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HSV-1 IgM ELISA Kit

HSV1 IgM Reaktivität: Human Colorimetric Competition ELISA Serum
Produktnummer ABIN1326851
  • Target Alle HSV-1 IgM (HSV1 IgM) Produkte
    HSV-1 IgM (HSV1 IgM) (Anti-Herpes Simplex Virus Type 1 IgM (HSV1 IgM))
    Reaktivität
    • 3
    • 1
    Human
    Nachweismethode
    Colorimetric
    Methodentyp
    Competition ELISA
    Applikation
    ELISA
    Verwendungszweck
    Diluted patient serum (serum diluent contains sorbent to remove Rheumatoid Factor and human IgG interference) is added to wells coated with purified antigen. IgM specific antibody, if present, binds to the antigen. All unbound materials are washed away and the enzyme conjugate is added to bind to the antibody-antigen complex, if present. Excess enzyme conjugate is washed off and substrate is added. The plate is incubated to allow the hydrolysis of the substrate by the enzyme. The intensity of the color generated is proportional to the amount of IgM specific antibody in the sample.
    Proben
    Serum
    Analytische Methode
    Qualitative
  • Plattentyp
    Pre-coated
    Beschränkungen
    Nur für Forschungszwecke einsetzbar
  • Lagerung
    4 °C
  • Target Alle HSV-1 IgM (HSV1 IgM) Produkte
    HSV-1 IgM (HSV1 IgM) (Anti-Herpes Simplex Virus Type 1 IgM (HSV1 IgM))
    Andere Bezeichnung
    HSV-1 IgM (HSV1 IgM Produkte)
    Substanzklasse
    Antibody, Antibody
    Hintergrund
    HSV-1 and 2 are virtually identical, sharing approximately 50% of their DNA and have over 80% of common antigens. Both types infect the body's mucosal surfaces, usually the mouth or genitals, and then establish latency in the nervous system. For both types, at least two-thirds of infected people have no symptoms, or symptoms too mild to notice. However, both types can recur and spread even when no symptoms are present. By the time they're teenagers or young adults, about 50% of Americans have HSV-1 antibodies in their blood. By the time they are over age 50, some 80-90% of Americans has HSV-1 antibodies. By comparison, almost all HSV-2 is encountered after childhood, when people become sexually active. HSV type 1 is the cause of most orofacial herpes and HSV encephalitis type 2 is the primary cause of initial and recurrent genital herpes and neonatal HSV. Reactivation of latent HSV infection is a frequent complication of immunosuppression due to cancer, transplantation and AIDS. Asymptomatic genital shedding of HSV-2 is more common than HSV-1 and occurs more frequently during the first 3 months after acquisition of primary type 2 disease than during later periods. The presence of HSV IgG antibody is indicative of previous exposure A significant increases in HSV IgG is an indicative of reactivation, current or recent infection. IgM antibody is present after primary HSV infection.
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