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The glycoprotein encoded by VWF functions as both an antihemophilic factor carrier and a platelet-vessel wall mediator in the blood coagulation system.
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Rat (Rattus) VWF ELISA Kit für Sandwich ELISA - ABIN367459
Baraka, Guemei, Gawad: Role of modulation of vascular endothelial growth factor and tumor necrosis factor-alpha in gastric ulcer healing in diabetic rats. in Biochemical pharmacology 2010
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Human VWF ELISA Kit für Sandwich ELISA - ABIN2703573
Ma, Su, Zhang, Ling, Yin, Bai, Ruan: The co-influence of VWD type 2B/2M mutations in the A1 domain and platelet GPIbα on the rate of cleavage to VWF by ADAMTS13. in Thrombosis research 2015
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Rat (Rattus) VWF ELISA Kit für Sandwich ELISA - ABIN416122
Imaoka, Sayama, Suzuki, Jindo, Sanbuissho: Effect of Hypertension on the Occurrence of Micro-hemorrhage in the Pancreatic Islet of Dahl Salt-sensitive Rats. in Journal of toxicologic pathology 2012
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Human VWF ELISA Kit für Sandwich ELISA - ABIN612793
Zimmerman, Ruggeri: von Willebrand disease. in Human pathology 1987
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Human VWF ELISA Kit für Sandwich ELISA - ABIN365425
Tong, Wan, Zhang, Duan, Tang, Chen, Tang, Su: Vascular endothelial cell injury partly induced by mesenteric lymph in heat stroke. in Inflammation 2014
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Human VWF ELISA Kit für Competition ELISA - ABIN4948138
Szpera-Go��dziewicz, Majcherek, Boruczkowski, Goździewicz, Dworacki, Wicherek, Bręborowicz: Circulating endothelial cells, circulating endothelial progenitor cells, and von Willebrand factor in pregnancies complicated by hypertensive disorders. in American journal of reproductive immunology (New York, N.Y. : 1989) 2017
Rat (Rattus) VWF ELISA Kit für Competition ELISA - ABIN4948139
Murata, Miyake, Takahashi, Suzuki, Fujiwara, Sato, Inoue, Kobayashi, Kanamoto: Low-Dose Sodium Nitrite Fluid Resuscitation Prevents Lethality From Crush Syndrome by Improving Nitric Oxide Consumption and Preventing Myoglobin Cytotoxicity in Kidney in A Rat Model. in Shock (Augusta, Ga.) 2016
Rabbit VWF ELISA Kit für Competition ELISA - ABIN415967
Du, Wu, Qing, Wang, Liang, Yu, Tang: Systemic and flap inflammatory response associates with thrombosis in flap venous crisis. in Inflammation 2015
Human VWF ELISA Kit für Sandwich ELISA - ABIN2685886
Morton, Griffin, Pepper, Barnes: The interaction between collagens and factor VIII/von Willebrand factor: investigation of the structural requirements for interaction. in Thrombosis research 1984
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von Willebrand Factor is steadily elevated throughout the course of dysrhythmia in NVAF patients treated with warfarin and in those with higher intensity of left atrium blood stasis.
The levels of VWF-cleaving protease ADAMTS13 (zeige ADAMTS13 ELISA Kits) among neonates were higher as compared with healthy adults, despite a significant elevation of VWF antigen (Ag) and Ristocetin cofactor (RiCof) noted in all neonates.
The pathogenic effects of candidate VWF gene mutations were explored in this study. Molecular dynamic simulations on p.M771I mutant VWF revealed distinct structural rearrangements including a large deviation in the E' domain, and significant loss of beta-sheet secondary structure.
investigated the roles of ADAMTS13 (zeige ADAMTS13 ELISA Kits) and VWF in thrombotic events of patients with Connective Tissue Diseases
vWF had a significant prognostic impact on cardiovascular mortality when OPG (zeige TNFRSF11B ELISA Kits) levels were low. Low levels of both OPG (zeige TNFRSF11B ELISA Kits) and act vWF were associated with a 99 % survival rate during the follow-up of five years.
Studied the significance of the von Willebrand factor (VWF)/ ADAMTS-13 (zeige ADAMTS13 ELISA Kits) ratio in advanced non-small-cell lung cancer (NSCLC). Findings suggest that the imbalance between VWF secretion and ADAMTS-13 (zeige ADAMTS13 ELISA Kits) may play a role in the hypercoagulability state in advanced NSCLC, and increase of the plasma VWF/ADAMTS-13 (zeige ADAMTS13 ELISA Kits) ratio may serve as an independent predictive factor for mortality in patients with advanced NSCLC.
Type 3 von Willebrand disease patients with the Pro2808Leufs*24 have bioavailable platelet-derived VWF that may produce a milder bleeding phenotype than other type 3s
vaso-occlusive crisis in sickle cell disease is associated with increased reactivity of VWF, without a pronounced ADAMTS-13 (zeige ADAMTS13 ELISA Kits) deficiency
Different patterns of VWF multimer loss were seen in heart failure patients treated with implantation of left ventricular assist device and total artificial heart, which may contribute to bleeding patterns.
Complex VWF-ADAMTS13 (zeige ADAMTS13 ELISA Kits)-mediated mechanisms disturb haemostasis in inflammatory bowel disease.
In conclusion, endothelial vWF knockdown prevented angiotensin II-induced ET-1 (zeige EDN1 ELISA Kits) upregulation through attenuation of NOX-mediated O2- production.
alterations in glycosylation of vWF and other adhesion proteins associated with the targeting of the alpha1,3-Gal (zeige GAL ELISA Kits)-epitope in mutant swine may have salutatory effects on the primate platelet activation observed in these xenografts.
Hemodynamic activation of vWF and increased plasma ADAMTS-13 (zeige ADAMTS13 ELISA Kits) may have contributed to reduced high-molecular-weight vWF multimers and impairment of the vWF-platelet aggregation pathway during mechanical circulatory support.
both the gpIb-VWF interaction and the integrin alpha(2 (zeige ITGA2 ELISA Kits))beta(1)-collagen interaction contribute to platelet adhesion under high shear stress; integrin alpha(II (zeige GSTA3 ELISA Kits))beta(1) makes a greater contribution to adhesion to type I collagen because less VWF is bound
this study shows that Von Willebrand factor protects against acute CCl4 (zeige CCL4 ELISA Kits)-induced hepatotoxicity through phospho-p38 MAPK (zeige MAPK14 ELISA Kits) signaling pathway inhibition
the ADAMTS13 (zeige ADAMTS13 ELISA Kits)-vWF axis is partially involved in the pathophysiology of kidney ischemic reperfusion injury.
Type 2N von Willebrand disease variants were associated with decreased VWF secretion and impaired factor VIII binding/stability.
identify a critical role for VWF in cerebral inflammation and blood-brain barrier damage after intracerebral haemorrhage
The low-molecular-weight heparin (LMWH) Tinzaparin inhibited Von Willebrand factor (VWF) fiber formation and vessel occlusion in tumor vessels by blocking thrombin (zeige F2 ELISA Kits)-induced endothelial cells (ECs) activation and vascular endothelial growth factor-A (VEGF-A (zeige VEGFA ELISA Kits))-mediated VWF release.
In a laser injury-induced thrombosis model, P-selectin (zeige SELP ELISA Kits) modulates thrombus propagation independently of VWF and TSP1 (zeige GZMA ELISA Kits)
Refrigeration-induced binding of VWF to platelets facilitates their rapid clearance by inducing GPIbalpha (zeige GP1BA ELISA Kits)-mediated signaling.
BLOC-2 (zeige HPS6 ELISA Kits) subunit HPS6 (zeige HPS6 ELISA Kits) deficiency affects the tubulation and secretion of von Willebrand factor from mouse endothelial cells
A novel single-domain antibody against von Willebrand factor A1 domain that interferes with VWF-platelet interactions in vivo. By using this sdAb, show that the A1 domain is pertinent to the participation of VWF in the inflammatory response.
These experiments delineate an unexpected pathway in which microbiota-triggered TLR2 signaling alters the synthesis of proadhesive VWF by the liver endothelium and favors platelet integrin-dependent thrombus growth.
The glycoprotein encoded by this gene functions as both an antihemophilic factor carrier and a platelet-vessel wall mediator in the blood coagulation system. It is crucial to the hemostasis process. Mutations in this gene or deficiencies in this protein result in von Willebrand's disease. An unprocessed pseudogene has been found on chromosome 22.
von Willebrand factor
, coagulation factor VIII VWF