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Results suggest that high expression of KIF3C in breast cancer may be associated with tumor progression and metastasis.
FMRP acts also as a molecular adaptor between RNA granules and the neurospecific kinesin KIF3C.
Results reveal that engineered homodimeric KIF3CC is targeted to the microtubule plus-end, acts as a potent catastrophe factor through an increase in microtubule catastrophe frequency, and does so by elimination of the dependence of the catastrophe rate on microtubule lifetime. Moreover, KIF3CC accelerates the catastrophe rate without altering the microtubule growth rate.
Study found that both KIF3AC and KIF3AB can traverse long distances over the microtubule track, revealing that these motors are adept and well-tuned for their specialized roles as cargo transporters
Data suggest biocatalysis by Kif3a homodimer, Kif3c homodimer, or Kif3a/Kif3c heterodimer vary; microtubule interaction is very fast for Kif3a homodimer; Kif3c homodimer interaction is much slower; Kif3a/Kif3c heterodimer interaction is intermediate.
KIF3C is an injury-specific kinesin that contributes to axon growth and regeneration by regulating and organizing the microtubule cytoskeleton in the growth cone.
may be a vesicle associated microtubule motor protein involved in muscle cell function
kinesin family member 3C
, kinesin-related protein
, Kinesin-like protein KIF3C
, kinesin-like protein KIF3C-like
, KIF3C variant protein
, kinesin-like protein KIF3C
, N-4 kinesin