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Angiotensin II has a role in increasing glomerular permeability by beta-arrestin mediated nephrin (zeige NPHS1 Proteine) endocytosis
After donor nephrectomy, increasing uAGT (zeige DPAGT1 Proteine) levels can be the result of activation of the intrarenal renin (zeige REN Proteine)-angiotensin system affecting the compensatory changes in the remaining kidney.
M235T polymorphism of the AGT (zeige AGXT Proteine) gene seems unrelated to the development or the clinical course of endometriosis.
AGT (zeige AGXT Proteine) missense polymorphisms are not associated with diabetic nephropathy in our subset of Slovenian type 2 diabetes mellitus patients
Association of AGT (zeige AGXT Proteine) single nucleotide polymorphism rs3789678 and gestational hypertension in Chinese population.
The T allele of AGT (zeige AGXT Proteine) may play a role in the pathogenesis of preeclampsia in South African Black women.
These data indicate that Ang II (zeige AGT Proteine)-AT2R (zeige AGTR2 Proteine) regulates human bone marrow MSC (zeige MSC Proteine) migration by signaling through the FAK (zeige PTK2 Proteine) and RhoA (zeige RHOA Proteine)/Cdc42 (zeige CDC42 Proteine) pathways.
Data suggest that up-regulaton of Ang-(1 (zeige ANGPT1 Proteine)-7) levels in follicular fluid correlates with increases in number of mature oocytes retrieved upon ovarian stimulation in preparation for in vitro fertilization.
Urinary angiotensinogen (zeige AGT Proteine) and renin (zeige REN Proteine) excretion are elevated in CKD patients. Both parameters are negatively associated with eGFR (zeige EGFR Proteine) and these associations are independent of urinary albumin (zeige ALB Proteine) excretion
Reduced IL-18 (zeige IL18 Proteine) serum concentration in children after HUS (zeige CFH Proteine) with no difference in its urine concentration may indicate a loss of the protective effects of this cytokine on renal function due to previously occurred HUS (zeige CFH Proteine).
ANG II is up-regulated in serum and heart tissues of mice with EAM and that ANG II significantly drives monocyte/macrophage infiltration through the C-C chemokine receptor 2/5 (CCR2/5) axis.
results established that A20 (zeige TNFAIP3 Proteine) is involved in the renoprotective effect by calcitriol via negatively modulating the NF-kappaB (zeige NFKB1 Proteine) pathway and necroptotic pathway in AngII-induced renal injury.
NLRP3 (zeige NLRP3 Proteine) gene deletion attenuates Ang II (zeige AGT Proteine)-induced NLRP3 (zeige NLRP3 Proteine) inflammasome activation, phenotypic transformation from a contractile phenotype to a synthetic phenotype and proliferation in primary mice Vascular Smooth Muscle Cells.
adipocyte-derived Agt (zeige AGXT Proteine) has essentially no contribution to the plasma concentration and no impact on blood pressure compared to liver-derived Agt (zeige AGXT Proteine).
Lung ischemia-reperfusion injury causes a dysregulation of circulating Ang 2 (zeige ANGPT2 Proteine) levels and plasma PREP (zeige PREP Proteine) activity, although no direct link between both phenomena could be shown.
Inhibition of TLR4 (zeige TLR4 Proteine) ameliorates AngII-impaired cavernosal relaxation, decreases TNF-alpha (zeige TNF Proteine) levels, and restores Nitric Oxide bioavailability, demonstrating that TLR4 (zeige TLR4 Proteine) partly mediates AngII-induced cavernosal dysfunction.
Our study is the first to show the important role of IL-6 (zeige IL6 Proteine) in regulating cardiac pathogenesis via inflammation and apoptosis during AngII-induced hypertension. We also provide a novel link between IL-6 (zeige IL6 Proteine)/STAT3 (zeige STAT3 Proteine) and EndoG (zeige ENDOG Proteine)/MEF2A (zeige MEF2A Proteine) pathway that affects cardiac hypertrophy during AngII stimulation.
this study demonstrated that Ang II (zeige AGT Proteine) could increase TRPC6 (zeige TRPC6 Proteine) induced Ca(2 (zeige CA2 Proteine)+) influx and enhance autophagy through increasing reactive oxygen species levels in podocytes, and autophagy could protect Ang II (zeige AGT Proteine)-treated podocytes.
These results implied that AngII could effectively induce EpiCs to differentiate into vascular smooth muscle-like cells through the AT1 receptor (zeige AGTRAP Proteine).
Results suggest the involvement of angiotensin II (Ang II), through its angiotensin type-1 receptor (AT1R (zeige AGTRAP Proteine)) in the inflammation induced by Aah (zeige ASPH Proteine) venom, in the heart and the aorta.
The protein encoded by this gene, pre-angiotensinogen or angiotensinogen precursor, is expressed in the liver and is cleaved by the enzyme renin in response to lowered blood pressure. The resulting product, angiotensin I, is then cleaved by angiotensin converting enzyme (ACE) to generate the physiologically active enzyme angiotensin II. The protein is involved in maintaining blood pressure and in the pathogenesis of essential hypertension and preeclampsia. Mutations in this gene are associated with susceptibility to essential hypertension, and can cause renal tubular dysgenesis, a severe disorder of renal tubular development. Defects in this gene have also been associated with non-familial structural atrial fibrillation, and inflammatory bowel disease.
alpha-1 antiproteinase, antitrypsin
, angiotensin I
, angiotensin II
, serine (or cysteine) proteinase inhibitor
, serpin A8
, angiotensinogen (PAT)
, angiotensin ll