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The authors discovered that PKR1 regulates epicardial-mesenchymal transition (EMT (zeige ITK ELISA Kits)) for epicardial-derived progenitor cell (EPDC), formation. This event affects at least three consequential steps during heart development: (i) EPDC and cardiomyocyte proliferation involved in thickening of an outer compact ventricular chamber wall, (ii) rhythmicity, (iii) formation of coronary circulation.
These results establish PKR1 via NFATc3 (zeige NFATC3 ELISA Kits) as a crucial modifier of mesenchymal-epithelial transition processing to the development of nephron.
show that MRAP2 significantly and specifically inhibits PKR1 signaling.
Data show that the prokineticins and their receptors PROK2 (zeige PROK2 ELISA Kits), PKR1 and PKR2 (zeige PROKR2 ELISA Kits) contributes to altered sensitivity in diabetic neuropathy and its inhibition blocked both allodynia and inflammatory events underlying disease.
These results suggest PKR1 to be a crucial player in the preadipocyte proliferation and differentiation.
Loss of PKR1 causes renal and cardiac structural and functional changes because of deficits in survival signaling, mitochondrial, and progenitor cell functions in found both organs.
The functional characteristics of coronary endothelial cells depend on the expression of PKR1 and PKR2 (zeige PROKR2 ELISA Kits) levels and the divergent signaling pathways used by these receptors.
Identification and molecular characterization of two closely related G protein-coupled receptors (prokineticin receptor)
PKR1 protein was localised to the labyrinth layer and showed the same pattern of expression as EG-VEGF (zeige Prok1 ELISA Kits) in mouse placenta.
Cardiomyocyte-PKR1 signaling upregulates its own ligand prokineticin-2 (zeige PROK2 ELISA Kits) that acts as a paracrine factor, triggering epicardial-derived progenitor cell proliferation/differentiation.
a significant association between PKR2 rs6053283 polymorphism and Recurrent pregnancy loss (RPL)(P=0.003), whereas no association was observed between PKR1 rs4627609 polymorphism and RPL (P=0.929) in the Chinese Han population.
Data suggest that prokineticins (PROK1 (zeige Prok1 ELISA Kits) and PROK2 (zeige PROK2 ELISA Kits)) and prokineticin receptors (PROKR1 and PROKR2 (zeige PROKR2 ELISA Kits)) act as main regulators of physiological functions of ovary, uterus, placenta, and testis. [REVIEW]
EG-VEGF (zeige Prok1 ELISA Kits) and its receptor PKR1 might play a role in the pathogenesis of adrenocortical tumors and could serve as prognostic markers for this rare malignant disease.
Expression of PROK1 (zeige Prok1 ELISA Kits) and PROKR1 was significantly higher in mid-gestation ovaries (17-20 wk) than at earlier gestations (8-11 and 14-16 wk).
Study corroborates the clinical relevance of the EG-VEGF (zeige Prok1 ELISA Kits) system in human early pregnancy, and provides evidence for the gene-gene interactions of EG-VEGF (zeige Prok1 ELISA Kits) and PROKR variants.
hCG (zeige CGA ELISA Kits) increases EG-VEGF (zeige Prok1 ELISA Kits), PROKR1 and PROKR2 (zeige PROKR2 ELISA Kits) mRNA and protein expression in a dose- and time-dependent manner, demonstrating a new role for hCG (zeige CGA ELISA Kits) in the regulation of EG-VEGF (zeige Prok1 ELISA Kits) and its receptors
Findings, together with the detection of sequence variants in PROKR1, PROK1 (zeige Prok1 ELISA Kits) and PROKR2 (zeige PROKR2 ELISA Kits) genes associated to HSCR (zeige EDNRB ELISA Kits) and, in some cases in combination with RET (zeige RET ELISA Kits) or GDNF mutations, provide evidence to consider them as susceptibility genes for HSCR (zeige EDNRB ELISA Kits).
The number of PKR1 is not reduced in preeclampsia.
Data suggest that smoking targets human Fallopian tubes via nAChRalpha-7 to increase tubal PROKR1, leading to alterations in the tubal microenvironment that could predispose to ectopic pregnancy.
Data show that expression of PK1 (zeige PKLR ELISA Kits) and PKR1 was detected in primary MM cells and myeloma cell lines.
PROK1 (zeige Prok1 ELISA Kits), acting via PROKR1, may be involved in the recruitment of monocytes to regressing CL and atretic follicles and their consequent activation therein.
G protein-coupled receptor for prokineticin
G protein-coupled receptor 73
, G-protein coupled receptor 73
, G protein-coupled receptor ZAQ
, G-protein coupled receptor ZAQ
, prokineticin receptor 1
, prokineticin receptor 1-like