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Mutation in PIN2 (zeige TERF1 Antikörper) conservative cysteine domain leads to increased intracellular accumulation and altered protein distribution within plasma membrane micro-domains.
The results suggest a peroxisomal ROS (zeige ROS1 Antikörper)-based auxin signaling pathway involving spatiotemporal-dependent CuAOzeta functional regulation of PIN2 (zeige TERF1 Antikörper) homeostasis, auxin distribution, and LR development.
Data indicate that neither the naturally occurring auxins indole-3-acetic acid and indole-3-butyric acid, nor the synthetic auxin analogs were able to inhibit endocytosis of the putative auxin transporter PIN (zeige DYNLL1 Antikörper)-FORMED2 (PIN2 (zeige TERF1 Antikörper)) in root epidermis cells.
AUX1 and PIN2 (zeige TERF1 Antikörper) protect lateral root (LR) formation in Arabidopsis during the early stages of iron (Fe) stress.
Data show that ROTUNDA3 (RON3; At4g24500) protein as a regulator of the protein phosphatase 2A-driven PIN (zeige DYNLL1 Antikörper)-FORMED (PIN (zeige DYNLL1 Antikörper)) auxin transport recycling and its importance in auxin transport-related plant developmental programs.
Brief treatment with indole-3-carbinol led to a reduction in the amount of PIN1 and to mislocalization of PIN2 (zeige TERF1 Antikörper).
PIN2 (zeige TERF1 Antikörper) auxin efflux carriers are differentially controlled in tricho- and atrichoblast cells. PIN2 (zeige TERF1 Antikörper) proteins show lower abundance at the plasma membrane of trichoblast cells, despite showing higher rates of intracellular trafficking in these cells.
under low-B conditions PIN1 is down-regulated and PIN2 (zeige TERF1 Antikörper) plays an important role in root meristem maintenance.
Reduction in PIN2 intensity, polarization, and endocytosis under low phosphate conditions is MAX2 dependent.
Functional interplay between protein kinase CK2 (zeige CSNK2A1 Antikörper) and salicylic acid sustains PIN2 (zeige TERF1 Antikörper) transcriptional expression.
Acts as a component of the auxin efflux carrier. Seems to be involved in the root-specific auxin transport, and mediates the root gravitropism. Its particular localization suggest a role in the translocation of auxin towards the elongation zone.