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Human Polyclonal AGER Primary Antibody für IHC (p), WB - ABIN3043529
Qin, Niu, Wang, Xu, Qiao, Gu: Heparanase induced by advanced glycation end products (AGEs) promotes macrophage migration involving RAGE and PI3K/AKT pathway. in Cardiovascular diabetology 2013
Show all 8 Pubmed References
Human Polyclonal AGER Primary Antibody für IHC (fro), IHC (p) - ABIN3044329
Wang, Zhang, Liu, Cui, Yang, Li, Du: Tanshinone II A down-regulates HMGB1, RAGE, TLR4, NF-kappaB expression, ameliorates BBB permeability and endothelial cell function, and protects rat brains against focal ischemia. in Brain research 2010
Show all 6 Pubmed References
Rat (Rattus) Polyclonal AGER Primary Antibody für FACS, IF (p) - ABIN725900
Huang, Yao, Zhang, Dong, Yu, Sheng: The effect of high-mobility group box 1 protein on activity of regulatory T cells after thermal injury in rats. in Shock (Augusta, Ga.) 2009
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Human Polyclonal AGER Primary Antibody für IHC (p), WB - ABIN548553
Taguchi, Blood, del Toro, Canet, Lee, Qu, Tanji, Lu, Lalla, Fu, Hofmann, Kislinger, Ingram, Lu, Tanaka, Hori, Ogawa, Stern, Schmidt: Blockade of RAGE-amphoterin signalling suppresses tumour growth and metastases. in Nature 2000
Show all 3 Pubmed References
Human Polyclonal AGER Primary Antibody für IF (p), IHC (p) - ABIN1386239
Durning, Preston-Hurlburt, Clark, Xu, Herold: The Receptor for Advanced Glycation Endproducts Drives T Cell Survival and Inflammation in Type 1 Diabetes Mellitus. in Journal of immunology (Baltimore, Md. : 1950) 2016
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Rat (Rattus) Polyclonal AGER Primary Antibody für FACS, IF (p) - ABIN725907
Zhu, Yao, Zhang, Dong, Yu, Sheng: Anti-RAGE antibody ameliorates severe thermal injury in rats through regulating cellular immune function. in Acta pharmacologica Sinica 2014
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Human Polyclonal AGER Primary Antibody für IHC (p), WB - ABIN1397268
Guan, Sheu, Wu, Chiang, Liu: ATP synthase subunit-β down-regulation aggravates diabetic nephropathy. in Scientific reports 2015
Human Polyclonal AGER Primary Antibody für ICC, IF - ABIN449216
Oztay, Sancar-Bas, Gezginci-Oktayoglu, Ercin, Bolkent: Exendin-4 partly ameliorates - hyperglycemia-mediated tissue damage in lungs of streptozotocin-induced diabetic mice. in Peptides 2018
Human Polyclonal AGER Primary Antibody für ELISA, ICC - ABIN6259820
Wu, Zheng, Bai, Xia, Zhang, Guo: HMGB1/RAGE axis mediates the apoptosis, invasion, autophagy, and angiogenesis of the renal cell carcinoma. in OncoTargets and therapy 2018
Results show that RAGE is activated by HMGB1 (zeige HMGB1 Antikörper) to induce EMT (zeige ITK Antikörper) in prostate cancer cells.
high mobility group box 1 (zeige HMGB1 Antikörper)-receptor for advanced glycation end-products (HMGB1 (zeige HMGB1 Antikörper)-RAGE) signaling pathway may be involved in the pathogenesis of preterm premature rupture of the membranes (pPROM (zeige SERPINH1 Antikörper)).
Results show that RAGE is upregulated in breast cancer tissues, and confirmed that RAGE was a direct target of miR (zeige MLXIP Antikörper)-328.
The results suggest that S100A12 (zeige S100A12 Antikörper) does not participate in the induction of inflammation in dental pulp. However, RAGE can participate in the inflammation in the pulp of males.
The results show for the first time that RAGE is present in neuronally-derived plasma exosomes, and suggest that exosomal RAGE may be a novel biomarker that reflects pathophysiological processes in the brain.
Decreased soluble RAGE in neutrophilic asthma is correlated with disease severity and RAGE G82S variants.
Our study provides novel evidence for a potential role of AGER in bridging human papillomavirus (HPV)-induced inflammation and cervical cancer.
Plasmatic RAGE level is significantly lower in patients with prosthetic-joint-associated infections.
Inhibition of GLO1 (zeige GLO1 Antikörper) in Glioblastoma Multiforme Increases DNA-AGEs, Stimulates RAGE Expression, and Inhibits Brain Tumor Growth in Orthotopic Mouse Models
a significant association between RAGE gene rs1800624 and rs1800625 polymorphisms and Age-related macular degeneration risk, is reported.
microglial RAGE activation in presence of amyloid beta-enriched environment contributes to the entorhinal cortex vulnerability.
Established a murine model of myocardial ischemia-reperfusion injury; investigated and found remote ischemic postconditioning protects against IR injury thru RAGE-HMGB1 (zeige HMGB1 Antikörper) Pathway.
In this study, we found that the wnt (zeige WNT2 Antikörper) co-receptor Lrp6 (zeige LRP6 Antikörper) was a potent positive regulator of beta-catenin (zeige CTNNB1 Antikörper) signaling in TDI-induced asthma models, both in vivo and in vitro. Additionally, for the first time, we demonstrated that RAGE could mediate phosphorylation of Lrp6 (zeige LRP6 Antikörper), suggesting a functional cross talk between RAGE and the canonical wnt (zeige WNT2 Antikörper)/beta-catenin (zeige CTNNB1 Antikörper) signaling pathway involved in mediating beta-catenin (zeige CTNNB1 Antikörper) activation.
Chronic unpredictable stress (CUS) promotes significant morphological changes and causes robust upregulation of HMGB1 (zeige HMGB1 Antikörper) messenger RNA in enriched hippocampal microglia and robust and persistent upregulation of RAGE messenger RNA. CUS increased surface expression of RAGE protein on hippocampal microglia and anhedonic behavior. RAGE knockout mice were resilient to stress-induced anhedonia.
study found that diabetes predisposes to more severe infections because of additional inflammatory output through dual activation of MyD88 (zeige MYD88 Antikörper) by not only TLR4 (zeige TLR4 Antikörper) but also RAGE
RAGE controls myocardial dysfunction and oxidative stress in high-fat fed mice by sustaining mitochondrial dynamics and autophagy-lysosome pathway.
these data provide a previously uncharacterized in vivo mechanism contingent on oligodeoxy-nucleotide -administered dose, where TLR9 (zeige TLR9 Antikörper) governs the primary response and RAGE plays a distinct and cooperative function in providing a pivotal role in balancing the immune response.
data demonstrate that under the diabetic condition, DRG neurons are directly affected by elevated levels of glucose, independent of vascular or glial signals, and dependent on RAGE expression.
findings suggest that HMGB1 (zeige HMGB1 Antikörper) induces the transcytosis of albumin (zeige ALB Antikörper) via RAGE-dependent Src (zeige SRC Antikörper) phosphorylation and Cav-1 (zeige CAV1 Antikörper) phosphorylation. These studies revealed a new mechanism of HMGB1 (zeige HMGB1 Antikörper)-induced endothelial hyperpermeability.
data suggest that S100A8 (zeige S100A8 Antikörper)/A9 acts directly on BV-2 microglial cells via binding to TLR4 (zeige TLR4 Antikörper) and RAGE on the membrane and then stimulates the secretion of proinflammatory cytokines through ERK (zeige EPHB2 Antikörper) and JNK (zeige MAPK8 Antikörper)-mediated NF-kappaB (zeige NFKB1 Antikörper) activity in BV-2 microglial cells.
Our results suggested that the increased RAGE expression in inflammatory circumstances and interaction with AGEs are risk factors in decreasing of aggrecan (zeige ACAN Antikörper) content in nucleus pulposus.
The advanced glycosylation end product (AGE) receptor encoded by this gene is a member of the immunoglobulin superfamily of cell surface receptors. It is a multiligand receptor, and besides AGE, interacts with other molecules implicated in homeostasis, development, and inflammation, and certain diseases, such as diabetes and Alzheimer's disease. Many alternatively spliced transcript variants encoding different isoforms, as well as non-protein-coding variants, have been described for this gene (PMID:18089847).
RAGE isoform NtRAGE-delta
, RAGE isoform sRAGE-delta
, advanced glycation end-products receptor
, receptor for advanced glycosylation end products
, advanced glycosylation end product-specific receptor variant 2
, advanced glycosylation end product-specific receptor variant 3
, advanced glycosylation end product-specific receptor variant 4
, advanced glycosylation end product-specific receptor variant 5
, advanced glycosylation end product-specific receptor variant 1
, advanced glycosylation end product-specific receptor variant 6
, advanced glycosylation end product-specific receptor variant 7
, advanced glycosylation end product-specific receptor variant 8
, advanced glycation end product receptor
, advanced glycosylation end product-specific receptor
, MAPK/MAK/MRK overlapping kinase
, renal tumor antigen