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anti-Human CSF2 Antikörper:
anti-Mouse (Murine) CSF2 Antikörper:
anti-Rat (Rattus) CSF2 Antikörper:
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Human Monoclonal CSF2 Primary Antibody für CyTOF, ELISA (Capture) - ABIN4899319
Hirst, Hallsworth, Peng, Lee et al.: Selective induction of eotaxin release by interleukin-13 or interleukin-4 in human airway smooth muscle cells is synergistic with interleukin-1beta and is mediated by the interleukin-4 receptor ... in American journal of respiratory and critical care medicine 2002
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Human Monoclonal CSF2 Primary Antibody für ELISA, WB - ABIN1724740
He, Shumansky, Connett, Anthonisen, Paré, Sandford: Association of genetic variations in the CSF2 and CSF3 genes with lung function in smoking-induced COPD. in The European respiratory journal 2008
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Human Monoclonal CSF2 Primary Antibody für FACS - ABIN4895921
Alisa, Boswell, Pathan, Ayaru, Williams, Behboudi: Human CD4(+) T cells recognize an epitope within alpha-fetoprotein sequence and develop into TGF-beta-producing CD4(+) T cells. in Journal of immunology (Baltimore, Md. : 1950) 2008
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Rat (Rattus) Monoclonal CSF2 Primary Antibody für CyTOF, FACS - ABIN4898951
Stojić-Vukanić, Nacka-Aleksić, Pilipović, Vujnović, Blagojević, Kosec, Dimitrijević, Leposavić: Aging diminishes the resistance of AO rats to EAE: putative role of enhanced generation of GM-CSF Expressing CD4+ T cells in aged rats. in Immunity & ageing : I & A 2015
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Human Polyclonal CSF2 Primary Antibody für FACS, IF - ABIN654647
Stone, Vanderman, Willey, Long, Register, Shively, Stehle, Loeser, Ferguson: Osteoarthritic changes in vervet monkey knees correlate with meniscus degradation and increased matrix metalloproteinase and cytokine secretion. in Osteoarthritis and cartilage / OARS, Osteoarthritis Research Society 2015
Data show that CD4 (zeige CD4 Antikörper) T cells from synovial fluid are enriched for granulocyte-macrophage colony-stimulating factor (GM-CSF) production, suggests that the GM-CSF programme may be a primary pathogenic process.
GM-CSF bioavailability plays a critical role in maintaining intestinal homeostasis. Decreased bioavailability coupled with the genetic risk markers and/or smoking results in aggressive ileal Crohn's disease behavior.
Data show that GIF (zeige GIF Antikörper) (ORF117 ) serves as a competitive decoy receptor by leveraging binding hotspots underlying the cognate receptor interactions of granulocyte macrophage colony-stimulating factor (GM-CSF) and interleukin-2 (IL-2 (zeige IL2 Antikörper)).
As translated to human disease, these findings suggest vaccine-mediated expansion of GM-CSF-producing T cells could be an effective prophylactic or therapeutic TB strategy.
In a subset of patients who received GM-CSF, circulating myeloid-derived suppressor cells (MDSC), and anti-GM-CSF-neutralizing antibodies (Nabs) were also modulated. The majority of patients developed anti-GM-CSF Nabs, which correlated with improved RFS and OS
this study shows that cortisol inhibits CSF2 via DNA methylation (zeige HELLS Antikörper) and inhibits invasion in first-trimester trophoblast cells
Upregulation of GM-CSF and M-CSF (zeige CSF1 Antikörper) production by endothelial cells, an effect that appears to be mediated by NF-kappaB (zeige NFKB1 Antikörper) and to be independent of IL-1 (zeige IL1A Antikörper), may be an additional mechanism through which IL-33 (zeige IL33 Antikörper) contributes to inflammatory activation of the vessel wall.
Oral and periodontal innate immunity is affected by HIV viremia and ART. GCF (zeige GUCY2F Antikörper) IL-8 (zeige IL8 Antikörper), G-CSF (zeige CSF3 Antikörper), as well as serum IL-8 (zeige IL8 Antikörper), MCP-1 (zeige CCL2 Antikörper) and GM-CSF may be useful biomarkers for the detection of disease presence and/or its severity due to HIV infection and ART use.
Data suggest that the intratumoral GM-CSF expression, as a potentially independent prognostic biomarker for recurrence, might improve conventional clinical and pathologic analysis to refine outcome prediction for clinically localized clear-cell renal cell carcinoma (zeige MOK Antikörper) (ccRCC) patients after surgery.
High GM-CSF expression is associated with breast Cancer.
TRAF6 (zeige TRAF6 Antikörper) is also required for GM-CSF-induced ubiquitination and activation of Akt (zeige AKT1 Antikörper).
CSF2 stimulates proliferation of trophectoderm cells by activation of the PI3K-and ERK1/2 MAPK (zeige MAPK1 Antikörper)-dependent MTOR (zeige FRAP1 Antikörper) signal transduction cascades.
Data show that the microbiota enhances respiratory defenses via granulocyte-macrophage colony-stimulating factor (GM-CSF) signaling, which stimulates pathogen killing and clearance by alveolar macrophages
The data indicate that GM-CSF drives chronic tissue damage and disability in experimental autoimmune encephalomyelitis via pleiotropic pathways, but it is dispensable during early lesion formation and the onset of neurologic deficits.
the release of IL-33 (zeige IL33 Antikörper) and GM-CSF from epithelial cells induces the activation of p65 (zeige NFkBP65 Antikörper) and the p38 (zeige CRK Antikörper)-MK2 (zeige KCNA2 Antikörper)/3 signaling module in Dendritic Cells, resulting in Th2 polarization and, finally, allergic inflammation.
results show T cell production of GM-CSF contributes to control of M. tuberculosis infection in the absence of other sources of GM-CSF, that multiple T cell subsets make GM-CSF in the lung over the course of infection and that GM-CSF can act directly on infected macrophages through a pathway requiring PPARgamma (zeige PPARG Antikörper) to limit bacterial growth
In conclusion, our study confirms the pathogenic role of GM-CSF in colitis-associated colorectal cancer development. GM-CSF favors tumor-permissive microenvironment by inducing MDSC generation and recruiting them into colonic tissues.
these data demonstrate that GM-CSF levels during radiotherapy can be used as a prognostic biomarker for lung and esophageal cancer
this study demonstrates that epithelial-derived GM-CSF is a critical early signal during allergic sensitization to cockroach allergen
These impaired macrophage functions in leukemic mice were significantly corrected by IL-3 (zeige IL-3 Antikörper) and GM-CSF treatment indicating the therapeutic benefit of these two cytokines in leukemia.
Both IL-6 (zeige IL6 Antikörper) protein production and transcript levels were downregulated by RA in respiratory tract epithelial cells (LETs) , but upregulated in macrophages (MACs). RA also increased transcript levels of MCP-1 (zeige CPT1B Antikörper), GMCSF, and IL-10 (zeige IL10 Antikörper) in MACs, but not in LETs. Conversely, when LETs, but not MACs, were exposed to RA
T-GM-CSF and -IL-3 (zeige IL-3 Antikörper) significantly, and reciprocally, blunted receptor binding and myeloid progenitor cell proliferation activity of both FL-GM-CSF and -IL-3 (zeige IL-3 Antikörper) in vitro and in vivo
It was concluded that CSF2 can act as a developmental programming agent but alone is not able to abolish the adverse effects of in vitro production on fetal characteristics.
These results demonstrated that bovine colonic cells seem capable to respond to E. bovis merozoite I infection by the upregulation of CXCL10 (zeige CXCL10 Antikörper) and GM-CSF gene transcription.
Data suggest exposure to maternal CSF2 from D5-D7 of development is fundamentally different for female/male blastocysts with respect to embryo elongation, characteristics of transcriptome/methylome, and endometrial interferon tau secretion at D15 (zeige MRPL16 Antikörper).
The increase in calving rate caused by CSF2 treatment involves, in part, more extensive development of extraembryonic membranes and capacity of the conceptus to secrete IFNT2 at day 15 of pregnancy.
immunolocalization studies confirmed the presence of granulocyte-macrophage colony stimulating factor(GM-CSF) in the germ cell line in bovine and human testes and addition of GM-CSF enhances several parameters of sperm motility
the conceptus, through its secretion of IFN-tau, stimulates maternal epithelial expression of COX-2 (zeige PTGS2 Antikörper) and GM-CSF during the peri (zeige PLIN1 Antikörper)-attachment period in the cow.
CSF2 affect embryonic development and enhance embryo competence for posttransfer survival.
Data indicate that differentially expressed IL-17 (zeige IL17A Antikörper), IL-22 (zeige IL22 Antikörper), and IL-23 (zeige IL23A Antikörper) levels are associated with K-ras (zeige HRAS Antikörper) in a stage-specific fashion along colorectal cancer progression and an association was established between mutant K-ras (zeige HRAS Antikörper) and GM-CSF and IFN-gamma (zeige IFNG Antikörper).
The protein encoded by this gene is a cytokine that controls the production, differentiation, and function of granulocytes and macrophages. The active form of the protein is found extracellularly as a homodimer. This gene has been localized to a cluster of related genes at chromosome region 5q31, which is known to be associated with interstitial deletions in the 5q- syndrome and acute myelogenous leukemia. Other genes in the cluster include those encoding interleukins 4, 5, and 13.
, granulocyte-macrophage colony-stimulating factor
, colony stimulating factor 2 (granulocyte-macrophage)
, colony-stimulating factor
, granulocyte-macrophage colony stimulating factor 2
, put. GM-CSF
, granulate-macrophage stimulating factor
, granulocyte-macrophage stimulation factor