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The ACE (zeige ACE Proteine) and AGT (zeige AGXT Proteine) gene polymorphisms are not associated with the progress of diabetes developing into retinopathy in Chinese patients with type 2 diabetes.
AGT (zeige AGXT Proteine) M235T and T174M variants contribute to an increased risk of developing preeclampsia (PE), and for M235T to PE severity.
Data, including data using network analysis, suggest that angiotensinogen (AGT), mitogen-activated protein kinase-14 (MAPK14 (zeige MAPK14 Proteine)), and prothrombin (zeige F2 Proteine) (F2) in placental villous tissues are core factors in early embryonic development; these studies involved proteomics and bioinformatics analysis of altered protein expression in placental villous tissue from early recurrent miscarriage patients in comparison to control tissues.
The AGT (zeige AGXT Proteine) (M235T) gene polymorphism do not seem to have a significant effect on the development of clinical properties or cardiovascular comordities of acromegalic patients.
Angiotensin II has a role in increasing glomerular permeability by beta-arrestin mediated nephrin (zeige NPHS1 Proteine) endocytosis
After donor nephrectomy, increasing uAGT (zeige DPAGT1 Proteine) levels can be the result of activation of the intrarenal renin (zeige REN Proteine)-angiotensin system affecting the compensatory changes in the remaining kidney.
M235T polymorphism of the AGT (zeige AGXT Proteine) gene seems unrelated to the development or the clinical course of endometriosis.
AGT (zeige AGXT Proteine) missense polymorphisms are not associated with diabetic nephropathy in our subset of Slovenian type 2 diabetes mellitus patients
Association of AGT (zeige AGXT Proteine) single nucleotide polymorphism rs3789678 and gestational hypertension in Chinese population.
The T allele of AGT (zeige AGXT Proteine) may play a role in the pathogenesis of preeclampsia in South African Black women.
ANG II is up-regulated in serum and heart tissues of mice with EAM and that ANG II significantly drives monocyte/macrophage infiltration through the C-C chemokine receptor 2/5 (CCR2/5) axis.
results established that A20 (zeige TNFAIP3 Proteine) is involved in the renoprotective effect by calcitriol via negatively modulating the NF-kappaB (zeige NFKB1 Proteine) pathway and necroptotic pathway in AngII-induced renal injury.
NLRP3 (zeige NLRP3 Proteine) gene deletion attenuates Ang II-induced NLRP3 (zeige NLRP3 Proteine) inflammasome activation, phenotypic transformation from a contractile phenotype to a synthetic phenotype and proliferation in primary mice Vascular Smooth Muscle Cells.
adipocyte-derived Agt (zeige AGXT Proteine) has essentially no contribution to the plasma concentration and no impact on blood pressure compared to liver-derived Agt (zeige AGXT Proteine).
Lung ischemia-reperfusion injury causes a dysregulation of circulating Ang 2 (zeige ANGPT2 Proteine) levels and plasma PREP (zeige PREP Proteine) activity, although no direct link between both phenomena could be shown.
Inhibition of TLR4 (zeige TLR4 Proteine) ameliorates AngII-impaired cavernosal relaxation, decreases TNF-alpha (zeige TNF Proteine) levels, and restores Nitric Oxide bioavailability, demonstrating that TLR4 (zeige TLR4 Proteine) partly mediates AngII-induced cavernosal dysfunction.
Our study is the first to show the important role of IL-6 (zeige IL6 Proteine) in regulating cardiac pathogenesis via inflammation and apoptosis during AngII-induced hypertension. We also provide a novel link between IL-6 (zeige IL6 Proteine)/STAT3 (zeige STAT3 Proteine) and EndoG (zeige ENDOG Proteine)/MEF2A (zeige MEF2A Proteine) pathway that affects cardiac hypertrophy during AngII stimulation.
this study demonstrated that Ang II could increase TRPC6 (zeige TRPC6 Proteine) induced Ca(2 (zeige CA2 Proteine)+) influx and enhance autophagy through increasing reactive oxygen species levels in podocytes, and autophagy could protect Ang II-treated podocytes.
These results implied that AngII could effectively induce EpiCs to differentiate into vascular smooth muscle-like cells through the AT1 receptor (zeige AGTRAP Proteine).
Results suggest the involvement of angiotensin II (Ang II), through its angiotensin type-1 receptor (AT1R (zeige AGTRAP Proteine)) in the inflammation induced by Aah (zeige ASPH Proteine) venom, in the heart and the aorta.
In conclusion, endothelial vWF (zeige VWF Proteine) knockdown prevented angiotensin II-induced ET-1 (zeige EDN1 Proteine) upregulation through attenuation of NOX-mediated O2- production.
Data suggest that intra-adrenal metabolism of Ang II to Ang III is required for zona glomerulosa cell-mediated relaxation of adrenal arterioles but not for aldosterone secretion.
NADPH oxidase (zeige NOX1 Proteine) plays an important role in proMMP-2 expression and activation and MMP-2 (zeige MMP2 Proteine) mediated SMC (zeige DYM Proteine) proliferation occurs through the involvement of Spm (zeige NPC1 Proteine)-Cer (zeige CBLN1 Proteine)-S1P (zeige MBTPS1 Proteine) signaling axis under ANG II stimulation of PASMCs
The metabolism of angiotensin II (Ang II) to angiotensin III (Ang III) and its role in the vasorelaxation response in adrenal arteries are reported.
The study identified the serine phosphorylation (p-Ser (zeige SIGLEC1 Proteine)) sites induced by PKC-Beta (zeige PRKCB Proteine) activation or AGT, which inhibits insulin (zeige INS Proteine)-induced p-Tyr (zeige TYR Proteine) sites on IRS2 (zeige IRS2 Proteine) and its signals in endothelial cells.
Data suggest up-regulation of AGT in granulosa cells and of Ang II in follicular fluid during preovulatory period; Ang II appears to amplify stimulatory effects of luteinizing hormone on secretion of progesterone/prostaglandins by granulosa cells.
Data suggest that angiotensin II promotes uptake/accumulation of iron (non-transferrin (zeige Tf Proteine) bound iron) into vascular endothelial cells; such iron accumulation appears to depend on activation of angiotensin type 1 receptor and promotes oxidative stress.
a critical role for H(2)O(2) in angiotensin-II signaling to the endothelial cytoskeleton in a novel pathway that is critically dependent on MARCKS, Rac1, and c-Abl.
The objective of this study was to characterize the profiles of Ang-(1-7), MAS receptor, ACE(2), NEP and PEP during the ovulatory process in cattle.
Fetal adrenal cells in primary culture respond to angiotensin-II by increasing aldosterone production and aldosterone synthase (zeige CYP11B2 Proteine) [P450c18/CYP11B2 (zeige CYP11B2 Proteine)] activity.
ANG II inhibits bTREK-1 K(+) channels by a Ca(2+)-dependent mechanism that does not require the depletion of membrane-associated PIP(2).
The protein encoded by this gene, pre-angiotensinogen or angiotensinogen precursor, is expressed in the liver and is cleaved by the enzyme renin in response to lowered blood pressure. The resulting product, angiotensin I, is then cleaved by angiotensin converting enzyme (ACE) to generate the physiologically active enzyme angiotensin II. The protein is involved in maintaining blood pressure and in the pathogenesis of essential hypertension and preeclampsia. Mutations in this gene are associated with susceptibility to essential hypertension, and can cause renal tubular dysgenesis, a severe disorder of renal tubular development. Defects in this gene have also been associated with non-familial structural atrial fibrillation, and inflammatory bowel disease.
alpha-1 antiproteinase, antitrypsin
, angiotensin I
, angiotensin II
, serine (or cysteine) proteinase inhibitor
, serpin A8
, angiotensin ll
, angiotensinogen (PAT)
, zC8A9.1 (angiotensinogen )
, Serpin A8