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anti-Human DIO1 Antikörper:
anti-Rat (Rattus) DIO1 Antikörper:
anti-Mouse (Murine) DIO1 Antikörper:
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Results identified a novel pH-dependent mechanism by which epigenetic reader, the PHD (zeige PDC Antikörper) fingers of DIDO, recognize the histone mark H3K4me3. The pH-sensing ability might be necessary for normal biological processes and those characterized by altered cellular pH.
wW observed that the DIO1 (zeige DIDO1 Antikörper) rs2235544 SNP modified the association between exposure to some of the organochlorine compounds (OCs) (specifically HCB and PCB153) and maternal thyroid hormone (zeige PTH Antikörper) levels. These results strengthen the hypothesis that the deiodinase enzymes play a role in explaining the disruption of thyroid hormones in relation to exposure to OCs.
data supports the notion that suppressed DIO1 (zeige DIDO1 Antikörper) expression and changes in local availability of thyroid hormones might favor a shift from a differentiated to a more proliferation-prone state of cancer tissues and cell lines
evaluated the Death Inducer-Obliterator (DIDO) (variants DIDO 1 (zeige DIDO1 Antikörper), 2 and 3) levels in CML (zeige BCR Antikörper), PV, ET and PMF (zeige PRB1 Antikörper) patients. Our data reported the DIDO 1 (zeige DIDO1 Antikörper), 2 and 3 differential expressions in Myeloproliferative Neoplasms.
results indicated that LXRalpha (zeige NR1H3 Antikörper) plays a specific and important role in activation of TH by regulating D1, and that LXRalpha (zeige NR1H3 Antikörper) binds to and regulates the hDIO1 promoter, competing with TRbeta (zeige TXNRD2 Antikörper) on specific sequences within the promoter.
The DIO1 (zeige DIDO1 Antikörper) gene is related to the depression.
Functional variants within the DIO1 (zeige DIDO1 Antikörper) gene that affect triiodothyronine (T3) levels seem not to be associated with cognitive functions.
All these results indicate that the oxidative stress downregulates the conversion of T4 to T3 through DIO1 (zeige DIDO1 Antikörper) function in HepG2 cells.
Thyroid hormone (zeige PTH Antikörper) deiodinases D1, D2, and D3 are differentially expressed in endothelial cells following thyroid hormone (zeige PTH Antikörper) exposure.
The pattern of expression and role of triiodothyronine (T3) receptors and type I 5'-deiodinase in breast carcinomas, benign breast diseases, lactational change, and normal breast epithelium.
Reductions in Dio1 expression reduce the expression of ApoA-I (zeige APOA1 Antikörper) in a 3,5,3'-triiodothyronine-/thyroid hormone (zeige PTH Antikörper) response element-independent manner.
Systemic changes in thyroid hormone (zeige PTH Antikörper) economy as a result of acute food deprivation are not dependent on the deiodinases D1 or D2 but are mediated in part by sequestration of type4 and type3 in tissues and their enhanced metabolism by deiodinase D3.
Results demonstrate changes in D1 activity in white adipose tissue under conditions of changing adiposity, and a stimulatory effect of leptin (zeige LEP Antikörper) on D1 activity in the tissue.
A decreased hepatic D1 activity could be the biochemical basis of some of the abnormal thyroid parameters observed in cystic fibrosis (zeige S100A8 Antikörper)
The type 1 deiodinase (D1) is an important source of Triiodothyronine in the euthyroid state.
HNF4alpha (zeige HNF4A Antikörper) regulates thyroid hormone (zeige PTH Antikörper) homeostasis through transcriptional regulation of the Dio1 gene with GATA4 (zeige GATA4 Antikörper) and KLF9 (zeige KLF9 Antikörper)
This study investigated deiodinase 1 (Dio1), deiodinase 2 (Dio2 (zeige DIO2 Antikörper)), and deiodinase 3 (Dio3 (zeige DIO3 Antikörper)) mRNA expression at the several zebrafish life stages and found life stage specific expression of these genes that were highly localized.
Apoptosis, a major form of cell death, is an efficient mechanism for eliminating unwanted cells and is of central importance for development and homeostasis in metazoan animals. In mice, the death inducer-obliterator-1 gene is upregulated by apoptotic signals and encodes a cytoplasmic protein that translocates to the nucleus upon apoptotic signal activation. When overexpressed, the mouse protein induced apoptosis in cell lines growing in vitro. This gene is similar to the mouse gene and therefore is thought to be involved in apoptosis. Alternatively spliced transcripts have been found for this gene, encoding multiple isoforms.
, thyroxine deiodinase type I (selenoprotein)
, type 1 DI
, type I iodothyronine deiodinase
, type-I 5'-deiodinase
, death-associated transcription factor 1
, death-inducer obliterator 1
, thyroxine deiodinase, type I
, thyroxine deiodinase type 1
, Type 1 DI
, Type-I 5'-deiodinase
, deiodinase, iodothyronine, type I
, type 1 deiodinase