibrosis (zeige S100A8 Antikörper) sputum likely via an antioxidant mechanism.
like heparin, polyP is a naturally occurring cofactor for the C1s:C1-INH interaction and thus an important regulator of complement activation
Hereditary angioedema with C1 inhibitor deficiency (C1-INH-HAE) is a rare autosomal dominant disease caused by mutations in the C1 inhibitor gene SERPING1. In the present study, the mutational spectrum of the SERPING1 gene in 19 patients of nine unrelated Swiss families were investigated.
Results identified a novel causative mutation in SERPING1, a deletion of two nucleotides on exon 3 in several affected members of two apparently unrelated families with a high frequency of hereditary angioedema.
This gene encodes a highly glycosylated plasma protein involved in the regulation of the complement cascade. Its protein inhibits activated C1r and C1s of the first complement component and thus regulates complement activation. Deficiency of this protein is associated with hereditary angioneurotic oedema (HANE). Alternative splicing results in multiple transcript variants encoding the same isoform.
, c1 Inh
, plasma protease C1 inhibitor
, serine (or cysteine) peptidase inhibitor, clade G, member 1
, serpin peptidase inhibitor, clade G (C1 inhibitor), member 1, (angioedema, hereditary)
, serpin peptidase inhibitor, clade G (C1 inhibitor), member 1
, plasma protease C1 inhibitor-like
, C1 Inh
, C1 esterase inhibitor
, C1-inhibiting factor
, complement component 1 inhibitor
, serine (or cysteine) proteinase inhibitor, clade G (C1 inhibitor), member 1
, serine (or cysteine) proteinase inhibitor, clade G, member 1
, serpin G1
, serine/cysteine proteinase inhibitor clade G member 1
, serine (or cysteine) proteinase inhibitor, clade G (C1 inhibitor), member 1, (angioedema, hereditary)
, serpin peptidase inhibitor, clade G, member 1
, complement component 1 inhibitor (angioedema, hereditary)
, factor XIIa inhibitor