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anti-Human TGFB3 Antikörper:
anti-Mouse (Murine) TGFB3 Antikörper:
anti-Rat (Rattus) TGFB3 Antikörper:
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Cow (Bovine) Polyclonal TGFB3 Primary Antibody für WB - ABIN2782224
Murata, Kinoshita: Establishment of proprotein convertase, furinA knocked-out lines in medaka, Oryzias latipes, and unique form of medaka furin-like prorprotein convertase (mflPC). in Comparative biochemistry and physiology. Toxicology & pharmacology : CBP 2015
This review highlights advances in the understanding of the cellular sources, activation processes, contextual determinants, and immunological roles of TGF-beta3 with comparisons to other TGF-beta (zeige TGFB1 Antikörper) isoforms.
This study showed that YOD1 (zeige YOD1 Antikörper) overexpression enhances cell migration by promoting TGF-beta3 signaling which may play an important role in lip and palate formation.
We suggest that increased TGFbeta3 levels are responsible for development of aggressive prostate cancer in African American patients as a consequence of development of resistance to inhibitory effects of TGFbeta (zeige TGFB1 Antikörper) on cell proliferation and induction of invasive metastatic behavior
data implies that miR (zeige MLXIP Antikörper)-140 is a potent chondrogenic differentiation inducer for iPSCs and also, we have showed increasing chondrogenic differentiation by using overexpression of miR (zeige MLXIP Antikörper)-140 and TGFbeta3.
The increase in TGF-beta3 found in inflammatory wound healing (WF) highlights its negative effect on wound healing, while the increased levels of sEng in granulating WF affects the leukocyte adhesion/transmigration through the endothelium, reducing the inflammatory response and favoring the wound healing.
Data indicate that TGFb1 (zeige TGFB1 Antikörper) and TGFb3, but not TGFb2 (zeige TGFB2 Antikörper), showed higher expression levels in invasive breast cancer compared to normal tissues.
Cancer cachexia promotes the development of adipose tissue (AT) fibrosis, in association with altered transforming growth factor-beta (TGFbeta (zeige TGFB1 Antikörper)) signaling, compromising AT organization and function.
Higher TGF-beta3 serum concentrations are a risk factor for uterine fibroids.
High expression of TGF-beta3 in preeclampsia decidua stimulates miR (zeige MLXIP Antikörper)-494 in decidual mesenchymal stem cells (MSC (zeige MSC Antikörper)) and attenuates the regulation of MSC (zeige MSC Antikörper) switching the macrophage toward M2 type, contributing to an immune imbalance at maternal-fetal interface.
The frequency GA genotype of transforming growth factor beta 3 (TGFbeta3) gene was associated with increased risk of non-syndromic cleft palate only (NS CPO (zeige CPOX Antikörper)).
Data indicated that autophagy was required for TGF-beta3 induced airway mucous hyper-production.
Results suggest that the miR (zeige MLXIP Antikörper)-140-3p is involved in osteoblast differentiation as a critical regulatory factor between Wnt3a (zeige WNT3A Antikörper) and TGFbeta3 signaling pathways.
Egr2 (zeige EGR2 Antikörper) and Egr3 (zeige EGR3 Antikörper) expressed in T cells cooperatively prevent humoral immune responses by supporting TGF-beta3 secretion.
We identified TGF-beta3 as the top-ranked gene, a critical component of the transforming growth factor-b (zeige CFB Antikörper) (TGF-b) and mitogen activated protein kinase (zeige MAPK1 Antikörper) (MAPK (zeige MAPK1 Antikörper)) signalling pathways in cardiac fibrosis
Data (including data from studies using knockout mice) suggest Garp/Lrrc32 (zeige LRRC32 Antikörper) is involved in up-regulation of Tgfb3 and is essential for normal embryogenesis of palate; knockout of Garp (zeige LRRC32 Antikörper) causes postnatal lethality, cleft palate, and decreased apoptosis and Smad2 (zeige SMAD2 Antikörper) phosphorylation in medial edge epithelial cells of palatal shelf of embryos. (Garp (zeige LRRC32 Antikörper) = glycoprotein A repetitions predominant (zeige LRRC32 Antikörper) protein; Smad2 (zeige SMAD2 Antikörper) = MAD homolog protein 2)
TGFb3-induced down-regulation of p63 (zeige CKAP4 Antikörper) in the medial edge epithelia of the palatal shelves is a pre-requisite for palatal fusion by facilitating periderm migration from, and reducing the proliferative potential of, the midline epithelial seam thereby preventing cleft palate.
Early activation of hepatic stellate cell and imbalance expression of TGF-beta1 (zeige TGFB1 Antikörper) and TGF-beta3 existed in ConA-induced acute autoimmune liver injury.
CCN4 (zeige WISP1 Antikörper) has a positive influence on chondrogenic differentiation by modulating the effects of TGF-beta3.
TGFbeta3 increases IRF6 expression and subsequently regulates SNAI2 expression; IRF6 appears to regulate epithelial mesenchymal transition during palatal fusion via SNAI2.
Analysis of the Tgf-beta-3 knockout mouse model has enabled identification of miRNAs with altered expression that may contribute to the cleft palate phenotype.
The addition of TGF-beta3 to the 3D cultures further up-regulates the expression of these genes and also induces the expression of mature tenocyte markers Tenomodulin (zeige TNMD Antikörper) and Thrombospondin-4 (zeige THBS4 Antikörper).
TGFbeta (zeige TGFB1 Antikörper) may play a role in the overall process of luteinization, but it appears not to influence steroidogenesis in luteinizing pig follicles.
In swine, TGF-beta3 mRNA is expressed throughout the oestrus cycle.
transforming growth factor beta (TGFbeta (zeige TGFB1 Antikörper)) is required for hematopoietic progenitor cell specification. The requirement for TGFbeta (zeige TGFB1 Antikörper) is two fold and sequential: autocrine via Tgfbeta1a and Tgfbeta1b produced in the endothelial cells themselves, followed by a paracrine input of Tgfbeta3 from the notochord, suggesting that the former programs the hemogenic endothelium and the latter drives endothelial-to-hematopoietic tran...
These data suggest Pez (zeige PTPN14 Antikörper) plays a crucial role in organogenesis by inducing TGFbeta (zeige TGFB1 Antikörper) and epithelial-mesenchymal transition.
Tissues exposed to TGF-beta3 had significantly increased glycosaminoglycan and total collagen protein production along with upregulated cartilage-specific gene expression, resulting in tissues with a higher Young's Modulus
Data show that TGF-beta (zeige TGFB1 Antikörper) pathways operate during ovarian fetal development, and fibrillin 3 (zeige FBN3 Antikörper) is highly expressed at a critical stage early in developing human and bovine fetal ovaries.
This gene encodes a member of the TGF-beta family of proteins. The encoded protein is secreted and is involved in embryogenesis and cell differentiation. Defects in this gene are a cause of familial arrhythmogenic right ventricular dysplasia 1.
transforming growth factor, beta 3
, transforming growth factor beta-3
, transforming growth factor beta-3 preproprotein
, transforming growth factor beta-3-like
, transforming growth factor-beta3
, transforming growth factor b3
, transforming growth factor-beta 3
, protein kinase
, tgf beta 3