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anti-Mouse (Murine) GTF2H4 Antikörper:
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functional genetic variants of GTF2H4 confer susceptibility to lung cancer.
GTF2H4 variants may not be associated with susceptibility to aspirin-exacerbated respiratory disease and obstructive symptoms in asthmatics.
A single nucleotide polymorphism variant within the general transcription factor IIH, polypeptide 4 gene, GTF2H4, on chromosome 6p21.33 was significantly associated with MS
General transcription factor IIH protects promoters from PC4-mediated repression by relieving the topological constraint imposed by PC4 through the ERCC3 helicase activity rather than by reducing the repressive activity of PC4 via its phosphorylation.
The second domain of TFIIH p62 subunit(residues 186-240) contains a region of high sequence conservation with an invariant tyrosine/phenylalanine motif which could play a key role as a protein-protein recognition module within TFIIH.
TFIIH uses an expanded proximal promoter to regulate c-myc expression
We conclude that the recruitment and activation of TFIIH represents a rate-limiting step for the emergence of HIV from latency.
Alterations of chromatin at the RNA polymerase II stall site, which depend on CSB and TFIIH at least, are necessary for the UV-induced translocation of CSA to the nuclear matrix.
XPG forms a stable complex with TFIIH, which is active in transcription and nucleotide excision repair
mechanism in which the helicase activity of XPB is not used for the opening and repair of damaged DNA, which is instead only driven by its ATPase activity, in combination with the helicase activity of XPD
The specific binding of the C-terminal acidic domain (AC-D) of the human TFIIEalpha subunit to the pleckstrin homology domain (PH-D) of the human TFIIH p62 subunit is demonstrated.
TAF7 interacts with the transcription factors, TFIIH and P-TEFb, resulting in the inhibition of their Pol II CTD kinase activities
TFIIH changes subunit composition in response to DNA damage. The CAK is released from the core during nucleotide excision repair (NER).
For 6-4 photoproducts, we show that TFIIH complexes carrying an NH(2)-terminal XPD mutated protein are also deficient in recruitment of NER proteins downstream of TFIIH
The frequency of congenital ichthyosis, collodion-baby type, was significantly higher in the TFIIH mutated group of trichothiodystrophy patients.
Study demonstrates that an essential initiation factor, TFB2, forms a network of interactions with DNA near the transcription start site and facilitates promoter melting but may not be essential for promoter recognition.
Component of the core-TFIIH basal transcription factor involved in nucleotide excision repair (NER) of DNA and, when complexed to CAK, in RNA transcription by RNA polymerase II (By similarity).
general transcription factor IIH, polypeptide 4
, general transcription factor II H, polypeptide 4
, ATP-binding cassette, sub-family F (GCN20), member 1
, General transcription factor IIH polypeptide 4
, general transcription factor IIH subunit 4
, general transcription factor IIH, polypeptide 4, 52kDa, gene 2
, general transcription factor IIH, polypeptide 4, 52kDa
, general transcription factor IIH subunit 4-like
, TFIIH basal transcription factor complex p52 subunit
, basic transcription factor 2 52 kDa subunit
, TFIIH transcription/DNA repair factor p52 subunit
, BTF2 p52
, general transcription factor IIH polypeptide 4
, general transcription factor IIH, polypeptide 4, 52kDa, gene 1